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Pseudo-DNA damage response in senescent cells
scientific journal article
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scholarly article
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
title
Pseudo-DNA damage response in senescent cells
(English)
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
author
Mikhail Blagosklonny
series ordinal
6
object named as
Mikhail V. Blagosklonny
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
Zoya N. Demidenko
series ordinal
2
object named as
Zoya N. Demidenko
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
author name string
Tatyana V. Pospelova
series ordinal
1
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
Elena I. Bukreeva
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
Valery A. Pospelov
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
Andrei V. Gudkov
series ordinal
5
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
publication date
15 December 2009
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
published in
Cell Cycle
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
volume
8
1 reference
stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
issue
24
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stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
page(s)
4112–4118
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stated in
PubMed
PubMed ID
19946210
retrieved
31 January 2017
describes a project that uses
ImageJ
1 reference
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Europe PubMed Central
retrieved
11 June 2022
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/PMC4970747/fullTextXML
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inferred from PubMed Central ID database lookup
cites work
MicroRNAs miR-146a/b negatively modulate the senescence-associated inflammatory mediators IL-6 and IL-8
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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17 March 2017
Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network
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stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
17 March 2017
DNA damage-dependent acetylation and ubiquitination of H2AX enhances chromatin dynamics
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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17 March 2017
Nitrogen oxide-releasing aspirin induces histone H2AX phosphorylation, ATM activation and apoptosis preferentially in S-phase cells: involvement of reactive oxygen species
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
17 March 2017
Focusing on foci: H2AX and the recruitment of DNA-damage response factors
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
17 March 2017
Genomic instability in mice lacking histone H2AX
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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17 March 2017
Cellular senescence: when bad things happen to good cells
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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7 April 2017
Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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7 April 2017
Telomere-dependent and telomere-independent origins of endogenous DNA damage in tumor cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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29 September 2017
Cellular quiescence caused by the Mdm2 inhibitor nutlin-3A.
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stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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29 September 2017
DNA damage foci in mitosis are devoid of 53BP1.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
mTOR mediates Wnt-induced epidermal stem cell exhaustion and aging.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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29 September 2017
Bacterial intoxication evokes cellular senescence with persistent DNA damage and cytokine signalling
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
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29 September 2017
Pharmacologic inhibition of MEK and PI-3K converges on the mTOR/S6 pathway to decelerate cellular senescence
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Rapamycin decelerates cellular senescence
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
The heat-induced gamma-H2AX response does not play a role in hyperthermic cell killing
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Limited role of murine ATM in oncogene-induced senescence and p53-dependent tumor suppression.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Aging: ROS or TOR.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Growth stimulation leads to cellular senescence when the cell cycle is blocked
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Control of senescence by CXCR2 and its ligands
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
C-MYC overexpression is required for continuous suppression of oncogene-induced senescence in melanoma cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Activation of the cellular DNA damage response in the absence of DNA lesions
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Modulation of the activities of AMP-activated protein kinase, protein kinase B, and mammalian target of rapamycin by limiting energy availability with 2-deoxyglucose
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
gamma-H2AX in bystander cells: not just a radiation-triggered event, a cellular response to stress mediated by intercellular communication
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Activation of the kinase activity of ATM by retinoic acid is required for CREB-dependent differentiation of neuroblastoma cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Hyperthermia activates a subset of ataxia-telangiectasia mutated effectors independent of DNA strand breaks and heat shock protein 70 status
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
2-deoxy-D-glucose reduces the level of constitutive activation of ATM and phosphorylation of histone H2AX.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Phosphorylation of histone H2AX at M phase in human cells without DNA damage response
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
The thorny path linking cellular senescence to organismal aging
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
ATM-dependent DNA damage-independent mitotic phosphorylation of H2AX in normally growing mammalian cells.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Full activation of PKB/Akt in response to insulin or ionizing radiation is mediated through ATM.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
PI 3-kinase related kinases: 'big' players in stress-induced signaling pathways
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Mitogen stimulation cooperates with telomere shortening to activate DNA damage responses and senescence signaling
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
H2AX may function as an anchor to hold broken chromosomal DNA ends in close proximity
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Identification of a novel protein kinase mediating Akt survival signaling to the ATM protein
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
p21Waf1/Cip1/Sdi1-induced growth arrest is associated with depletion of mitosis-control proteins and leads to abnormal mitosis and endoreduplication in recovering cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
29 September 2017
Constitutive histone H2AX phosphorylation and ATM activation are strongly amplified during mitogenic stimulation of lymphocytes
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
3 June 2018
Distribution of p53 binding protein 1 (53BP1) and phosphorylated H2A.X during mouse preimplantation development in the absence of DNA damage
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
27 November 2018
Aging-suppressants: cellular senescence (hyperactivation) and its pharmacologic deceleration.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
27 November 2018
Activation of DNA damage response signaling in mouse embryonic stem cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
27 November 2018
Phosphorylation of histone H2AX on Ser 139 and activation of ATM during oxidative burst in phorbol ester-treated human leukocytes.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
27 November 2018
G1/S arrest induced by histone deacetylase inhibitor sodium butyrate in E1A + Ras-transformed cells is mediated through down-regulation of E2F activity and stabilization of beta-catenin.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
27 November 2018
Living with DNA breaks is an everyday reality for cells adapted to high NaCl.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4970747
retrieved
27 November 2018
A senescence-like phenotype distinguishes tumor cells that undergo terminal proliferation arrest after exposure to anticancer agents
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/19946210
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Loss of p21 CDKN1A impairs entry to quiescence and activates a DNA damage response in normal fibroblasts induced to quiescence
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/19946210
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
DNA lesions and DNA damage response: even long lasting relationships need a "break"
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/19946210
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.4161/CC.8.24.10215
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4628297
OpenCitations bibliographic resource ID
4628297
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4628297
PMCID
4970747
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4628297
PubMed ID
19946210
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4628297
ResearchGate publication ID
40039346
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