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Defective ALK5 signaling in the neural crest leads to increased postmigratory neural crest cell apoptosis and severe outflow tract defects
scientific journal article
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scholarly article
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
title
Defective ALK5 signaling in the neural crest leads to increased postmigratory neural crest cell apoptosis and severe outflow tract defects
(English)
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
main subject
Transforming growth factor, beta receptor II
1 reference
stated in
GOA release 2020-03-11
Activin A receptor, type 1
1 reference
stated in
GOA release 2020-03-11
Transforming growth factor, beta receptor I
1 reference
stated in
GOA release 2020-03-11
apoptotic process
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inferred from title
author
Vesa Kaartinen
series ordinal
6
0 references
author name string
Jikui Wang
series ordinal
1
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
Andre Nagy
series ordinal
2
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
Jonas Larsson
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
Marek Dudas
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
Henry M. Sucov
series ordinal
5
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
publication date
1 November 2006
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stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
published in
BMC Developmental Biology
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
volume
6
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
page(s)
51
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
exact match
https://scigraph.springernature.com/pub.10.1186/1471-213x-6-51
0 references
cites work
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Cardiac outflow tract defects in mice lacking ALK2 in neural crest cells
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PubMed Central
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19 March 2017
BMP receptor IA is required in mammalian neural crest cells for development of the cardiac outflow tract and ventricular myocardium
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Craniofacial defects in mice lacking BMP type I receptor Alk2 in neural crest cells
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19 March 2017
Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling
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PubMed Central
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Myostatin Signals through a Transforming Growth Factor β-Like Signaling Pathway To Block Adipogenesis
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Conditional inactivation of Tgfbr2 in cranial neural crest causes cleft palate and calvaria defects
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19 March 2017
BMP signaling is required for septation of the outflow tract of the mammalian heart
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Activin type IIA and IIB receptors mediate Gdf11 signaling in axial vertebral patterning
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Altered apoptosis pattern during pharyngeal arch artery remodelling is associated with aortic arch malformations in Tgfbeta2 knock-out mice
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Abnormal angiogenesis but intact hematopoietic potential in TGF-beta type I receptor-deficient mice
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Inactivation of the beta-catenin gene by Wnt1-Cre-mediated deletion results in dramatic brain malformation and failure of craniofacial development
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Fate of the mammalian cardiac neural crest
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Neural crest cells contribute to normal aorticopulmonary septation
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Cardiac neural crest cells provide new insight into septation of the cardiac outflow tract: aortic sac to ventricular septal closure.
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GDF15/MIC-1 functions as a protective and antihypertrophic factor released from the myocardium in association with SMAD protein activation.
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27 November 2018
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Bmp6 and Bmp7 are required for cushion formation and septation in the developing mouse heart
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27 November 2018
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Cardiac arterial pole alignment is sensitive to FGF8 signaling in the pharynx
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reference URL
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12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1186/1471-213X-6-51
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
Dimensions Publication ID
1025002962
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PMCID
1635039
0 references
PubMed ID
17078885
1 reference
stated in
PubMed
PubMed ID
17078885
retrieved
24 January 2017
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