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Neurodegeneration: new clues on inclusions
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instance of
scholarly article
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
review article
1 reference
stated in
Europe PubMed Central
title
Neurodegeneration: new clues on inclusions
(English)
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
main subject
neurodegeneration
1 reference
based on heuristic
inferred from title
author name string
Lansbury PT Jr
series ordinal
1
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
Kosik KS
series ordinal
2
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
publication date
1 January 2000
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
published in
Chemistry and Biology
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
volume
7
1 reference
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Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
issue
1
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
page(s)
R9-R12
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Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
cites work
Translating cell biology into therapeutic advances in Alzheimer's disease
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
retrieved
7 January 2021
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inferred from DOI database lookup
Structural neurology: are seeds at the root of neuronal degeneration?
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
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Tau mutations in frontotemporal dementia
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
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inferred from DOI database lookup
A stop-codon mutation in the BRI gene associated with familial British dementia
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
based on heuristic
inferred from DOI database lookup
A human peptidyl-prolyl isomerase essential for regulation of mitosis
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
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Structural and functional analysis of the mitotic rotamase Pin1 suggests substrate recognition is phosphorylation dependent
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
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Sequence-specific and phosphorylation-dependent proline isomerization: a potential mitotic regulatory mechanism
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
based on heuristic
inferred from DOI database lookup
The essential mitotic peptidyl-prolyl isomerase Pin1 binds and regulates mitosis-specific phosphoproteins
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
based on heuristic
inferred from DOI database lookup
The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
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inferred from DOI database lookup
Mitotic mechanisms in Alzheimer's disease?
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Aberrant expression of mitotic cdc2/cyclin B1 kinase in degenerating neurons of Alzheimer's disease brain.
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
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inferred from DOI database lookup
A conformation- and phosphorylation-dependent antibody recognizing the paired helical filaments of Alzheimer's disease.
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
based on heuristic
inferred from DOI database lookup
Cytoskeletal pathology in familial cerebral amyloid angiopathy (British type) with non-neuritic amyloid plaque formation.
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
based on heuristic
inferred from DOI database lookup
Familial cerebral amyloid angiopathy (British type) with nonneuritic amyloid plaque formation may be due to a novel amyloid protein
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
based on heuristic
inferred from DOI database lookup
Alzheimer's disease and Down's syndrome: Sharing of a unique cerebrovascular amyloid fibril protein
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
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7 January 2021
based on heuristic
inferred from DOI database lookup
C-terminal fragments of alpha- and beta-tubulin form amyloid fibrils in vitro and associate with amyloid deposits of familial cerebral amyloid angiopathy, British type
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1074-5521%2800%2900068-5
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Identifiers
DOI
10.1016/S1074-5521(00)00068-5
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
PubMed ID
10662685
1 reference
stated in
Europe PubMed Central
PubMed ID
10662685
retrieved
29 July 2017
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