(Q35494903)

8 August 2017

7 references

13 March 2019

14 March 2019

24 March 2019

17 June 2019

31 July 2019

15 December 2019

3 May 2021

Regulation of RAGE for attenuating progression of diabetic vascular complications (English)

1 reference

8 August 2017

6 references

13 March 2019

14 March 2019

24 March 2019

17 June 2019

31 July 2019

15 December 2019

cell surface receptor

1 reference

31 July 2019

1 reference

15 December 2019

1 reference

3 May 2021

Myat Thu Thu Win

1

1 reference

8 August 2017

Yasuhiko Yamamoto

2

1 reference

8 August 2017

Seiichi Munesue

3

1 reference

8 August 2017

Hidehito Saito

4

1 reference

8 August 2017

Dong Han

5

1 reference

8 August 2017

So Motoyoshi

6

1 reference

8 August 2017

Tarek Kamal

7

1 reference

8 August 2017

Takuro Ohara

8

1 reference

8 August 2017

Takuo Watanabe

9

1 reference

8 August 2017

Hiroshi Yamamoto

10

1 reference

8 August 2017

29 October 2011

8 references

Experimental Diabetes Research

8 August 2017

13 March 2019

14 March 2019

24 March 2019

17 June 2019

31 July 2019

15 December 2019

3 May 2021

8 references

8 August 2017

13 March 2019

14 March 2019

24 March 2019

17 June 2019

31 July 2019

15 December 2019

3 May 2021

2012

1 reference

8 August 2017

894605

1 reference

Creative Commons Attribution 3.0 Unported

8 August 2017

7 references

13 March 2019

14 March 2019

24 March 2019

17 June 2019

31 July 2019

15 December 2019

on focus list of Wikimedia project

3 May 2021

copyright status

copyrighted

0 references

ScienceSource

0 references

cites work

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Interaction of the RAGE cytoplasmic domain with diaphanous-1 is required for ligand-stimulated cellular migration through activation of Rac1 and Cdc42

27 July 2018

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Glycation of low-density lipoprotein results in the time-dependent accumulation of cholesteryl esters and apolipoprotein B-100 protein in primary human monocyte-derived macrophages

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Benfotiamine prevents macro- and microvascular endothelial dysfunction and oxidative stress following a meal rich in advanced glycation end products in individuals with type 2 diabetes

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Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily

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The pattern recognition receptor (RAGE) is a counterreceptor for leukocyte integrins: a novel pathway for inflammatory cell recruitment

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Novel splice variants of the receptor for advanced glycation end-products expressed in human vascular endothelial cells and pericytes, and their putative roles in diabetes-induced vascular injury

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27 July 2018

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27 July 2018

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27 July 2018

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Glycolaldehyde, a reactive intermediate for advanced glycation end products, plays an important role in the generation of an active ligand for the macrophage scavenger receptor

27 July 2018

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Glycation impairs high-density lipoprotein function

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N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

27 July 2018

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RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

27 July 2018

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

27 July 2018

1 reference

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Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts

27 July 2018

1 reference

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Depletion of reactive advanced glycation endproducts from diabetic uremic sera using a lysozyme-linked matrix

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Identification of galectin-3 as a high-affinity binding protein for advanced glycation end products (AGE): a new member of the AGE-receptor complex

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Nonenzymic, Polyvalent Anion-catalyzed Formation of Methylglyoxal as an Explanation of Its Presence in Physiological Systems

27 July 2018

1 reference

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The glyoxalase system: new developments towards functional characterization of a metabolic pathway fundamental to biological life

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Advanced glycosylation products quench nitric oxide and mediate defective endothelium-dependent vasodilatation in experimental diabetes

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Two novel rat liver membrane proteins that bind advanced glycosylation endproducts: relationship to macrophage receptor for glucose-modified proteins

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Non-enzymatic glycation of apolipoprotein B in the sera of diabetic and non-diabetic subjects

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

RAGE biology, atherosclerosis and diabetes

27 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Deletion of the receptor for advanced glycation end products reduces glomerulosclerosis and preserves renal function in the diabetic OVE26 mouse

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

RAGE mediates oxidized LDL-induced pro-inflammatory effects and atherosclerosis in non-diabetic LDL receptor-deficient mice

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Angiotensin II upregulates RAGE expression on podocytes: role of AT2 receptors

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Serum endogenous secretory RAGE level is an independent risk factor for the progression of carotid atherosclerosis in type 1 diabetes.

