(Q34511210)

2 August 2017

title

Involvement of CD95-independent caspase 8 activation in arsenic trioxide-induced apoptosis (English)

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2 August 2017

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Kitamura K

1

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2 August 2017

Minami Y

2

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2 August 2017

Yamamoto K

3

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2 August 2017

Akao Y

4

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2 August 2017

Kiyoi H

5

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2 August 2017

Saito H

6

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2 August 2017

Naoe T

7

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2 August 2017

publication date

1 October 2000

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2 August 2017

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2 August 2017

volume

14

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2 August 2017

issue

10

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2 August 2017

page(s)

1743-1750

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describes a project that uses

2 August 2017

NB4/As

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cites work

Complete remission after treatment of acute promyelocytic leukemia with arsenic trioxide

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenic trioxide as an inducer of apoptosis and loss of PML/RAR alpha protein in acute promyelocytic leukemia cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Toxic effects of arsenic (As3+) and other metal ions on acute promyelocytic leukemia cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

The PML-RARα fusion mRNA generated by the t(15;17) translocation in acute promyelocytic leukemia encodes a functionally altered RAR

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Chromosomal translocation t(15;17) in human acute promyelocytic leukemia fuses RARα with a novel putative transcription factor, PML

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Retinoic acid, but not arsenic trioxide, degrades the PLZF/RARalpha fusion protein, without inducing terminal differentiation or apoptosis, in a RA-therapy resistant t(11;17)(q23;q21) APL patient

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenic-induced PML targeting onto nuclear bodies: implications for the treatment of acute promyelocytic leukemia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Conjugation with the ubiquitin-related modifier SUMO-1 regulates the partitioning of PML within the nucleus

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

PIC-1/SUMO-1-modified PML-retinoic acid receptor alpha mediates arsenic trioxide-induced apoptosis in acute promyelocytic leukemia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenic and apoptosis in the treatment of acute promyelocytic leukemia

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Retinoic acid and arsenic synergize to eradicate leukemic cells in a mouse model of acute promyelocytic leukemia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenic induces apoptosis in B-cell leukaemic cell lines in vitro: activation of caspases and down-regulation of Bcl-2 protein

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

The induction of apoptosis and cell cycle arrest by arsenic trioxide in lymphoid neoplasms

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Apoptosis and growth inhibition in malignant lymphocytes after treatment with arsenic trioxide at clinically achievable concentrations

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenic trioxide induces apoptosis in neuroblastoma cell lines through the activation of caspase 3 in vitro

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenite induces apoptosis via a direct effect on the mitochondrial permeability transition pore

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenic trioxide-induced apoptosis and differentiation are associated respectively with mitochondrial transmembrane potential collapse and retinoic acid signaling pathways in acute promyelocytic leukemia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

BCL-2 family members and the mitochondria in apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

The role of caspases in development, immunity, and apoptotic signal transduction: lessons from knockout mice.

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Mitochondrial permeability transition is a central coordinating event of apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Collapse of the inner mitochondrial transmembrane potential is not required for apoptosis of HL60 cells.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Mutant AF-2 domain of PML-RARalpha in retinoic acid-resistant NB4 cells: differentiation induced by RA is triggered directly through PML-RARalpha and its down-regulation in acute promyelocytic leukemia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Reactions of arsenic(III) and arsenic(V) species with glutathione

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Interactions of rat red blood cell sulfhydryls with arsenate and arsenite

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Tumor promoter arsenite activates extracellular signal-regulated kinase through a signaling pathway mediated by epidermal growth factor receptor and Shc.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Signal transduction pathways regulated by arsenate and arsenite

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Mitochondrial control of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Mitochondria and apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Arsenic trioxide sensitivity is associated with low level of glutathione in cancer cells

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Involvement of reactive oxygen species and caspase 3 activation in arsenite-induced apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

The anti-cancer drug etoposide can induce caspase-8 processing and apoptosis in the absence of CD95 receptor-ligand interaction

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Fas ligand-independent, FADD-mediated activation of the Fas death pathway by anticancer drugs.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

The tyrosine kinase lck is required for CD95-independent caspase-8 activation and apoptosis in response to ionizing radiation.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2401900

7 January 2021

Identifiers

DOI

10.1038/SJ.LEU.2401900

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Dimensions Publication ID

2 August 2017

1028484710

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1 reference