(Q35064075)

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The Pseudomonas syringae pv. tomato type III effector HopM1 suppresses Arabidopsis defenses independent of suppressing salicylic acid signaling and of targeting AtMIN7.

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Europe PubMed Central

PubMed publication ID

6 August 2017

The Pseudomonas syringae pv. tomato type III effector HopM1 suppresses Arabidopsis defenses independent of suppressing salicylic acid signaling and of targeting AtMIN7. (English)

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Europe PubMed Central

PubMed publication ID

6 August 2017

Medical uses of salicylic acid

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based on heuristic

inferred from title

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Pseudomonas syringae

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Pseudomonas syringae

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based on heuristic

inferred from title

Justin Whitehill

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Justin Whitehill

3

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Anju Gangadharan

1

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Europe PubMed Central

PubMed publication ID

6 August 2017

Mysore-Venkatarau Sreerekha

2

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Europe PubMed Central

PubMed publication ID

6 August 2017

Jong Hyun Ham

4

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Europe PubMed Central

PubMed publication ID

6 August 2017

David Mackey

5

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Europe PubMed Central

PubMed publication ID

6 August 2017

language of work or name

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publication date

6 December 2013

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Europe PubMed Central

PubMed publication ID

6 August 2017

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Europe PubMed Central

PubMed publication ID

6 August 2017

8

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Europe PubMed Central

PubMed publication ID

6 August 2017

12

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Europe PubMed Central

PubMed publication ID

6 August 2017

e82032

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Europe PubMed Central

PubMed publication ID

6 August 2017

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Creative Commons Attribution 3.0 Unported

6 December 2013

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April 2022 Public Data File from Crossref

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Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3855835/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

From pioneers to team players: TGA transcription factors provide a molecular link between different stress pathways

1 reference

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1 reference

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Effector-triggered immunity blocks pathogen degradation of an immunity-associated vesicle traffic regulator in Arabidopsis

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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The plant immune system

1 reference

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A bacterial virulence protein suppresses host innate immunity to cause plant disease

1 reference

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Host-microbe interactions: shaping the evolution of the plant immune response

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Identification of a bacterial type III effector family with G protein mimicry functions.

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Two Pseudomonas syringae type III effectors inhibit RIN4-regulated basal defense in Arabidopsis

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The response regulator 2 mediates ethylene signalling and hormone signal integration in Arabidopsis.

1 reference

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A family of conserved bacterial effectors inhibits salicylic acid-mediated basal immunity and promotes disease necrosis in plants

1 reference

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SNARE-protein-mediated disease resistance at the plant cell wall.

1 reference

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8 July 2018

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1 reference

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The salicylic acid loop in plant defense

1 reference

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Inducers of plant systemic acquired resistance regulate NPR1 function through redox changes

1 reference

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bZIP transcription factors in Arabidopsis

1 reference

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Isochorismate synthase is required to synthesize salicylic acid for plant defence

1 reference

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The Pseudomonas syringae Hrp pathogenicity island has a tripartite mosaic structure composed of a cluster of type III secretion genes bounded by exchangeable effector and conserved effector loci that contribute to parasitic fitness and pathogenicity

1 reference

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1 reference

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1 reference

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DNA repair proteins are directly involved in regulation of gene expression during plant immune response.

1 reference

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Pseudomonas syringae pv. tomato DC3000 HopPtoM (CEL ORF3) is important for lesion formation but not growth in tomato and is secreted and translocated by the Hrp type III secretion system in a chaperone-dependent manner.

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In vivo interaction between NPR1 and transcription factor TGA2 leads to salicylic acid-mediated gene activation in Arabidopsis

1 reference

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1 reference

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1 reference

https://pubmed.ncbi.nlm.nih.gov/24324742

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1 reference

https://pubmed.ncbi.nlm.nih.gov/24324742

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LSD1 regulates salicylic acid induction of copper zinc superoxide dismutase in Arabidopsis thaliana

1 reference

https://pubmed.ncbi.nlm.nih.gov/24324742

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1 reference

https://pubmed.ncbi.nlm.nih.gov/24324742

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based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PONE.0082032

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

2013PLoSO...882032G

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PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

ResearchGate publication ID

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