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mir-17-92 cluster is required for and sufficient to induce cardiomyocyte proliferation in postnatal and adult hearts
scientific journal article
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scholarly article
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
title
mir-17-92 cluster is required for and sufficient to induce cardiomyocyte proliferation in postnatal and adult hearts
(English)
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
main subject
regulation of DNA nucleotidylexotransferase activity
1 reference
stated in
Gene Ontology release 2020-05-02
Gene Ontology ID
GO:1904939
regulation of cardiac ventricle formation
1 reference
stated in
Gene Ontology release 2020-05-02
Gene Ontology ID
GO:1904942
positive regulation of DNA nucleotidylexotransferase activity
1 reference
stated in
Gene Ontology release 2020-05-02
Gene Ontology ID
GO:1904941
positive regulation of cardiac ventricle formation
1 reference
stated in
Gene Ontology release 2020-05-02
Gene Ontology ID
GO:1904944
negative regulation of DNA nucleotidylexotransferase activity
1 reference
stated in
Gene Ontology release 2020-05-02
Gene Ontology ID
GO:1904940
negative regulation of cardiac ventricle formation
1 reference
stated in
Gene Ontology release 2020-05-02
Gene Ontology ID
GO:1904943
Phosphatase and tensin homolog
1 reference
stated in
GOA release 2020-03-11
author
Zhanpeng Huang
series ordinal
2
object named as
Zhan-Peng Huang
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Zhiqiang Lin
object named as
Zhiqiang Lin
series ordinal
9
1 reference
stated in
ORCID Public Data File 2021
author name string
Jinghai Chen
series ordinal
1
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Hee Young Seok
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Jian Ding
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Masaharu Kataoka
series ordinal
5
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Zheng Zhang
series ordinal
6
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Xiaoyun Hu
series ordinal
7
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Gang Wang
series ordinal
8
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Si Wang
series ordinal
10
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Willam T. Pu
series ordinal
11
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Ronglih Liao
series ordinal
12
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stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
Da-Zhi Wang
series ordinal
13
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stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
publication date
7 June 2013
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
published in
Circulation Research
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
volume
112
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stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
issue
12
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stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
page(s)
1557–1566
1 reference
stated in
PubMed
PubMed ID
23575307
retrieved
1 February 2017
cites work
Germline deletion of the miR-17∼92 cluster causes skeletal and growth defects in humans
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PubMed Central
reference URL
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miR-17~92 cooperates with RB pathway mutations to promote retinoblastoma
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miR-19 is a key oncogenic component of mir-17-92
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PubMed Central
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MicroRNA MiR-17 retards tissue growth and represses fibronectin expression
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17 March 2017
Neuregulin1/ErbB4 signaling induces cardiomyocyte proliferation and repair of heart injury
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MicroRNA-92a controls angiogenesis and functional recovery of ischemic tissues in mice
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miRiad roles for the miR-17-92 cluster in development and disease
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PubMed Central
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17 March 2017
Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters
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17 March 2017
Lymphoproliferative disease and autoimmunity in mice with increased miR-17-92 expression in lymphocytes
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PubMed Central
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17 March 2017
Expression of microRNAs is dynamically regulated during cardiomyocyte hypertrophy
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PubMed Central
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17 March 2017
A microRNA expression signature of human solid tumors defines cancer gene targets
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c-Myc-regulated microRNAs modulate E2F1 expression
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7 April 2017
Transient regenerative potential of the neonatal mouse heart
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7 April 2017
Heart regeneration in zebrafish
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29 September 2017
The miR-17-92 microRNA cluster regulates multiple components of the TGF-β pathway in neuroblastoma
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29 September 2017
Zebrafish heart regeneration occurs by cardiomyocyte dedifferentiation and proliferation
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29 September 2017
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29 September 2017
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29 September 2017
Cardiomyocyte cell cycle activation improves cardiac function after myocardial infarction
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29 September 2017
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29 September 2017
Cardiac regeneration: repopulating the heart
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PubMed Central
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29 September 2017
Regulation of myocardial contractility and cell size by distinct PI3K-PTEN signaling pathways
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29 September 2017
Cyclin D1 overexpression promotes cardiomyocyte DNA synthesis and multinucleation in transgenic mice
1 reference
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PubMed Central
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29 September 2017
A dynamic epicardial injury response supports progenitor cell activity during zebrafish heart regeneration.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3756475
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27 November 2018
Targeted expression of cyclin D2 results in cardiomyocyte DNA synthesis and infarct regression in transgenic mice
1 reference
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PubMed Central
reference URL
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retrieved
27 November 2018
Identifiers
DOI
10.1161/CIRCRESAHA.112.300658
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1449885
OpenCitations bibliographic resource ID
1449885
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1449885
PMCID
3756475
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1449885
PubMed ID
23575307
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1449885
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