(Q28572647)

31 January 2017

title

Contribution of myocardin in the hypoxia-induced phenotypic switching of rat pulmonary arterial smooth muscle cells (English)

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31 January 2017

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Wei Jie

1

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31 January 2017

Junli Guo

2

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31 January 2017

Zhihua Shen

3

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31 January 2017

Xiaoyan Wang

4

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31 January 2017

Shaojiang Zheng

5

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31 January 2017

Guoping Wang

6

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31 January 2017

Qilin Ao

7

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31 January 2017

publication date

1 December 2010

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Experimental and Molecular Pathology

31 January 2017

published in

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31 January 2017

volume

89

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31 January 2017

issue

3

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31 January 2017

page(s)

301–306

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Time-dependent phenotypic and contractile changes of pulmonary artery in chronic hypoxia-induced pulmonary hypertension

31 January 2017

cites work

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PKC activates BKCa channels in rat pulmonary arterial smooth muscle via cGMP-dependent protein kinase

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Platelet-derived growth factor-BB and Ets-1 transcription factor negatively regulate transcription of multiple smooth muscle cell differentiation marker genes

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Smooth muscle cells in transplant atherosclerotic lesions are originated from recipients, but not bone marrow progenitor cells

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Platelet-derived growth factor-BB-mediated activation of Akt suppresses smooth muscle-specific gene expression through inhibition of mitogen-activated protein kinase and redistribution of serum response factor

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Control of phenotypic plasticity of smooth muscle cells by bone morphogenetic protein signaling through the myocardin-related transcription factors

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The serum response factor coactivator myocardin is required for vascular smooth muscle development

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S-nitrosothiols signal hypoxia-mimetic vascular pathology

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Role of 12-lipoxygenase in hypoxia-induced rat pulmonary artery smooth muscle cell proliferation

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Forced expression of myocardin is not sufficient for induction of smooth muscle differentiation in multipotential embryonic cells

7 January 2021

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Modulation of pulmonary vascular smooth muscle cell phenotype in hypoxia: role of cGMP-dependent protein kinase.

7 January 2021

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Modulation of pulmonary vascular smooth muscle cell phenotype in hypoxia: role of cGMP-dependent protein kinase and myocardin

7 January 2021

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Transdifferentiation of pulmonary arteriolar endothelial cells into smooth muscle-like cells regulated by myocardin involved in hypoxia-induced pulmonary vascular remodelling.

7 January 2021

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7 January 2021

Identifiers

DOI

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31 January 2017

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