(Q34712305)

4 August 2017

Polyglutamine domain modulates the TBP-TFIIB interaction: implications for its normal function and neurodegeneration (English)

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4 August 2017

Meyer J Friedman

1

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4 August 2017

Anjali G Shah

2

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4 August 2017

Zhi-Hui Fang

3

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4 August 2017

Elizabeth G Ward

4

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4 August 2017

Stephen T Warren

5

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4 August 2017

Shihua Li

6

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4 August 2017

Xiao-Jiang Li

7

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4 August 2017

language of work or name

English

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publication date

11 November 2007

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4 August 2017

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4 August 2017

10

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4 August 2017

1519-1528

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4 August 2017

12

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https://scigraph.springernature.com/pub.10.1038/nn2011

4 August 2017

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cites work

Analysis of Sp1 in vivo reveals multiple transcriptional domains, including a novel glutamine-rich activation motif

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7 January 2021

Transcriptional activation modulated by homopolymeric glutamine and proline stretches

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7 January 2021

Transcription of eukaryotic protein-coding genes

1 reference

7 January 2021

Early vertebrate evolution of the TATA-binding protein, TBP

1 reference

7 January 2021

Behavioral Disorder, Dementia, Ataxia, and Rigidity in a Large Family With TATA Box-Binding Protein Mutation

1 reference

7 January 2021

A neurological disease caused by an expanded CAG trinucleotide repeat in the TATA-binding protein gene: a new polyglutamine disease?

1 reference

7 January 2021

SCA17, a novel autosomal dominant cerebellar ataxia caused by an expanded polyglutamine in TATA-binding protein

1 reference

7 January 2021

Clinical features and neuropathology of autosomal dominant spinocerebellar ataxia (SCA17).

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7 January 2021

SCA17 homozygote showing Huntington's disease-like phenotype.

1 reference

7 January 2021

Trinucleotide repeat expansion in SCA17/TBP in white patients with Huntington's disease-like phenotype.

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7 January 2021

Glutamine repeats and neurodegeneration

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7 January 2021

Polyglutamine neurodegenerative diseases and regulation of transcription: assembling the puzzle

1 reference

7 January 2021

Biochemistry and structural biology of transcription factor IID (TFIID)

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7 January 2021

Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin

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7 January 2021

Polyglutamine-expanded ataxin-7 antagonizes CRX function and induces cone-rod dystrophy in a mouse model of SCA7

1 reference

7 January 2021

CAG repeat expansion in the TATA box-binding protein gene causes autosomal dominant cerebellar ataxia.

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7 January 2021

A role for TBP dimerization in preventing unregulated gene expression

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7 January 2021

Fundamental cellular processes do not require vertebrate-specific sequences within the TATA-binding protein

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7 January 2021

Mutant small heat-shock protein 27 causes axonal Charcot-Marie-Tooth disease and distal hereditary motor neuropathy

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7 January 2021

Hsp27 and axonal growth in adult sensory neurons in vitro

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7 January 2021

The neuroprotective effects of heat shock protein 27 overexpression in transgenic animals against kainate-induced seizures and hippocampal cell death

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7 January 2021

Heat shock protein 27 rescues motor neurons following nerve injury and preserves muscle function

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7 January 2021

Morphological basis for the spectrum of clinical deficits in spinocerebellar ataxia 17 (SCA17).

1 reference

7 January 2021

Intergenerational instability and marked anticipation in SCA-17

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7 January 2021

Role for the amino-terminal region of human TBP in U6 snRNA transcription

1 reference

7 January 2021

Basal components of the transcription apparatus (RNA polymerase II, TATA-binding protein) contain activation domains: is the repetitive C-terminal domain (CTD) of RNA polymerase II a "portable enhancer domain"?

1 reference

7 January 2021

A severely defective TATA-binding protein-TFIIB interaction does not preclude transcriptional activation in vivo

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7 January 2021

Selective use of TBP and TFIIB revealed by a TATA-TBP-TFIIB array with altered specificity

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7 January 2021

Histone deacetylase inhibitors as therapeutics for polyglutamine disorders

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7 January 2021

Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death

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7 January 2021

ATAXIN-1 interacts with the repressor Capicua in its native complex to cause SCA1 neuropathology

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7 January 2021

Diseases of unstable repeat expansion: mechanisms and common principles

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7 January 2021

Transcriptional regulation and the role of diverse coactivators in animal cells

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7 January 2021

Structure and organisation of a murine gene encoding small heat-shock protein Hsp25.

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7 January 2021

Heat shock protein 27 is involved in neurite extension and branching of dorsal root ganglion neurons in vitro.

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7 January 2021

Dynamic expression of Hsp27 in the presence of mutant ataxin-3.

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7 January 2021

Decreased expression of Hsp27 and Hsp70 in transformed lymphoblastoid cells from patients with spinocerebellar ataxia type 7.

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7 January 2021

Heat shock protein 27 prevents cellular polyglutamine toxicity and suppresses the increase of reactive oxygen species caused by huntingtin

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7 January 2021

Constitutive expression of heat shock protein HSP25 in the central nervous system of the developing and adult mouse.

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7 January 2021

Small heat shock proteins in inherited peripheral neuropathies

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7 January 2021

Huntingtin aggregate-associated axonal degeneration is an early pathological event in Huntington's disease mice.

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7 January 2021

Expanded polyglutamines in Caenorhabditis elegans cause axonal abnormalities and severe dysfunction of PLM mechanosensory neurons without cell death

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7 January 2021

Method to introduce stable, expanded, polyglutamine-encoding CAG/CAA trinucleotide repeats into CAG repeat-containing genes

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7 January 2021

Dimerization of TFIID when not bound to DNA.

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7 January 2021

Interaction of Huntington disease protein with transcriptional activator Sp1

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7 January 2021

Identifiers

DOI

10.1038/NN2011

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4 August 2017

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