(Q28568098)

31 January 2017

title

Identification and characterisation of the novel amyloid-beta peptide-induced protein p17 (English)

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31 January 2017

Klaus Heese

3

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author name string

Saheen Nehar

series ordinal

1

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31 January 2017

Manisha Mishra

series ordinal

2

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31 January 2017

language of work or name

English

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publication date

6 October 2009

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31 January 2017

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31 January 2017

volume

583

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31 January 2017

issue

19

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31 January 2017

page(s)

3247–3253

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The last neuronal division: a unifying hypothesis for the pathogenesis of Alzheimer's disease.

31 January 2017

cites work

1 reference

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The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

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Cell biology of the amyloid beta-protein precursor and the mechanism of Alzheimer's disease

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Identification of a new synaptic vesicle protein 2B mRNA transcript which is up-regulated in neurons by amyloid beta peptide fragment (1-42).

21 January 2018

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Brain site-specific gene expression analysis in Alzheimer's disease patients

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Clonal cell lines from the rat central nervous system.

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Growth of a rat neuroblastoma cell line in serum-free supplemented medium

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Sequential treatment of SH-SY5Y cells with retinoic acid and brain-derived neurotrophic factor gives rise to fully differentiated, neurotrophic factor-dependent, human neuron-like cells

21 January 2018

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.09.018

Characterizing the new transcription regulator protein p60TRP

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Interleukin-6 and neural stem cells: more than gliogenesis

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Amyloid precursor protein overexpression depresses excitatory transmission through both presynaptic and postsynaptic mechanisms.

21 January 2018

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Beta-amyloid peptide at sublethal concentrations downregulates brain-derived neurotrophic factor functions in cultured cortical neurons.

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Brain-Derived Neurotrophic Factor/TrkB Signaling in Memory Processes

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Neuroprotection by brain-derived neurotrophic factor is mediated by extracellular signal-regulated kinase and phosphatidylinositol 3-kinase.

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Mono- and multisite phosphorylation enhances Bcl2's antiapoptotic function and inhibition of cell cycle entry functions

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Inactivation of Bcl-2 by phosphorylation

21 January 2018

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The protein kinase C family for neuronal signaling

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A cell biological perspective on Alzheimer's disease

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Bcl-2 Phosphorylation by p38 MAPK

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Intraneuronal -Amyloid Expression Downregulates the Akt Survival Pathway and Blunts the Stress Response

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GABAB receptor antagonists elevate both mRNA and protein levels of the neurotrophins nerve growth factor (NGF) and brain-derived neurotrophic factor (BDNF) but not neurotrophin-3 (NT-3) in brain and spinal cord of rats

21 January 2018

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Region-Specific Neurotrophin Imbalances in Alzheimer Disease

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BCL-2 is phosphorylated and inactivated by an ASK1/Jun N-terminal protein kinase pathway normally activated at G(2)/M.

21 January 2018

1 reference

24 May 2022

https://opencitations.net/index/coci/api/v1/citations/10.1128/MCB.19.12.8469

Identifiers

DOI

10.1016/J.FEBSLET.2009.09.018

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31 January 2017

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