(Q43551075)

24 November 2017

title

Cytochrome c release occurs via Ca2+-dependent and Ca2+-independent mechanisms that are regulated by Bax. (English)

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24 November 2017

3

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24 November 2017

4

Orrenius S

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24 November 2017

Gogvadze V

1

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24 November 2017

Robertson JD

series ordinal

2

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24 November 2017

language of work or name

English

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publication date

22 March 2001

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Journal of Biological Chemistry

24 November 2017

published in

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24 November 2017

volume

276

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24 November 2017

issue

22

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24 November 2017

page(s)

19066-19071

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Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

24 November 2017

cites work

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

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Implication of mitochondria in apoptosis.

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Apoptosis: checkpoint at the mitochondrial frontier.

21 January 2018

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Mitochondrial physiology and pathology; concepts of programmed death of organelles, cells and organisms

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Mitochondria at the crossroad of apoptotic cell death

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Molecular mechanisms of apoptosis induced by cytotoxic chemicals

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Mitochondria and apoptosis

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Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

21 January 2018

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Importance of the redox state of cytochrome c during caspase activation in cytosolic extracts

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M100614200

Hypotonic fragility of outer membrane and activation of external pathway of NADH oxidation in rat liver mitochondria are increased with age.

21 January 2018

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Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart

21 January 2018

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Bcl-2 family: life-or-death switch

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bax directly induces release of cytochrome c from isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Expression and purification of full-length human Bax alpha.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

The release of cytochrome c from mitochondria during apoptosis of NGF-deprived sympathetic neurons is a reversible event

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bid acts on the permeability transition pore complex to induce apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

PK11195, a ligand of the mitochondrial benzodiazepine receptor, facilitates the induction of apoptosis and reverses Bcl-2-mediated cytoprotection.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Release of cytochrome c from liver mitochondria during permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Mitochondria as all-round players of the calcium game

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100614200

21 January 2018

Identifiers

DOI

10.1074/JBC.M100614200

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24 November 2017

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