(Q35543254)

English

Decreased metalloprotease 9 induction, cardiac fibrosis, and higher autophagy after pressure overload in mice lacking the transcriptional regulator p8.

scientific article published on 20 July 2011

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

Decreased metalloprotease 9 induction, cardiac fibrosis, and higher autophagy after pressure overload in mice lacking the transcriptional regulator p8. (English)

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

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author name string

Serban P Georgescu

1

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

Mark J Aronovitz

2

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

Juan L Iovanna

3

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

Richard D Patten

4

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

John M Kyriakis

5

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

Sandro Goruppi

6

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

publication date

20 July 2011

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

American Journal of Physiology - Cell Physiology

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

301

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

C1046-56

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3213912/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

Autophagy and the integrated stress response

1 reference

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12 July 2018

Ablation of C/EBP homologous protein attenuates endoplasmic reticulum-mediated apoptosis and cardiac dysfunction induced by pressure overload

1 reference

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FoxO, autophagy, and cardiac remodeling

1 reference

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12 July 2018

Deficiency of the transcriptional regulator p8 results in increased autophagy and apoptosis, and causes impaired heart function

1 reference

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Effect of matrix metalloproteinase-9 knockout on vein graft remodelling in mice

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Autophagy in cardiovascular disease.

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Cardiac autophagy is a maladaptive response to hemodynamic stress

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Re-employment of developmental transcription factors in adult heart disease

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Helix-loop-helix protein p8, a transcriptional regulator required for cardiomyocyte hypertrophy and cardiac fibroblast matrix metalloprotease induction

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Gata4 is required for maintenance of postnatal cardiac function and protection from pressure overload-induced heart failure

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Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy

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Toward transcriptional therapies for the failing heart: chemical screens to modulate genes

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Loss or inhibition of uPA or MMP-9 attenuates LV remodeling and dysfunction after acute pressure overload in mice

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The pro-hypertrophic basic helix-loop-helix protein p8 is degraded by the ubiquitin/proteasome system in a protein kinase B/Akt- and glycogen synthase kinase-3-dependent manner, whereas endothelin induction of p8 mRNA and renal mesangial cell hypert

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Identifiers

10.1152/AJPCELL.00211.2011

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

8 August 2017

ResearchGate publication ID

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