(Q34758376)

4 August 2017

title

Plasmodium falciparum field isolates use complement receptor 1 (CR1) as a receptor for invasion of erythrocytes (English)

1 reference

4 August 2017

main subject

Plasmodium falciparum

0 references

1 reference

Carmenza Spadafora

2

0 references

Gordon A Awandare

object named as

Gordon A Awandare

1

0 references

Gordon A Awandare

object named as

Awandare, G.A.

0 references

author name string

J Kathleen Moch

3

1 reference

4 August 2017

Sheetij Dutta

4

1 reference

4 August 2017

J David Haynes

5

1 reference

4 August 2017

José A Stoute

6

1 reference

4 August 2017

Spadafora, C.

0 references

Moch, J.K.

0 references

Dutta, S.

0 references

Haynes, J.D.

0 references

Stoute, J.A.

0 references

language of work or name

1 reference

18 January 2011

1 reference

full work available at URL

4 August 2017

2011

0 references

number of pages

4

1 reference

based on heuristic

inferred from page(s)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3070236

1 reference

21 March 2022

Molecular and Biochemical Parasitology

1 reference

4 August 2017

volume

177

1 reference

4 August 2017

issue

1

1 reference

4 August 2017

page(s)

57-60

1 reference

Complement receptor 1 is the host erythrocyte receptor for Plasmodium falciparum PfRh4 invasion ligand

4 August 2017

cites work

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Complement receptor 1 is a sialic acid independent erythrocyte receptor of Plasmodium falciparum

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Complement Receptor 1: disease associations and therapeutic implications.

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Variation in use of erythrocyte invasion pathways by Plasmodium falciparum mediates evasion of human inhibitory antibodies

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Structural basis of antigenic escape of a malaria vaccine candidate

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Invasion pathways and malaria severity in Kenyan Plasmodium falciparum clinical isolates

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Complement receptor 1 polymorphisms associated with resistance to severe malaria in Kenya

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Plasmodium falciparum erythrocyte invasion through glycophorin C and selection for Gerbich negativity in human populations

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Plasmodium falciparum field isolates commonly use erythrocyte invasion pathways that are independent of sialic acid residues of glycophorin A

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Diversity of the vaccine candidate AMA-1 of Plasmodium falciparum

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Invasion of erythrocytes by Plasmodium falciparum malaria parasites: evidence for receptor heterogeneity and two receptors

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Falciparum malaria parasites invade erythrocytes that lack glycophorin A and B (MkMk). Strain differences indicate receptor heterogeneity and two pathways for invasion

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Evidence for a switching mechanism in the invasion of erythrocytes by Plasmodium falciparum

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

A functional single-nucleotide polymorphism in the CR1 promoter region contributes to protection against cerebral malaria

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Loss of red blood cell-complement regulatory proteins and increased levels of circulating immune complexes are associated with severe malarial anemia

26 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Apical membrane antigen 1 plays a central role in erythrocyte invasion by Plasmodium species

26 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Red cell surface changes and erythrophagocytosis in children with severe plasmodium falciparum anemia

26 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3070236

Competitive inhibition by soluble erythrocyte glycoproteins of penetration by Plasmodium falciparum

26 October 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21251929

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1016/J.MOLBIOPARA.2011.01.005

1 reference

4 August 2017

1 reference

4 August 2017

1 reference