(Q31111749)

28 June 2017

Pseudophosphorylation and glycation of tau protein enhance but do not trigger fibrillization in vitro (English)

1 reference

28 June 2017

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Mihaela Necula

1

1 reference

28 June 2017

Jeff Kuret

series ordinal

2

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28 June 2017

language of work or name

English

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publication date

10 September 2004

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Journal of Biological Chemistry

28 June 2017

10

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based on heuristic

inferred from page(s)

published in

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28 June 2017

279

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28 June 2017

49694-49703

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28 June 2017

48

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Functions and malfunctions of the tau proteins

28 June 2017

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Tau protein and the neurofibrillary pathology of Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Staging the pathological assembly of truncated tau protein into paired helical filaments in Alzheimer's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Deamidation and isoaspartate formation in smeared tau in paired helical filaments. Unusual properties of the microtubule-binding domain of tau.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Phosphate analysis and dephosphorylation of modified tau associated with paired helical filaments

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

New phosphorylation sites identified in hyperphosphorylated tau (paired helical filament-tau) from Alzheimer's disease brain using nanoelectrospray mass spectrometry

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Proline-directed and non-proline-directed phosphorylation of PHF-tau.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Biopsy-derived adult human brain tau is phosphorylated at many of the same sites as Alzheimer's disease paired helical filament tau.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Detection of phosphorylated Ser262 in fetal tau, adult tau, and paired helical filament tau.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Specific tau phosphorylation sites correlate with severity of neuronal cytopathology in Alzheimer's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Abnormal tau phosphorylation at Ser396 in Alzheimer's disease recapitulates development and contributes to reduced microtubule binding.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Phosphorylation of Ser262 strongly reduces binding of tau to microtubules: distinction between PHF-like immunoreactivity and microtubule binding.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Casein kinase 1 delta phosphorylates tau and disrupts its binding to microtubules

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Abnormal phosphorylation of tau and the mechanism of Alzheimer neurofibrillary degeneration: sequestration of microtubule-associated proteins 1 and 2 and the disassembly of microtubules by the abnormal tau

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Phosphorylation that detaches tau protein from microtubules (Ser262, Ser214) also protects it against aggregation into Alzheimer paired helical filaments

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Structural and functional implications of tau hyperphosphorylation: information from phosphorylation-mimicking mutated tau proteins.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Phosphorylation-mimicking glutamate clusters in the proline-rich region are sufficient to simulate the functional deficiencies of hyperphosphorylated tau protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

The in vitro formation of recombinant tau polymers: effect of phosphorylation and glycation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Advanced Maillard reaction end products are associated with Alzheimer disease pathology

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Advanced glycation end products contribute to amyloidosis in Alzheimer disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Ligand-dependent tau filament formation: implications for Alzheimer's disease progression.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Differential assembly of human tau isoforms in the presence of arachidonic acid

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Anionic Micelles and Vesicles Induce Tau Fibrillization in Vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

The structural basis of monoclonal antibody Alz50's selectivity for Alzheimer's disease pathology.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

A static laser light scattering assay for surfactant-induced tau fibrillization

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Oxidative regulation of fatty acid-induced tau polymerization.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

The endogenous and cell cycle-dependent phosphorylation of tau protein in living cells: implications for Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Electron microscopy as a quantitative method for investigating tau fibrillization

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

In vitro polymerization of tau protein monitored by laser light scattering: method and application to the study of FTDP-17 mutants

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Assembly of tau protein into Alzheimer paired helical filaments depends on a local sequence motif ((306)VQIVYK(311)) forming beta structure

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

The Gompertz Curve as a Growth Curve

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Apolipoprotein E is a kinetic but not a thermodynamic inhibitor of amyloid formation: implications for the pathogenesis and treatment of Alzheimer disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Evidence for an intermediate in tau filament formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Models of amyloid seeding in Alzheimer's disease and scrapie: mechanistic truths and physiological consequences of the time-dependent solubility of amyloid proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Rapid Anionic Micelle-mediated α-Synuclein Fibrillization in Vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Alzheimer's disease hyperphosphorylated tau sequesters normal tau into tangles of filaments and disassembles microtubules.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Maillard reaction products in tissue proteins: new products and new perspectives

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Accumulation of abnormally phosphorylated tau precedes the formation of neurofibrillary tangles in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

The extent of phosphorylation of fetal tau is comparable to that of PHF-tau from Alzheimer paired helical filaments

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Aberrant tau phosphorylation by glycogen synthase kinase-3beta and JNK3 induces oligomeric tau fibrils in COS-7 cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

A nucleated assembly mechanism of Alzheimer paired helical filaments

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Promotion of hyperphosphorylation by frontotemporal dementia tau mutations

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Assembly of Alzheimer-like filaments from full-length tau protein.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Rapid assembly of Alzheimer-like paired helical filaments from microtubule-associated protein tau monitored by fluorescence in solution.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Assembly of microtubule-associated protein tau into Alzheimer-like filaments induced by sulphated glycosaminoglycans.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Alzheimer's disease: paired helical filaments and cytomembranes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Models for the length distributions of actin filaments: I. Simple polymerization and fragmentation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Effects of jasplakinolide on the kinetics of actin polymerization. An explanation for certain in vivo observations

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

A sequence of cytoskeleton changes related to the formation of neurofibrillary tangles and neuropil threads.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

Tau protein isoforms, phosphorylation and role in neurodegenerative disorders11These authors contributed equally to this work

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405527200

21 January 2018

Identifiers

DOI

10.1074/JBC.M405527200

1 reference

release_6g5sehw7cfdojmezaoruzr7ya4

28 June 2017

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/6g5sehw7cfdojmezaoruzr7ya4

mapped directly with Wikidata item

24 November 2022

based on heuristic

1 reference