(Q34681297)

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Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS)

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scholarly article

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Europe PubMed Central

3 August 2017

Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS) (English)

1 reference

Europe PubMed Central

3 August 2017

1 reference

based on heuristic

inferred from title

reactive oxygen species

1 reference

based on heuristic

inferred from title

0 references

Daniel L. Kastner

8

object named as

Daniel L Kastner

1 reference

Europe PubMed Central

3 August 2017

5

object named as

Heiyoung Park

1 reference

Europe PubMed Central

3 August 2017

author name string

Ariel C Bulua

1

1 reference

Europe PubMed Central

3 August 2017

Anna Simon

2

1 reference

Europe PubMed Central

3 August 2017

Ravikanth Maddipati

3

1 reference

Europe PubMed Central

3 August 2017

Martin Pelletier

4

1 reference

Europe PubMed Central

3 August 2017

Kye-Young Kim

6

1 reference

Europe PubMed Central

3 August 2017

Michael N Sack

7

1 reference

Europe PubMed Central

3 August 2017

Richard M Siegel

9

1 reference

Europe PubMed Central

3 August 2017

language of work or name

0 references

publication date

31 January 2011

1 reference

Europe PubMed Central

3 August 2017

Journal of Experimental Medicine

1 reference

Europe PubMed Central

3 August 2017

208

1 reference

Europe PubMed Central

3 August 2017

3

1 reference

Europe PubMed Central

3 August 2017

519-533

1 reference

Europe PubMed Central

3 August 2017

A role for mitochondria in NLRP3 inflammasome activation

1 reference

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Essential regulation of cell bioenergetics by constitutive InsP3 receptor Ca2+ transfer to mitochondria.

1 reference

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7 July 2018

Inflammasome activation in NADPH oxidase defective mononuclear phagocytes from patients with chronic granulomatous disease

1 reference

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7 July 2018

Concerted action of wild-type and mutant TNF receptors enhances inflammation in TNF receptor 1-associated periodic fever syndrome

1 reference

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Altered redox state of monocytes from cryopyrin-associated periodic syndromes causes accelerated IL-1beta secretion

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Therapeutic use of coenzyme Q10 and coenzyme Q10-related compounds and formulations.

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TLR activation of the transcription factor XBP1 regulates innate immune responses in macrophages.

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HVCN1 modulates BCR signal strength via regulation of BCR-dependent generation of reactive oxygen species

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Reactive oxygen species-independent activation of the IL-1beta inflammasome in cells from patients with chronic granulomatous disease

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Thioredoxin-interacting protein links oxidative stress to inflammasome activation

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Mitochondrial uncoupling protein 2 inhibits mast cell activation and reduces histamine content

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7 July 2018

Riboflavin kinase couples TNF receptor 1 to NADPH oxidase

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Regulation of superoxide dismutase genes: implications in disease

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Role of increased ROS dissipation in prevention of T1D.

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7 July 2018

The p53-p66shc-Manganese Superoxide Dismutase (MnSOD) network: a mitochondrial intrigue to generate reactive oxygen species.

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Autoimmune thyroiditis and ROS.

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Redox signalling and the inflammatory response in rheumatoid arthritis.

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Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

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Mutation of the Cyba gene encoding p22phox causes vestibular and immune defects in mice

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7 July 2018

Ero1 and redox homeostasis in the endoplasmic reticulum.

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Activation of the NALP3 inflammasome is triggered by low intracellular potassium concentration

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ATP activates a reactive oxygen species-dependent oxidative stress response and secretion of proinflammatory cytokines in macrophages

1 reference

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7 July 2018

Targeting antioxidants to mitochondria by conjugation to lipophilic cations.

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Neonatal-onset multisystem inflammatory disease responsive to interleukin-1beta inhibition

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Hereditary auto-inflammatory disorders and biologics

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7 July 2018

Abnormal disulfide-linked oligomerization results in ER retention and altered signaling by TNFR1 mutants in TNFR1-associated periodic fever syndrome (TRAPS)

1 reference

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Tumour necrosis factor receptor 1 mediates endoplasmic reticulum stress-induced activation of the MAP kinase JNK

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Clinical significance of P46L and R92Q substitutions in the tumour necrosis factor superfamily 1A gene

1 reference

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Neutrophils and immunity: challenges and opportunities

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Dynamic regulation of pro- and anti-inflammatory cytokines by MAPK phosphatase 1 (MKP-1) in innate immune responses

1 reference

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Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

1 reference

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Mitochondria, oxidants, and aging

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Prevention of cold-associated acute inflammation in familial cold autoinflammatory syndrome by interleukin-1 receptor antagonist

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Mutant forms of tumour necrosis factor receptor I that occur in TNF-receptor-associated periodic syndrome retain signalling functions but show abnormal behaviour.

1 reference

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Enhanced autoimmunity, arthritis, and encephalomyelitis in mice with a reduced oxidative burst due to a mutation in the Ncf1 gene

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Infevers: an evolving mutation database for auto-inflammatory syndromes

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NOX enzymes and the biology of reactive oxygen

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Specificity of a third kind: reactive oxygen and nitrogen intermediates in cell signaling

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Hydrogen peroxide as second messenger in lymphocyte activation

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The TNF receptor-associated periodic syndrome (TRAPS): emerging concepts of an autoinflammatory disorder

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Reversible oxidation and inactivation of protein tyrosine phosphatases in vivo

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Diphenyleneiodonium, an NAD(P)H oxidase inhibitor, also potently inhibits mitochondrial reactive oxygen species production

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Reactive oxygen species rapidly increase endothelial ICAM-1 ability to bind neutrophils without detectable upregulation

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10.1084/JEM.20102049

1 reference

Europe PubMed Central

3 August 2017

1 reference

Europe PubMed Central

3 August 2017

1 reference

Europe PubMed Central

3 August 2017

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