(Q35545556)

8 August 2017

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title

Cerebrovascular effects of amyloid-beta peptides: mechanisms and implications for Alzheimer's dementia (English)

1 reference

8 August 2017

0 references

1 reference

10.1023/A:1025092617651

https://api.crossref.org/works/10.1023/A:1025092617651

15 March 2024

1

Costantino Iadecola

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language of work or name

English

1 reference

National Center for Biotechnology Information

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=14514024

15 March 2024

publication date

1 October 2003

1 reference

full work available at URL

8 August 2017

http://www.springerlink.com/index/pdf/10.1023/A:1025092617651

1 reference

Crossref

DOI

10.1023/A:1025092617651

https://api.crossref.org/works/10.1023/A:1025092617651

Cellular and Molecular Neurobiology

15 March 2024

published in

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8 August 2017

23

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8 August 2017

681-689

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8 August 2017

4-5

1 reference

https://scigraph.springernature.com/pub.10.1023/a:1025092617651

8 August 2017

0 references

cites work

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Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

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Exogenous A beta1-40 reproduces cerebrovascular alterations resulting from amyloid precursor protein overexpression in mice

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Cerebrovascular autoregulation is profoundly impaired in mice overexpressing amyloid precursor protein

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Mutant genes in familial Alzheimer's disease and transgenic models

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Altered brain activation in cognitively intact individuals at high risk for Alzheimer's disease

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APPSw transgenic mice develop age-related A beta deposits and neuropil abnormalities, but no neuronal loss in CA1.

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Interactions of oxidants with vascular signaling systems

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Alzheimer's disease and Down's syndrome: Sharing of a unique cerebrovascular amyloid fibril protein

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A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease

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Angiotensin II stimulates NADH and NADPH oxidase activity in cultured vascular smooth muscle cells

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Alterations in cerebral blood flow and glucose utilization in mice overexpressing the amyloid precursor protein

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Declining brain activity in cognitively normal apolipoprotein E epsilon 4 heterozygotes: A foundation for using positron emission tomography to efficiently test treatments to prevent Alzheimer's disease.

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Cerebral vessels in ageing and Alzheimer's disease

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Cellular actions of beta-amyloid precursor protein and its soluble and fibrillogenic derivatives

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A beta-peptides enhance vasoconstriction in cerebral circulation

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Apoptosis is induced by beta-amyloid in cultured central nervous system neurons

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NADH oxidoreductase is a major source of superoxide anion in bovine coronary artery endothelium

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Characteristics of the in vitro vasoactivity of beta-amyloid peptides

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Evidence of neuronal oxidative damage in Alzheimer's disease.

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beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

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Identifiers

DOI

10.1023/A:1025092617651

1 reference

Dimensions Publication ID

8 August 2017

0 references

1 reference