(Q30471428)

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Neurofibrillary lesions in experimental aluminum-induced encephalopathy and Alzheimer's disease share immunoreactivity for amyloid precursor protein, A beta, alpha 1-antichymotrypsin and ubiquitin-protein conjugates

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Europe PubMed Central

PubMed publication ID

21 June 2017

Neurofibrillary lesions in experimental aluminum-induced encephalopathy and Alzheimer's disease share immunoreactivity for amyloid precursor protein, A beta, alpha 1-antichymotrypsin and ubiquitin-protein conjugates (English)

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PubMed publication ID

21 June 2017

Alzheimer's disease

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Christos D Katsetos

4

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Katsetos CD

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PubMed publication ID

21 June 2017

author name string

Huang Y

1

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Europe PubMed Central

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21 June 2017

Herman MM

2

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Europe PubMed Central

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21 June 2017

Liu J

3

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Europe PubMed Central

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21 June 2017

Wills MR

5

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21 June 2017

Savory J

6

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21 June 2017

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publication date

1 October 1997

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21 June 2017

number of pages

8

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21 June 2017

771

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21 June 2017

2

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21 June 2017

213-220

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Europe PubMed Central

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21 June 2017

Alpha 1-antichymotrypsin is associated solely with amyloid deposits containing the beta-protein. Amyloid and cell localization of alpha 1-antichymotrypsin.

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Expression patterns of beta-amyloid precursor protein (beta-APP) in neural and nonneural human tissues from Alzheimer's disease and control subjects

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A long-term intravenous model of aluminum maltol toxicity in rabbits: tissue distribution, hepatic, renal, and neuronal cytoskeletal changes associated with systemic exposure

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Lysosomal hydrolases of different classes are abnormally distributed in brains of patients with Alzheimer disease

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Ubiquitin-protein conjugates in Alzheimer's lesions

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Expression of beta amyloid protein precursor mRNAs: recognition of a novel alternatively spliced form and quantitation in Alzheimer's disease using PCR.

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The ubiquitin system for protein degradation

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Protein determination using bicinchoninic acid in the presence of sulfhydryl reagents

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A comparative study of the peroxidase-antiperoxidase method and an avidin-biotin complex method for studying polypeptide hormones with radioimmunoassay antibodies

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Aging and cellular maturation cause changes in ubiquitin-eye lens protein conjugates

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The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor

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Neuronal cytoskeletal lesions induced in the CNS by intraventricular and intravenous aluminium maltol in rabbits

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Precursor of amyloid protein in Alzheimer disease undergoes fast anterograde axonal transport

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Ubiquitin is a component of neurofibrillary tangles in a variety of neurodegenerative diseases

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Ubiquitin is a common factor in intermediate filament inclusion bodies of diverse type in man, including those of Parkinson's disease, Pick's disease, and Alzheimer's disease, as well as Rosenthal fibres in cerebellar astrocytomas, cytoplasmic bodie

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Ubiquitin in neurodegenerative diseases

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Ubiquitin and the molecular pathology of chronic degenerative diseases

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Inherited Neurodegenerative Diseases and Transgenic Models

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Distribution pattern and functional state of alpha 1-antichymotrypsin in plaques and vascular amyloid in Alzheimer's disease. A immunohistochemical study with monoclonal antibodies against native and inactivated alpha 1-antichymotrypsin

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Partial reversal of aluminium-induced neurofibrillary degeneration by desferrioxamine in adult male rabbits

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Tau immunoreactivity associated with aluminum maltolate-induced neurofibrillary degeneration in rabbits

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Quantitative image analysis of temporal changes in tau and neurofilament proteins during the course of acute experimental neurofibrillary degeneration; non-phosphorylated epitopes precede phosphorylation

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Markers of axonal injury in post mortem human brain

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Expression of beta-amyloid precursor protein in reactive astrocytes following neuronal damage

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Induction of Alzheimer-like beta-amyloid immunoreactivity in the brains of rabbits with dietary cholesterol

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Protease inhibitor domain encoded by an amyloid protein precursor mRNA associated with Alzheimer's disease

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Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications

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Amyloid precursor protein in the cerebral cortex is rapidly and persistently induced by loss of subcortical innervation

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Spectrum of morphological appearance of amyloid deposits in Alzheimer's disease

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Ultrastructural studies of the cells forming amyloid fibers in classical plaques.

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10.1016/S0006-8993(97)00780-4

1 reference

Europe PubMed Central

PubMed publication ID

21 June 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

21 June 2017

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