(Q31431906)

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Differential cellular regulation of the mitochondrial permeability transition in an in vitro model of 1,3-dinitrobenzene-induced encephalopathy

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Europe PubMed Central

PubMed publication ID

2 July 2017

Differential cellular regulation of the mitochondrial permeability transition in an in vitro model of 1,3-dinitrobenzene-induced encephalopathy (English)

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

author name string

R B Tjalkens

1

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

M M Ewing

2

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

M A Philbert

3

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

language of work or name

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publication date

1 August 2000

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Europe PubMed Central

PubMed publication ID

2 July 2017

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

874

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Europe PubMed Central

PubMed publication ID

2 July 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

165-177

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

1 reference

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The mitochondrial permeability transition

1 reference

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The mitochondrial permeability transition is required for tumor necrosis factor alpha-mediated apoptosis and cytochrome c release

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Selective vulnerability in acute energy deprivation syndromes

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The Mitochondrial Permeability Transition Pore is Modulated by Oxidative Agents Through Both Pyridine Nucleotides and Glutathione at Two Separate Sites

1 reference

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Modulation of the mitochondrial permeability transition pore by pyridine nucleotides and dithiol oxidation at two separate sites.

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Kinetic evidence for a heart mitochondrial pore activated by Ca2+, inorganic phosphate and oxidative stress. A potential mechanism for mitochondrial dysfunction during cellular Ca2+ overload

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Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

1 reference

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Differences in the activation of the mitochondrial permeability transition among brain regions in the rat correlate with selective vulnerability

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Cyclosporin A, but not FK 506, protects mitochondria and neurons against hypoglycemic damage and implicates the mitochondrial permeability transition in cell death.

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Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

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BCL-2 family members and the mitochondria in apoptosis

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The Ca2+ transport mechanisms of mitochondria and Ca2+ uptake from physiological-type Ca2+ transients

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Oxidative stress, thiol reagents, and membrane potential modulate the mitochondrial permeability transition by affecting nucleotide binding to the adenine nucleotide translocase

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Bak BH3 peptides antagonize Bcl-xL function and induce apoptosis through cytochrome c-independent activation of caspases

1 reference

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Increasing or decreasing nervous activity modulates the severity of the glio-vascular lesions of 1,3-dinitrobenzene in the rat: effects of the tremorgenic pyrethroid, Bifenthrin, and of anaesthesia.

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Regulation of Bcl-2 protein expression in human neuroblastoma SH-SY5Y cells: positive and negative effects of protein kinases C and A, respectively.

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1,3-Dinitrobenzene metabolism and toxicity in seminiferous tubules isolated from rats of different ages.

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Mitochondrial manganese superoxide dismutase prevents neural apoptosis and reduces ischemic brain injury: suppression of peroxynitrite production, lipid peroxidation, and mitochondrial dysfunction.

1 reference

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Role for Bcl-xL as an inhibitor of cytosolic cytochrome C accumulation in DNA damage-induced apoptosis

1 reference

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Pivotal role of mitochondrial calcium uptake in neural cell apoptosis and necrosis

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Role of phosphate and other proton-donating anions in respiration-coupled transport of Ca2+ by mitochondria

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Regulation of Ca 2+ release from mitochondria by the oxidation-reduction state of pyridine nucleotides

1 reference

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7 January 2021

Bcl-2 and Bax interactions in mitochondria probed with green fluorescent protein and fluorescence resonance energy transfer

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Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

1 reference

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Endogenous bax translocation in SH-SY5Y human neuroblastoma cells and cerebellar granule neurons undergoing apoptosis

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Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

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Bcl-2 potentiates the maximal calcium uptake capacity of neural cell mitochondria

1 reference

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Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

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Mitochondrial permeability transition in hepatocytes induced by t-BuOOH: NAD(P)H and reactive oxygen species

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Contribution of the mitochondrial permeability transition to lethal injury after exposure of hepatocytes to t-butylhydroperoxide

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The binding properties and biological activities of Bcl-2 and Bax in cells exposed to apoptotic stimuli

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Protective effect of trifluoperazine on the mitochondrial damage induced by Ca2+ plus prooxidants.

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1,3-Dinitrobenzene-induced encephalopathy in rats

1 reference

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The mitochondrial permeability transition mediates both necrotic and apoptotic death of hepatocytes exposed to Br-A23187.

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Increased oxidative metabolism and oxidative stress in m-dinitrobenzene neurotoxicity

1 reference

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Bcl-2 family proteins

1 reference

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Early metabolic changes during m-dinitrobenzene neurotoxicity and the possible role of oxidative stress

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Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

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Molecular characterization of mitochondrial apoptosis-inducing factor

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Bcl-xL prevents cell death following growth factor withdrawal by facilitating mitochondrial ATP/ADP exchange

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The role of reactive oxygen species in mitochondrial permeability transition

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Transfection of C6 glioma cells with the bax gene and increased sensitivity to treatment with cytosine arabinoside

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Investigation of the bicinchoninic acid protein assay: identification of the groups responsible for color formation

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Studies on the neurotoxicity of 6,7-dihydroxy-1-methyl-1,2,3,4-tetrahydroisoquinoline (salsolinol) in SH-SY5Y cells.

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Lysophosphatidic acid induces necrosis and apoptosis in hippocampal neurons

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based on heuristic

inferred from DOI database lookup

Mitochondrial permeability transition in the central nervous system: induction by calcium cycling-dependent and -independent pathways

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based on heuristic

inferred from DOI database lookup

Identifiers

10.1016/S0006-8993(00)02546-4

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 July 2017

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