(Q28547015)

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Disruption of Transcriptional Coactivator Sub1 Leads to Genome-Wide Re-distribution of Clustered Mutations Induced by APOBEC in Active Yeast Genes

scientific article (publication date: May 2015)

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7 March 2020

Disruption of Transcriptional Coactivator Sub1 Leads to Genome-Wide Re-distribution of Clustered Mutations Induced by APOBEC in Active Yeast Genes (English)

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7 March 2020

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7 March 2020

Igor B. Rogozin

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7 March 2020

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Artem G Lada

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7 March 2020

Alok Dhar

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7 March 2020

Dmitrii E Polev

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7 March 2020

Alexey E Masharsky

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7 March 2020

Youri I Pavlov

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5 May 2015

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7 March 2020

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7 March 2020

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7 March 2020

e1005217

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7 March 2020

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7 March 2020

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5 May 2015

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Hypermutation in human cancer genomes: footprints and mechanisms

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Genome-wide mutation avalanches induced in diploid yeast cells by a base analog or an APOBEC deaminase

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AID/APOBEC cytosine deaminase induces genome-wide kataegis

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Expression of human AID in yeast induces mutations in context similar to the context of somatic hypermutation at G-C pairs in immunoglobulin genes

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AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversification

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C-terminal domain of transcription cofactor PC4 reveals dimeric ssDNA binding site

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Replication protein A: a heterotrimeric, single-stranded DNA-binding protein required for eukaryotic DNA metabolism

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29 September 2017

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29 September 2017

AID and APOBEC deaminases: balancing DNA damage in epigenetics and immunity

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The RNA editing enzyme APOBEC1 induces somatic mutations and a compatible mutational signature is present in esophageal adenocarcinomas.

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29 September 2017

Break-induced replication is a source of mutation clusters underlying kataegis

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Mechanisms of base substitution mutagenesis in cancer genomes

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Clustered and genome-wide transient mutagenesis in human cancers: Hypermutation without permanent mutators or loss of fitness

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DNA polymerases ζ and Rev1 mediate error-prone bypass of non-B DNA structures

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DNA sequences that interfere with transcription: implications for genome function and stability.

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29 September 2017

An APOBEC cytidine deaminase mutagenesis pattern is widespread in human cancers

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Evidence for APOBEC3B mutagenesis in multiple human cancers

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29 September 2017

DNA deaminases induce break-associated mutation showers with implication of APOBEC3B and 3A in breast cancer kataegis

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29 September 2017

APOBEC3B is an enzymatic source of mutation in breast cancer.

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29 September 2017

APOBEC3B can impair genomic stability by inducing base substitutions in genomic DNA in human cells.

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A role for the RNA pol II-associated PAF complex in AID-induced immune diversification

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Clustered mutations in yeast and in human cancers can arise from damaged long single-strand DNA regions.

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Transcription as a source of genome instability

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Replication protein A (RPA) hampers the processive action of APOBEC3G cytosine deaminase on single-stranded DNA

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29 September 2017

Guanine repeat-containing sequences confer transcription-dependent instability in an orientation-specific manner in yeast

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29 September 2017

Mutator effects and mutation signatures of editing deaminases produced in bacteria and yeast.

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Mutator phenotypes due to DNA replication infidelity.

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29 September 2017

Stochastic properties of processive cytidine DNA deaminases AID and APOBEC3G.

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29 September 2017

Transcriptional pausing and stalling causes multiple clustered mutations by human activation-induced deaminase

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29 September 2017

Transcription induces strand-specific mutations at the 5' end of human genes

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29 September 2017

Impact of phosphorylation and phosphorylation-null mutants on the activity and deamination specificity of activation-induced cytidine deaminase

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29 September 2017

Too many mutants with multiple mutations

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29 September 2017

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29 September 2017

Activation-induced cytidine deaminase action is strongly stimulated by mutations of the THO complex

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29 September 2017

Evolution and diversification of lamprey antigen receptors: evidence for involvement of an AID-APOBEC family cytosine deaminase

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29 September 2017

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29 September 2017

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29 September 2017

Clusters of mutations from transient hypermutability

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29 September 2017

Activation-induced cytidine deaminase (AID) can target both DNA strands when the DNA is supercoiled

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29 September 2017

The single-strand DNA binding activity of human PC4 prevents mutagenesis and killing by oxidative DNA damage

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29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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3 June 2018

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3 June 2018

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3 June 2018

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27 November 2018

Comparison of the differential context-dependence of DNA deamination by APOBEC enzymes: correlation with mutation spectra in vivo

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27 November 2018

Processive AID-catalysed cytosine deamination on single-stranded DNA simulates somatic hypermutation

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27 November 2018

Somatic hypermutation in the heavy chain locus correlates with transcription.

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27 November 2018

Identifiers

10.1371/JOURNAL.PGEN.1005217

2 references

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25941824%20AND%20SRC:MED&resulttype=core&format=json

7 March 2020

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2 references

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25941824%20AND%20SRC:MED&resulttype=core&format=json

7 March 2020

2 references

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25941824%20AND%20SRC:MED&resulttype=core&format=json

7 March 2020

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0 references

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