(Q35557565)

English

Basic fibroblast growth factor (bFGF) regulation of the plasma membrane calcium ATPase (PMCA) as part of an anti-apoptotic mechanism of action

scientific article published on October 2003

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scholarly article

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Europe PubMed Central

8 August 2017

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Europe PubMed Central

Basic fibroblast growth factor (bFGF) regulation of the plasma membrane calcium ATPase (PMCA) as part of an anti-apoptotic mechanism of action (English)

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Europe PubMed Central

8 August 2017

apoptotic process

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author name string

John J Peluso

1

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Europe PubMed Central

8 August 2017

language of work or name

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publication date

1 October 2003

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Europe PubMed Central

8 August 2017

Biochemical Pharmacology

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Europe PubMed Central

8 August 2017

66

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Europe PubMed Central

8 August 2017

8

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Europe PubMed Central

8 August 2017

1363-1369

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Europe PubMed Central

8 August 2017

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Fibroblast growth factors

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Basic fibroblast growth factor maintains calcium homeostasis and granulosa cell viability by stimulating calcium efflux via a PKC delta-dependent pathway

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The plasma membrane calcium pump--a physiological perspective on its regulation

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Plasma membrane Ca2+ ATPase isoform 2b interacts preferentially with Na+/H+ exchanger regulatory factor 2 in apical plasma membranes

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Short-term regulation of the proximal tubule Na+,K+-ATPase: increased/decreased Na+,K+-ATPase activity mediated by protein kinase C isoforms

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G protein-coupled receptors regulate Na+,K+-ATPase activity and endocytosis by modulating the recruitment of adaptor protein 2 and clathrin

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FGF-2 inhibits apoptosis in human teratocarcinoma cells during differentiation on collagen substratum

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Nerve growth factor and basic fibroblast growth factor protect rat cerebellar granule cells in culture against ethanol-induced cell death.

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bFGF inhibits the activation of caspase-3 and apoptosis of P19 embryonal carcinoma cells during neuronal differentiation

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Basic fibroblast growth factor inhibited Ca2+ ionophore-induced apoptotic cell death of cultured cortical neurons from embryonic rats

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Growth factors prevent changes in Bcl-2 and Bax expression and neuronal apoptosis induced by nitric oxide

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Apoptosis occurs in the oligodendroglial lineage, and is prevented by basic fibroblast growth factor

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Chemosensitization by fibroblast growth factor-2 is not dependent upon proliferation, S-phase accumulation, or p53 status

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Identifiers

10.1016/S0006-2952(03)00486-6

1 reference

Europe PubMed Central

8 August 2017

1 reference

Europe PubMed Central

8 August 2017

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