(Q34253716)

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Natural resistance to ascorbic acid induced oxidative stress is mainly mediated by catalase activity in human cancer cells and catalase-silencing sensitizes to oxidative stress

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Natural resistance to ascorbic acid induced oxidative stress is mainly mediated by catalase activity in human cancer cells and catalase-silencing sensitizes to oxidative stress (English)

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

oxidative stress

0 references

Ulrike Kämmerer

2

object named as

Ulrike Kämmerer

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

author name string

Christoph Klingelhoeffer

1

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Monika Koospal

3

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Bettina Mühling

4

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Manuela Schneider

5

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Michaela Kapp

6

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Alexander Kübler

7

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Christoph-Thomas Germer

8

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Christoph Otto

9

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

language of work or name

1 reference

based on heuristic

inferred from title

publication date

2 May 2012

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

BMC Complementary and Alternative Medicine

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

12

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

61

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

copyright license

Creative Commons Attribution 2.0 Generic

2 May 2012

1 reference

April 2022 Public Data File from Crossref

copyright status

1 reference

April 2022 Public Data File from Crossref

Runx2-mediated bcl-2 gene expression contributes to nitric oxide protection against hydrogen peroxide-induced osteoblast apoptosis

1 reference

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Therapeutic strategies by modulating oxygen stress in cancer and inflammation

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1 reference

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1 reference

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1 reference

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1 reference

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The emerging role of reactive oxygen species in cancer therapy

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Iron supplements: the quick fix with long-term consequences

1 reference

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Reactive species mechanisms of cellular hypoxia-reoxygenation injury

1 reference

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6 July 2018

Antioxidant enzyme levels in cancer

1 reference

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6 July 2018

Hydrogen peroxide mediates the killing of U937 tumor cells elicited by pharmacologically attainable concentrations of ascorbic acid: cell death prevention by extracellular catalase or catalase from cocultured erythrocytes or fibroblasts.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3404974

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Decrease in catalase activity and loss of the 11p chromosome arm in the course of SV40 transformation of human fibroblasts.

1 reference

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High-dose vitamin C versus placebo in the treatment of patients with advanced cancer who have had no prior chemotherapy. A randomized double-blind comparison

1 reference

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Prooxidant states and tumor promotion

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3404974

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Antioxidant and xenobiotic-metabolizing enzyme gene expression in doxorubicin-resistant MCF-7 breast cancer cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3404974

6 July 2018

Production of large amounts of hydrogen peroxide by human tumor cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3404974

6 July 2018

Supplemental ascorbate in the supportive treatment of cancer: Prolongation of survival times in terminal human cancer

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3404974

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Failure of high-dose vitamin C (ascorbic acid) therapy to benefit patients with advanced cancer. A controlled trial

1 reference

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Supplemental ascorbate in the supportive treatment of cancer: Reevaluation of prolongation of survival times in terminal human cancer

1 reference

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ERK promotes hydrogen peroxide-induced apoptosis through caspase-3 activation and inhibition of Akt in renal epithelial cells.

1 reference

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Vitamin C preferential toxicity for malignant melanoma cells

1 reference

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Oxidative stress therapy for solid tumors - a proposal

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

https://pubmed.ncbi.nlm.nih.gov/22551313

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Downregulation of catalase by reactive oxygen species via hypermethylation of CpG island II on the catalase promoter

1 reference

https://pubmed.ncbi.nlm.nih.gov/22551313

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based on heuristic

inferred from PubMed ID database lookup

Growth suppression of human leukemic cells in vitro by L-ascorbic acid

1 reference

https://pubmed.ncbi.nlm.nih.gov/22551313

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

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1 reference

https://pubmed.ncbi.nlm.nih.gov/22551313

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based on heuristic

inferred from PubMed ID database lookup

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1 reference

https://pubmed.ncbi.nlm.nih.gov/22551313

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cytotoxic effect of ascorbate and its derivatives on cultured malignant and nonmalignant cell lines

1 reference

https://pubmed.ncbi.nlm.nih.gov/22551313

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1186/1472-6882-12-61

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Dimensions Publication ID

0 references

PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

ResearchGate publication ID

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