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Evaluating cytochrome c diffusion in the intermembrane spaces of mitochondria during cytochrome c release
scientific journal article
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Statements
instance of
scholarly article
1 reference
stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
title
Evaluating cytochrome c diffusion in the intermembrane spaces of mitochondria during cytochrome c release
(English)
1 reference
stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
main subject
mitochondrion
1 reference
based on heuristic
inferred from title
author name string
Kieran Gillick
series ordinal
1
1 reference
stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
Martin Crompton
series ordinal
2
1 reference
stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
language of work or name
English
0 references
publication date
1 March 2008
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stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
published in
Journal of Cell Science
1 reference
stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
volume
121
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stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
issue
Pt 5
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stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
page(s)
618–626
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stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
cites work
The mitochondrial fission protein hFis1 requires the endoplasmic reticulum gateway to induce apoptosis
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PubMed Central
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19 March 2017
OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion
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19 March 2017
Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins
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PubMed Central
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19 March 2017
Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death
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PubMed Central
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19 March 2017
Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death
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PubMed Central
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19 March 2017
Endoplasmic reticulum BIK initiates DRP1-regulated remodelling of mitochondrial cristae during apoptosis.
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19 March 2017
Oligomeric Bax is a component of the putative cytochrome c release channel MAC, mitochondrial apoptosis-induced channel
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PubMed Central
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19 March 2017
Cyclophilin-D promotes the mitochondrial permeability transition but has opposite effects on apoptosis and necrosis
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PubMed Central
reference URL
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19 March 2017
Cell death: critical control points
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PubMed Central
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19 March 2017
tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
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19 March 2017
Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis
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19 March 2017
Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis
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19 March 2017
Movement of Bax from the cytosol to mitochondria during apoptosis
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19 March 2017
Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane
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PubMed Central
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7 April 2017
The mitochondrial permeability transition pore and its role in cell death
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PubMed Central
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7 April 2017
A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
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7 April 2017
Mitochondrial permeabilization relies on BH3 ligands engaging multiple prosurvival Bcl-2 relatives, not Bak.
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PubMed Central
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29 September 2017
Intersection between mitochondrial permeability pores and mitochondrial fusion/fission.
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29 September 2017
Mechanisms of cytochrome c release from mitochondria
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PubMed Central
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29 September 2017
Bid, a BH3-only multi-functional molecule, is at the cross road of life and death
1 reference
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PubMed Central
reference URL
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29 September 2017
Bid-cardiolipin interaction at mitochondrial contact site contributes to mitochondrial cristae reorganization and cytochrome C release
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
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29 September 2017
On the involvement of mitochondrial intermembrane junctional complexes in apoptosis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
29 September 2017
tBID Homooligomerizes in the mitochondrial membrane to induce apoptosis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
29 September 2017
Breaking the mitochondrial barrier
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
29 September 2017
BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
29 September 2017
Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
29 September 2017
Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
29 September 2017
Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
29 September 2017
BCL-2 Selectively Interacts with the BID-induced Open Conformer of BAK, Inhibiting BAK Auto-oligomerization
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
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3 June 2018
Biphasic translocation of Bax to mitochondria
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
3 June 2018
The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
3 June 2018
Cytochrome c as an electron shuttle between the outer and inner mitochondrial membranes.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
3 June 2018
Ca2+-dependent control of the permeability properties of the mitochondrial outer membrane and voltage-dependent anion-selective channel (VDAC).
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
27 November 2018
Activation of calcium-independent phospholipase A (iPLA) in brain mitochondria and release of apoptogenic factors by BAX and truncated BID.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
27 November 2018
tBid interaction with cardiolipin primarily orchestrates mitochondrial dysfunctions and subsequently activates Bax and Bak.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
27 November 2018
Bid activates multiple mitochondrial apoptotic mechanisms in primary hepatocytes after death receptor engagement
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
27 November 2018
Early changes in intramitochondrial cardiolipin distribution during apoptosis.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
27 November 2018
A large Ca2+-dependent channel formed by recombinant ADP/ATP carrier from Neurospora crassa resembles the mitochondrial permeability transition pore
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
27 November 2018
Mitochondria and neuronal glutamate excitotoxicity.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2316949
retrieved
27 November 2018
The course of etoposide-induced apoptosis in Jurkat cells lacking p53 and Bax
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/18252800
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Hierarchical regulation of mitochondrion-dependent apoptosis by BCL-2 subfamilies
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/18252800
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Bax and the mitochondrial permeability transition cooperate in the release of cytochrome c during endoplasmic reticulum-stress-induced apoptosis
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/18252800
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1242/JCS.021303
1 reference
stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
PMCID
2316949
0 references
PubMed ID
18252800
1 reference
stated in
PubMed
PubMed ID
18252800
retrieved
31 January 2017
ResearchGate publication ID
5599067
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