(Q28587940)
Statements
A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner (English)
Siamak Rassadi
Arjun Krishnaswamy
Brigitte Pié
Russell McConnell
Ellis Cooper
Michele H Jacob
1 September 2005
25
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