(Q60530540)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

title

Beta-amyloid-induced calcium influx induces apoptosis in culture by oxidative stress rather than tau phosphorylation (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

1 reference

1 reference

F J Ekinci

1

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

M D Linsley

series ordinal

2

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

T B Shea

series ordinal

3

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

publication date

1 March 2000

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

published in

Molecular Brain Research

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

volume

76

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

page(s)

389-395

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

issue

2

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

Apoptosis of retrogradely degenerating neurons occurs in association with the accumulation of perikaryal mitochondria and oxidative damage to the nucleus

5 November 2019

cites work

1 reference

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7 January 2021

Abnormal phosphorylation of tau and the mechanism of Alzheimer neurofibrillary degeneration: sequestration of microtubule-associated proteins 1 and 2 and the disassembly of microtubules by the abnormal tau

1 reference

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7 January 2021

beta-Amyloid peptide fragment 25-35 potentiates the calcium-dependent release of excitatory amino acids from depolarized hippocampal slices.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Accumulation of abnormally phosphorylated tau precedes the formation of neurofibrillary tangles in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Vitamin E protects nerve cells from amyloid beta protein toxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Hydrogen peroxide mediates amyloid beta protein toxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Do defects in mitochondrial energy metabolism underlie the pathology of neurodegenerative diseases?

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

beta-amyloid fibrils induce tau phosphorylation and loss of microtubule binding.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Hyperphosphorylation of Tau and filopodial retraction following microinjection of protein kinase C catalytic subunits

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

beta-Amyloid neurotoxicity: a discussion of in vitro findings

1 reference

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7 January 2021

Activation of the L voltage-sensitive calcium channel by mitogen-activated protein (MAP) kinase following exposure of neuronal cells to beta-amyloid. MAP kinase mediates beta-amyloid-induced neurodegeneration.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Apoptosis mediated neurotoxicity induced by chronic application of beta amyloid fragment 25-35.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Catecholamines potentiate amyloid beta-peptide neurotoxicity: involvement of oxidative stress, mitochondrial dysfunction, and perturbed calcium homeostasis.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Alzheimer's presenilin mutation sensitizes neural cells to apoptosis induced by trophic factor withdrawal and amyloid beta-peptide: involvement of calcium and oxyradicals.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Neurodegeneration mediated by glutamate and beta-amyloid peptide: a comparison and possible interaction

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

beta-Amyloid protein amplifies calcium signalling in central neurons from the adult mouse.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Defective brain microtubule assembly in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Calpain-mediated proteolysis of microtubule-associated protein 2 (MAP-2) is inhibited by phosphorylation by cAMP-dependent protein kinase, but not by Ca2+/calmodulin-dependent protein kinase II.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Beta-amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Calcium influx and the Ca2+-calmodulin complex are involved in interferon-gamma-induced expression of HLA class II molecules on HL-60 cells

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

NMDA-dependent superoxide production and neurotoxicity.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Beta/A4-evoked degeneration of differentiated SH-SY5Y human neuroblastoma cells

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Phosphorylation, calpain proteolysis and tubulin binding of recombinant human tau isoforms.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Tau in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Calcium-destabilizing and neurodegenerative effects of aggregated beta-amyloid peptide are attenuated by basic FGF.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Neurons may live for decades with neurofibrillary tangles.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Calcium/calmodulin-dependent protein kinase IIalpha mediates activation of mitogen-activated protein kinase and cytosolic phospholipase A2 in norepinephrine-induced arachidonic acid release in rabbit aortic smooth muscle cells

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

RNA oxidation is a prominent feature of vulnerable neurons in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

3-Hydroxykynurenine, an endogenous oxidative stress generator, causes neuronal cell death with apoptotic features and region selectivity.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Binding of amyloid beta-peptide to mitochondrial hydroxyacyl-CoA dehydrogenase (ERAB): regulation of an SDR enzyme activity with implications for apoptosis in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Differences between vascular and plaque core amyloid in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Apoptotic death in an in vitro model of neuronal oxidative stress.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Increased surface phosphatidylserine is an early marker of neuronal apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Voltage-dependent potentiation of L-type Ca2+ channels due to phosphorylation by cAMP-dependent protein kinase

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Isolation and quantification of soluble Alzheimer's beta-peptide from biological fluids

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Calcium influx recruits an additional class of kinases to hyperphosphorylate tau

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Calcium influx into human neuroblastoma cells induces ALZ-50 immunoreactivity: involvement of calpain-mediated hydrolysis of protein kinase C

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Beta-amyloid and ionophore A23187 evoke tau hyperphosphorylation by distinct intracellular pathways: differential involvement of the calpain/protein kinase C system

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Oxidative stress in neurodegenerative diseases

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Amyloid-β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative Stress

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

The free radical antioxidant vitamin E protects cortical synaptosomal membranes from amyloid beta-peptide(25-35) toxicity but not from hydroxynonenal toxicity: relevance to the free radical hypothesis of Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Superoxide free radical and intracellular calcium mediate A beta(1-42) induced endothelial toxicity.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Oxidative stress induces a form of programmed cell death with characteristics of both apoptosis and necrosis in neuronal cells.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

cAMP activates MAP kinase and Elk-1 through a B-Raf- and Rap1-dependent pathway

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Neurotoxicity of A beta amyloid protein in vitro is not altered by calcium channel blockade

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

The endoplasmic reticulum stress-responsive protein GRP78 protects neurons against excitotoxicity and apoptosis: suppression of oxidative stress and stabilization of calcium homeostasis.

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

Hyperphosphorylated tau in SY5Y cells: similarities and dissimilarities to abnormally hyperphosphorylated tau from Alzheimer disease brain

1 reference

https://api.crossref.org/works/10.1016%2FS0169-328X%2800%2900025-5

7 January 2021

10.1016/S0169-328X(00)00025-5

Identifiers

DOI

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

1 reference