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

RAGE ligation affects T cell activation and controls T cell differentiation

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Receptor for advanced glycation end products (RAGEs) and experimental diabetic neuropathy

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Beneficial effects of C36, a novel breaker of advanced glycation endproducts cross-links, on the cardiovascular system of diabetic rats

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Decreased plasma soluble RAGE in patients with hypercholesterolemia: effects of statins

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Low circulating endogenous secretory receptor for AGEs predicts cardiovascular mortality in patients with end-stage renal disease

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

RAGE control of diabetic nephropathy in a mouse model: effects of RAGE gene disruption and administration of low-molecular weight heparin

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Development of an ELISA for esRAGE and its application to type 1 diabetic patients

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

A severe diabetic nephropathy model with early development of nodule-like lesions induced by megsin overexpression in RAGE/iNOS transgenic mice

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Upregulation of RAGE and its ligands in proliferative retinal disease

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

RAGE modulates vascular inflammation and atherosclerosis in a murine model of type 2 diabetes

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Nifedipine inhibits gene expression of receptor for advanced glycation end products (RAGE) in endothelial cells by suppressing reactive oxygen species generation

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cells

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Advanced glycation end product interventions reduce diabetes-accelerated atherosclerosis.

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Fetal or neonatal low-glycotoxin environment prevents autoimmune diabetes in NOD mice

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

RAGE drives the development of glomerulosclerosis and implicates podocyte activation in the pathogenesis of diabetic nephropathy

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

N-phenacylthiazolium bromide decreases renal and increases urinary advanced glycation end products excretion without ameliorating diabetic nephropathy in C57BL/6 mice

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Glycation amplifies lipoprotein(a)-induced alterations in the generation of fibrinolytic regulators from human vascular endothelial cells

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Breakers of advanced glycation end products restore large artery properties in experimental diabetes.

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Cloning and expression of a cell surface receptor for advanced glycosylation end products of proteins

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205669

Receptor for Advanced Glycation End Products Activation Injures Primary Sensory Neurons via Oxidative Stress

26 September 2018

1 reference

Crossref

https://api.crossref.org/works/10.1155%2F2012%2F894605

A role for macrophage scavenger receptors in atherosclerosis and susceptibility to infection

21 January 2018

1 reference

Crossref

https://api.crossref.org/works/10.1155%2F2012%2F894605

A soluble form of the receptor for advanced glycation endproducts (RAGE) is produced by proteolytic cleavage of the membrane-bound form by the sheddase a disintegrin and metalloprotease 10 (ADAM10)

21 January 2018

1 reference

12 December 2020

Advanced glycation end-product of low density lipoprotein activates the toll-like 4 receptor pathway implications for diabetic atherosclerosis

1 reference

12 December 2020

Enhanced expression of receptor for advanced glycation end-products is associated with low circulating soluble isoforms of the receptor in Type 2 diabetes

1 reference

12 December 2020

Non-enzymatic glycation of lipoprotein(a) in vitro and in vivo

1 reference

12 December 2020

Advanced glycation end products and their receptors co-localise in rat organs susceptible to diabetic microvascular injury

1 reference

12 December 2020

The relationship between accumulation of advanced glycation end products and expression of vascular endothelial growth factor in human diabetic retinas

1 reference

12 December 2020

Pericytes and the pathogenesis of diabetic retinopathy

1 reference

12 December 2020

Expression profiling of endogenous secretory receptor for advanced glycation end products in human organs

1 reference

12 December 2020

Roles of the AGE-RAGE system in vascular injury in diabetes

1 reference

12 December 2020

The role of galectin-3 in endocytosis of advanced glycation end products and modified low density lipoproteins

1 reference

12 December 2020

Suppression of RAGE as a basis of simvastatin-dependent plaque stabilization in type 2 diabetes

1 reference

12 December 2020

Inhibition of diabetic leukostasis and blood-retinal barrier breakdown with a soluble form of a receptor for advanced glycation end products

1 reference

12 December 2020

Serum levels of soluble form of receptor for advanced glycation end products (sRAGE) may reflect tissue RAGE expression in diabetes

1 reference

12 December 2020

Soluble receptor for AGE (RAGE) is a novel independent predictor of all-cause and cardiovascular mortality in type 1 diabetes

1 reference

12 December 2020

Identifiers

DOI

10.1155/2012/894605

1 reference

8 August 2017

1 reference

8 August 2017

1 reference