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Inhibition of glycogen synthase kinase 3[beta] activity with lithium in vitro attenuates sepsis-induced changes in muscle protein turnover
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scholarly article
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PubMed
PubMed ID
20926980
retrieved
31 January 2017
title
Inhibition of glycogen synthase kinase 3[beta] activity with lithium in vitro attenuates sepsis-induced changes in muscle protein turnover
(English)
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PubMed
PubMed ID
20926980
retrieved
31 January 2017
main subject
sepsis
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author name string
Stephen Bertsch
series ordinal
1
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stated in
PubMed
PubMed ID
20926980
retrieved
31 January 2017
Charles H. Lang
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2
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PubMed
PubMed ID
20926980
retrieved
31 January 2017
Thomas C. Vary
series ordinal
3
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stated in
PubMed
PubMed ID
20926980
retrieved
31 January 2017
publication date
1 March 2011
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PubMed
PubMed ID
20926980
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31 January 2017
published in
Shock
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PubMed
PubMed ID
20926980
retrieved
31 January 2017
volume
35
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stated in
PubMed
PubMed ID
20926980
retrieved
31 January 2017
issue
3
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stated in
PubMed
PubMed ID
20926980
retrieved
31 January 2017
page(s)
266–274
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PubMed
PubMed ID
20926980
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31 January 2017
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Europe PubMed Central
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11 June 2022
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3602924/fullTextXML
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Local insulin-like growth factor I prevents sepsis-induced muscle atrophy
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29 September 2017
Endotoxin and interferon-gamma inhibit translation in skeletal muscle cells by stimulating nitric oxide synthase activity
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When apoptosis meets autophagy: deciding cell fate after trauma and sepsis
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Acute alcohol intoxication increases atrogin-1 and MuRF1 mRNA without increasing proteolysis in skeletal muscle
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Regulation of muscle protein synthesis during sepsis and inflammation
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Protein kinase B/Akt: a nexus of growth factor and cytokine signaling in determining muscle mass
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29 September 2017
Role of glycogen synthase kinase-3 in insulin resistance and type 2 diabetes
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eIF2B, a mediator of general and gene-specific translational control
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Pharmacological inhibitors of glycogen synthase kinase 3.
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29 September 2017
Lithium stabilizes the CCAAT/enhancer-binding protein alpha (C/EBPalpha) through a glycogen synthase kinase 3 (GSK3)-independent pathway involving direct inhibition of proteasomal activity
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29 September 2017
GSK-3beta negatively regulates skeletal myotube hypertrophy
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Glycogen synthase kinase-3beta is a negative regulator of cardiomyocyte hypertrophy
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Muscle wasting in a rat model of long-lasting sepsis results from the activation of lysosomal, Ca2+ -activated, and ubiquitin-proteasome proteolytic pathways.
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PubMed Central
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Insulin resistance accelerates muscle protein degradation: Activation of the ubiquitin-proteasome pathway by defects in muscle cell signaling
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PubMed Central
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3 June 2018
GSK-3beta inhibitors reduce protein degradation in muscles from septic rats and in dexamethasone-treated myotubes
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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3 June 2018
Toll-like receptor-mediated cytokine production is differentially regulated by glycogen synthase kinase 3.
1 reference
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PubMed Central
reference URL
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3 June 2018
Glycogen synthase kinase-3 is the predominant insulin-regulated eukaryotic initiation factor 2B kinase in skeletal muscle.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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3 June 2018
Glycogen synthase kinase-3 is rapidly inactivated in response to insulin and phosphorylates eukaryotic initiation factor eIF-2B.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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3 June 2018
Sepsis-induced changes in protein synthesis: differential effects on fast- and slow-twitch muscles
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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3 June 2018
Branched-chain amino acids protect against dexamethasone-induced soleus muscle atrophy in rats.
1 reference
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PubMed Central
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27 November 2018
A potential role for Akt/FOXO signalling in both protein loss and the impairment of muscle carbohydrate oxidation during sepsis in rodent skeletal muscle.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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27 November 2018
IFN-gamma synergizes with LPS to induce nitric oxide biosynthesis through glycogen synthase kinase-3-inhibited IL-10.
1 reference
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PubMed Central
reference URL
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27 November 2018
Oxidative stress-induced insulin resistance in rat skeletal muscle: role of glycogen synthase kinase-3.
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PubMed Central
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27 November 2018
Inhibiting glycogen synthase kinase 3beta in sepsis.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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27 November 2018
GSK-3beta inhibitors attenuate the organ injury/dysfunction caused by endotoxemia in the rat.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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27 November 2018
Protein breakdown in muscle from burned rats is blocked by insulin-like growth factor i and glycogen synthase kinase-3beta inhibitors
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602924
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27 November 2018
Influence of sepsis in rats on muscle protein turnover in vivo and in tissue incubated under different in vitro conditions
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/20926980
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Regulation of eukaryotic initiation factor-2 expression during sepsis
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/20926980
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Differential regulation of skeletal muscle protein turnover by insulin and IGF-I after bacteremia
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/20926980
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Diminished ERK 1/2 and p38 MAPK phosphorylation in skeletal muscle during sepsis
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/20926980
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Does leucine, leucyl-tRNA, or some metabolite of leucine regulate protein synthesis and degradation in skeletal and cardiac muscle?
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/20926980
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
IGF-I/IGFBP-3 binary complex modulates sepsis-induced inhibition of protein synthesis in skeletal muscle
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/20926980
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Phosphorylation of eukaryotic initiation factor eIF2Bepsilon in skeletal muscle during sepsis
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/20926980
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1097/SHK.0B013E3181FD068C
1 reference
stated in
PubMed
PubMed ID
20926980
retrieved
31 January 2017
Fatcat ID
release_tjou2lihh5avbiaqtfo4aixut4
1 reference
stated in
Fatcat
reference URL
https://api.fatcat.wiki/v0/release/tjou2lihh5avbiaqtfo4aixut4
retrieved
24 November 2022
based on heuristic
mapped directly with Wikidata item
PMCID
3602924
0 references
PubMed ID
20926980
1 reference
stated in
PubMed
PubMed ID
20926980
retrieved
31 January 2017
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