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Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk
scientific journal article
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instance of
scholarly article
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
title
Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk
(English)
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
main subject
Interleukin 1 receptor-like 2
1 reference
stated in
GOA release 2020-03-11
keratinocyte
1 reference
based on heuristic
inferred from title
author
Sabine Werner
series ordinal
8
object named as
Sabine Werner
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
Jan Kisielow
object named as
Jan Kisielow
series ordinal
9
0 references
Manfred Kopf
object named as
Manfred Kopf
series ordinal
10
0 references
Jean-Christophe Renauld
1 reference
stated in
ORCID Public Data File 2021
Anthony J Coyle
series ordinal
6
object named as
Anthony J. Coyle
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
author name string
Luigi Tortola
series ordinal
1
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
Esther Rosenwald
series ordinal
2
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
Brian Abel
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
Hal Blumberg
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
Matthias Schäfer
series ordinal
5
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
Jean-Christoph Renauld
series ordinal
7
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
language of work or name
English
0 references
publication date
1 November 2012
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
published in
Journal of Clinical Investigation
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
volume
122
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
issue
11
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
page(s)
3965–3976
1 reference
stated in
PubMed
PubMed ID
23064362
retrieved
31 January 2017
cites work
Interleukin-36 (IL-36) ligands require processing for full agonist (IL-36α, IL-36β, and IL-36γ) or antagonist (IL-36Ra) activity
1 reference
stated in
PubMed Central
reference URL
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17 March 2017
IL-36R ligands are potent regulators of dendritic and T cells
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PubMed Central
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17 March 2017
Distinct roles of IL-23 and IL-17 in the development of psoriasis-like lesions in a mouse model
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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17 March 2017
IL-1F5, -F6, -F8, and -F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression
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PubMed Central
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17 March 2017
An autoinflammatory disease with deficiency of the interleukin-1-receptor antagonist
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17 March 2017
Genome-wide scan reveals association of psoriasis with IL-23 and NF-kappaB pathways
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17 March 2017
SCART scavenger receptors identify a novel subset of adult gammadelta T cells
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17 March 2017
Efficacy and safety of ustekinumab, a human interleukin-12/23 monoclonal antibody, in patients with psoriasis: 76-week results from a randomised, double-blind, placebo-controlled trial (PHOENIX 1)
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PubMed Central
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17 March 2017
Opposing activities of two novel members of the IL-1 ligand family regulate skin inflammation
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PubMed Central
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17 March 2017
A large-scale genetic association study confirms IL12B and leads to the identification of IL23R as psoriasis-risk genes
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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17 March 2017
Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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17 March 2017
Interleukin (IL)-1F6, IL-1F8, and IL-1F9 signal through IL-1Rrp2 and IL-1RAcP to activate the pathway leading to NF-kappaB and MAPKs
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PubMed Central
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17 March 2017
Two novel IL-1 family members, IL-1 delta and IL-1 epsilon, function as an antagonist and agonist of NF-kappa B activation through the orphan IL-1 receptor-related protein 2
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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17 March 2017
Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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7 April 2017
Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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7 April 2017
Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Imiquimod clears tumors in mice independent of adaptive immunity by converting pDCs into tumor-killing effector cells
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Pivotal role of dermal IL-17-producing γδ T cells in skin inflammation
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Interleukin-36-receptor antagonist deficiency and generalized pustular psoriasis
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Mutations in IL36RN/IL1F5 are associated with the severe episodic inflammatory skin disease known as generalized pustular psoriasis.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Cutting edge: Identification of a motile IL-17-producing gammadelta T cell population in the dermis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
CCL20, γδ T cells, and IL-22 in corneal epithelial healing
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Cutting edge: A critical functional role for IL-23 in psoriasis.
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Averting inflammation by targeting the cytokine environment
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PubMed Central
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29 September 2017
The IL-1 family: regulators of immunity
1 reference
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PubMed Central
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29 September 2017
Skin immune sentinels in health and disease
1 reference
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
An autoinflammatory disease due to homozygous deletion of the IL1RN locus
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PubMed Central
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29 September 2017
Critical regulation of early Th17 cell differentiation by interleukin-1 signaling
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PubMed Central
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29 September 2017
Cutting edge: NKT cells constitutively express IL-23 receptor and RORgammat and rapidly produce IL-17 upon receptor ligation in an IL-6-independent fashion
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
IL-22 is required for Th17 cell-mediated pathology in a mouse model of psoriasis-like skin inflammation
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Pathogenesis and therapy of psoriasis
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
A human interleukin-12/23 monoclonal antibody for the treatment of psoriasis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2-dependent mechanisms with implications for psoriasis pathogenesis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
29 September 2017
Latent TGFbeta1 overexpression in keratinocytes results in a severe psoriasis-like skin disorder
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Spontaneous development of psoriasis in a new animal model shows an essential role for resident T cells and tumor necrosis factor-alpha
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Resolution of psoriasis upon blockade of IL-15 biological activity in a xenograft mouse model
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Absence of IL-1 signaling and reduced inflammatory response in IL-1 type I receptor-deficient mice
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Inflammatory skin disease in transgenic mice that express high levels of interleukin 1 alpha in basal epidermis
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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29 September 2017
Generalized exacerbation of psoriasis associated with imiquimod cream treatment of superficial basal cell carcinomas
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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3 June 2018
Psoriasis induced by topical imiquimod
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
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3 June 2018
IL-23 production by cosecretion of endogenous p19 and transgenic p40 in keratin 14/p40 transgenic mice: evidence for enhanced cutaneous immunity
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
3 June 2018
IL-22 is required for imiquimod-induced psoriasiform skin inflammation in mice
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Expression of interleukin (IL)-1 family members upon stimulation with IL-17 differs in keratinocytes derived from patients with psoriasis and healthy donors.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Cutting edge: crucial role of IL-1 and IL-23 in the innate IL-17 response of peripheral lymph node NK1.1- invariant NKT cells to bacteria.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
IL-1RL2 and its ligands contribute to the cytokine network in psoriasis.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Interleukin-1 and IL-23 induce innate IL-17 production from gammadelta T cells, amplifying Th17 responses and autoimmunity.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Interleukin-17-producing gammadelta T cells selectively expand in response to pathogen products and environmental signals
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Psoriasis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Imiquimod-induced psoriasis-like skin inflammation in mice is mediated via the IL-23/IL-17 axis.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
In vitro and in situ expression of IL-23 by keratinocytes in healthy skin and psoriasis lesions: enhanced expression in psoriatic skin.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Psoriasis-like cutaneous inflammation in mice lacking interleukin-1 receptor antagonist.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Antigen-specific T cell sensitization is impaired in IL-17-deficient mice, causing suppression of allergic cellular and humoral responses
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3484446
retrieved
27 November 2018
Inter-regulation of Th17 cytokines and the IL-36 cytokines in vitro and in vivo: implications in psoriasis pathogenesis
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/23064362
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
c-Jun and JunB antagonistically control cytokine-regulated mesenchymal-epidermal interaction in skin
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/23064362
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Human keratinocytes express functional Toll-like receptor 3, 4, 5, and 9
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/23064362
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
IL-22 is expressed by Th17 cells in an IL-23-dependent fashion, but not required for the development of autoimmune encephalomyelitis
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/23064362
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1172/JCI63451
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2320532
OpenCitations bibliographic resource ID
2320532
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2320532
PMCID
3484446
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2320532
PubMed ID
23064362
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2320532
ResearchGate publication ID
232247030
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