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AGO61-dependent GlcNAc modification primes the formation of functional glycans on α-dystroglycan
scientific journal article
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scholarly article
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
title
AGO61-dependent GlcNAc modification primes the formation of functional glycans on α-dystroglycan
(English)
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
main subject
Protein O-linked mannose beta 1,4-N-acetylglucosaminyltransferase 2
1 reference
stated in
GOA release 2020-03-11
author
Hirokazu Yagi
object named as
Hirokazu Yagi
series ordinal
1
0 references
Kay-Hooi Khoo
object named as
Kay-Hooi Khoo
series ordinal
8
0 references
Koichi Kato
object named as
Koichi Kato
series ordinal
10
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author name string
Naoki Nakagawa
series ordinal
2
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
Takuya Saito
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
Hiroshi Kiyonari
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
Takaya Abe
series ordinal
5
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
Tatsushi Toda
series ordinal
6
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
Sz-Wei Wu
series ordinal
7
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
Shogo Oka
series ordinal
9
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
language of work or name
English
0 references
publication date
21 November 2013
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
published in
Scientific Reports
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
volume
3
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
page(s)
3288
1 reference
stated in
PubMed
PubMed ID
24256719
retrieved
25 January 2017
copyright license
Creative Commons Attribution 3.0 Unported
start time
21 November 2013
1 reference
stated in
April 2022 Public Data File from Crossref
copyright status
copyrighted
0 references
cites work
SGK196 is a glycosylation-specific O-mannose kinase required for dystroglycan function
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Mutations in GDP-mannose pyrophosphorylase B cause congenital and limb-girdle muscular dystrophies associated with hypoglycosylation of α-dystroglycan
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Mutations in B3GALNT2 cause congenital muscular dystrophy and hypoglycosylation of α-dystroglycan
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Dystroglycan organizes axon guidance cue localization and axonal pathfinding
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Mutations in ISPD cause Walker-Warburg syndrome and defective glycosylation of α-dystroglycan
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
ISPD loss-of-function mutations disrupt dystroglycan O-mannosylation and cause Walker-Warburg syndrome
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Dystroglycan function requires xylosyl- and glucuronyltransferase activities of LARGE
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
POMT2 mutations cause alpha-dystroglycan hypoglycosylation and Walker-Warburg syndrome
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Demonstration of mammalian protein O-mannosyltransferase activity: coexpression of POMT1 and POMT2 required for enzymatic activity
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Mutations in the human LARGE gene cause MDC1D, a novel form of congenital muscular dystrophy with severe mental retardation and abnormal glycosylation of alpha-dystroglycan
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Mutations in the O-mannosyltransferase gene POMT1 give rise to the severe neuronal migration disorder Walker-Warburg syndrome
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Muscular dystrophy and neuronal migration disorder caused by mutations in a glycosyltransferase, POMGnT1
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Mutations in the fukutin-related protein gene (FKRP) cause a form of congenital muscular dystrophy with secondary laminin alpha2 deficiency and abnormal glycosylation of alpha-dystroglycan
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
An ancient retrotransposal insertion causes Fukuyama-type congenital muscular dystrophy
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Primary structure of dystrophin-associated glycoproteins linking dystrophin to the extracellular matrix
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
19 March 2017
Deciphering the glycosylome of dystroglycanopathies using haploid screens for lassa virus entry
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Adeno-associated viral-mediated LARGE gene therapy rescues the muscular dystrophic phenotype in mouse models of dystroglycanopathy
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Missense mutations in β-1,3-N-acetylglucosaminyltransferase 1 (B3GNT1) cause Walker-Warburg syndrome.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Identification of mutations in TMEM5 and ISPD as a cause of severe cobblestone lissencephaly
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Exome sequencing and functional validation in zebrafish identify GTDC2 mutations as a cause of Walker-Warburg syndrome
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Human natural killer-1 sulfotransferase (HNK-1ST)-induced sulfate transfer regulates laminin-binding glycans on α-dystroglycan
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Absence of post-phosphoryl modification in dystroglycanopathy mouse models and wild-type tissues expressing non-laminin binding form of α-dystroglycan
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Like-acetylglucosaminyltransferase (LARGE)-dependent modification of dystroglycan at Thr-317/319 is required for laminin binding and arenavirus infection.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
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29 September 2017
O-GlcNAc-specific antibody CTD110.6 cross-reacts with N-GlcNAc2-modified proteins induced under glucose deprivation
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
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29 September 2017
Distinct functions of glial and neuronal dystroglycan in the developing and adult mouse brain.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
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29 September 2017
HNK-1 epitope-carrying tenascin-C spliced variant regulates the proliferation of mouse embryonic neural stem cells.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
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29 September 2017
Site mapping and characterization of O-glycan structures on alpha-dystroglycan isolated from rabbit skeletal muscle.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
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29 September 2017
O-mannosyl phosphorylation of alpha-dystroglycan is required for laminin binding.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Muscular dystrophies due to glycosylation defects
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Residual laminin-binding activity and enhanced dystroglycan glycosylation by LARGE in novel model mice to dystroglycanopathy.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
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29 September 2017
Dystroglycan: from biosynthesis to pathogenesis of human disease.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
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29 September 2017
Dystrophin-glycoprotein complex: post-translational processing and dystroglycan function.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
29 September 2017
Effects of fukutin deficiency in the developing mouse brain
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3836086
retrieved
2 June 2018
HNK-1 sulfotransferase-dependent sulfation regulating laminin-binding glycans occurs in the post-phosphoryl moiety on α-dystroglycan.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSREP03288
retrieved
21 January 2018
Characterization of site-specific O-glycan structures within the mucin-like domain of alpha-dystroglycan from human skeletal muscle
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSREP03288
retrieved
21 January 2018
O-Mannose and O-N-acetyl galactosamine glycosylation of mammalian α-dystroglycan is conserved in a region-specific manner
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/24256719
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Biosynthesis of dystroglycan: processing of a precursor propeptide
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/24256719
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1038/SREP03288
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1509892
Dimensions Publication ID
1000108173
0 references
OpenCitations bibliographic resource ID
1509892
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1509892
PMCID
3836086
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1509892
PubMed ID
24256719
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1509892
ResearchGate publication ID
258822718
0 references
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