(Q33313348)

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Neuroprotective properties of gallic acid from Sanguisorbae radix on amyloid beta protein (25--35)-induced toxicity in cultured rat cortical neurons

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scholarly article

1 reference

Europe PubMed Central

25 July 2017

Neuroprotective properties of gallic acid from Sanguisorbae radix on amyloid beta protein (25--35)-induced toxicity in cultured rat cortical neurons (English)

1 reference

Europe PubMed Central

25 July 2017

Sanguisorba officinalis

1 reference

1 reference

author name string

Ju Yeon Ban

1

1 reference

Europe PubMed Central

25 July 2017

Ha Thi Thuy Nguyen

2

1 reference

Europe PubMed Central

25 July 2017

Hee-Ju Lee

3

1 reference

Europe PubMed Central

25 July 2017

Soon Ock Cho

4

1 reference

Europe PubMed Central

25 July 2017

Hyun Soo Ju

5

1 reference

Europe PubMed Central

25 July 2017

Ju Yeon Kim

6

1 reference

Europe PubMed Central

25 July 2017

Kihwan Bae

7

1 reference

Europe PubMed Central

25 July 2017

Kyung-Sik Song

8

1 reference

Europe PubMed Central

25 July 2017

Yeon Hee Seong

9

1 reference

Europe PubMed Central

25 July 2017

publication date

2008

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16 September 2022

full work available at URL

https://www.jstage.jst.go.jp/article/bpb/31/1/31_1_149/_pdf

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19 March 2024

https://api.unpaywall.org/v2/10.1248/bpb.31.149?email=unpaywall_01@example.com

Biological and Pharmaceutical Bulletin

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Europe PubMed Central

25 July 2017

31

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Europe PubMed Central

25 July 2017

149-153

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Europe PubMed Central

25 July 2017

1

1 reference

Europe PubMed Central

25 July 2017

online access status

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19 March 2024

https://api.unpaywall.org/v2/10.1248/bpb.31.149?email=unpaywall_01@example.com

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Selective induction of cell death in cancer cells by gallic acid.

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Neurotoxic Abeta peptides increase oxidative stress in vivo through NMDA-receptor and nitric-oxide-synthase mechanisms, and inhibit complex IV activity and induce a mitochondrial permeability transition in vitro

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Catechin and epicatechin from Smilacis chinae rhizome protect cultured rat cortical neurons against amyloid beta protein (25-35)-induced neurotoxicity through inhibition of cytosolic calcium elevation

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Activation of the L voltage-sensitive calcium channel by mitogen-activated protein (MAP) kinase following exposure of neuronal cells to beta-amyloid. MAP kinase mediates beta-amyloid-induced neurodegeneration.

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Antioxidant, gallic acid, induces apoptosis in HL-60RG cells

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Blockade of 5-HT(3) receptor with MDL 72222 and Y 25130 reduces beta-amyloid protein (25--35)-induced neurotoxicity in cultured rat cortical neurons

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Structure-activity relationship analysis of antioxidant ability and neuroprotective effect of gallic acid derivatives

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Ca2+-Dependent caspase activation by gallic acid derivatives

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Different generation of inhibitors against gallic acid-induced apoptosis produces different sensitivity to gallic acid

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Pomolic acid derivatives from the root of Sanguisorba officinalis

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Beta-amyloid, neuronal death and Alzheimer's disease

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Black raspberry extract and fractions contain angiogenesis inhibitors.

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Amyloid beta-peptide-associated free radical oxidative stress, neurotoxicity, and Alzheimer's disease.

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Signal transduction during amyloid-beta-peptide neurotoxicity: role in Alzheimer disease

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Evidence that amyloid beta-peptide-induced lipid peroxidation and its sequelae in Alzheimer's disease brain contribute to neuronal death

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Protective effect of Terminalia belerica Roxb. and gallic acid against carbon tetrachloride induced damage in albino rats.

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Beta-amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage

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Role of reactive oxygen species in gallic acid-induced apoptosis

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Gallic acid inhibits ribonucleotide reductase and cyclooxygenases in human HL-60 promyelocytic leukemia cells.

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Calcium signals induced by amyloid beta peptide and their consequences in neurons and astrocytes in culture

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In vitro and in vivo oxidative stress associated with Alzheimer's amyloid beta-peptide (1-42)

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Beta-amyloid peptide fragment 31-35 induces apoptosis in cultured cortical neurons.

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Identifiers

10.1248/BPB.31.149

1 reference

Europe PubMed Central

25 July 2017

1 reference

Europe PubMed Central

25 July 2017

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