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Disruption of cellular transport: a common cause of neurodegeneration?
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scholarly article
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
review article
1 reference
stated in
Europe PubMed Central
title
Disruption of cellular transport: a common cause of neurodegeneration?
(English)
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
author name string
Andrew H Crosby
series ordinal
1
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
publication date
1 May 2003
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stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
published in
Lancet Neurology
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
volume
2
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
issue
5
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
page(s)
311-316
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
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Molecular basis of inherited spastic paraplegias
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Is the transportation highway the right road for hereditary spastic paraplegia?
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Progressive spastic paraparesis: hereditary spastic paraplegia and its relation to primary and amyotrophic lateral sclerosis
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Hereditary (familial) spastic paraplegia; further clinical and pathologic observations.
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Familial amyotrophic lateral sclerosis
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Mitochondria and degenerative disorders
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Endocytic traffic in polarized epithelial cells: role of the actin and microtubule cytoskeleton
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Cruising along microtubule highways: how membranes move through the secretory pathway
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Motor-cargo interactions: the key to transport specificity.
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Mechanisms of trafficking in axons and dendrites: implications for development and neurodegeneration.
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7 January 2021
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Disruption of dynein/dynactin inhibits axonal transport in motor neurons causing late-onset progressive degeneration.
1 reference
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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Cloning by insertional mutagenesis of a cDNA encoding Caenorhabditis elegans kinesin heavy chain
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A kinesin heavy chain (KIF5A) mutation in hereditary spastic paraplegia (SPG10)
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7 January 2021
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Charcot-Marie-Tooth disease type 2A caused by mutation in a microtubule motor KIF1Bbeta
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Defective kinesin heavy chain behavior in mouse kinesin light chain mutants
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SPG20 is mutated in Troyer syndrome, an hereditary spastic paraplegia.
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The Vps4p AAA ATPase regulates membrane association of a Vps protein complex required for normal endosome function
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7 January 2021
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Spastin, a new AAA protein, is altered in the most frequent form of autosomal dominant spastic paraplegia
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Spastin, the protein mutated in autosomal dominant hereditary spastic paraplegia, is involved in microtubule dynamics
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The product of the ATM gene is a 370-kDa nuclear phosphoprotein
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ATM immunolocalization in mouse neuronal endosomes: implications for ataxia-telangiectasia.
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Further evidence that SPG3A gene mutations cause autosomal dominant hereditary spastic paraplegia
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Crossref
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Mutations in a newly identified GTPase gene cause autosomal dominant hereditary spastic paraplegia
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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The dynamin family of mechanoenzymes: pinching in new places
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The gene encoding alsin, a protein with three guanine-nucleotide exchange factor domains, is mutated in a form of recessive amyotrophic lateral sclerosis
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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A gene encoding a putative GTPase regulator is mutated in familial amyotrophic lateral sclerosis 2
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Infantile-onset ascending hereditary spastic paralysis is associated with mutations in the alsin gene
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Progress in the pathogenesis of amyotrophic lateral sclerosis
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Differential expression of inflammation- and apoptosis-related genes in spinal cords of a mutant SOD1 transgenic mouse model of familial amyotrophic lateral sclerosis
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7 January 2021
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Slowing of axonal transport is a very early event in the toxicity of ALS-linked SOD1 mutants to motor neurons
1 reference
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Going new places using an old MAP: tau, microtubules and human neurodegenerative disease.
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Tau blocks traffic of organelles, neurofilaments, and APP vesicles in neurons and enhances oxidative stress
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Tau proteins with frontotemporal dementia-17 mutations have both altered expression levels and phosphorylation profiles in differentiated neuroblastoma cells.
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reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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inferred from DOI database lookup
An R5L tau mutation in a subject with a progressive supranuclear palsy phenotype.
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Mis-sense mutation Val→Ile in exon 17 of amyloid precursor protein gene in Japanese familial Alzheimer's disease
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Kinesin-mediated axonal transport of a membrane compartment containing beta-secretase and presenilin-1 requires APP
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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APP on the move.
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Identification and localization of huntingtin in brain and human lymphoblastoid cell lines with anti-fusion protein antibodies
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
based on heuristic
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Huntingtin-associated protein 1 interacts with hepatocyte growth factor-regulated tyrosine kinase substrate and functions in endosomal trafficking
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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HIP14, a novel ankyrin domain-containing protein, links huntingtin to intracellular trafficking and endocytosis
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Huntingtin-associated protein 1 (HAP1) interacts with the p150Glued subunit of dynactin
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
based on heuristic
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Function of Hrs in endocytic trafficking and signalling
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Hrs interacts with sorting nexin 1 and regulates degradation of epidermal growth factor receptor
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Hrs and hbp: possible regulators of endocytosis and exocytosis
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https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Wild-type and mutant huntingtins function in vesicle trafficking in the secretory and endocytic pathways
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Toxic proteins in neurodegenerative disease
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS1474-4422%2803%2900383-1
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7 January 2021
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Identifiers
DOI
10.1016/S1474-4422(03)00383-1
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
PubMed ID
12849185
1 reference
stated in
Europe PubMed Central
PubMed ID
12849185
retrieved
6 August 2017
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