(Q40827697)

24 September 2017

Bid acts on the permeability transition pore complex to induce apoptosis (English)

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24 September 2017

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11

Kroemer G

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24 September 2017

Zamzami N

1

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24 September 2017

El Hamel C

2

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24 September 2017

Maisse C

3

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24 September 2017

Brenner C

4

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24 September 2017

Muñoz-Pinedo C

5

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24 September 2017

Belzacq AS

6

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24 September 2017

Costantini P

7

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24 September 2017

Vieira H

8

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24 September 2017

Loeffler M

9

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24 September 2017

Molle G

10

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24 September 2017

language of work or name

English

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publication date

1 December 2000

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24 September 2017

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24 September 2017

19

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24 September 2017

6342-6350

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24 September 2017

54

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1204030

24 September 2017

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cites work

Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Purification and liposomal reconstitution of permeability transition pore complex

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Solution structure of BID, an intracellular amplifier of apoptotic signaling

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Oxidation of a critical thiol residue of the adenine nucleotide translocator enforces Bcl-2-independent permeability transition pore opening and apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Cyclophilin-D binds strongly to complexes of the voltage-dependent anion channel and the adenine nucleotide translocase to form the permeability transition pore

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Opening of the mitochondrial permeability transition pore causes matrix expansion and outer membrane rupture in Fas-mediated hepatic apoptosis in mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

A cytomegalovirus-encoded mitochondria-localized inhibitor of apoptosis structurally unrelated to Bcl-2.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

BCL-2 family members and the mitochondria in apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Biochemical and genetic analysis of the mitochondrial response of yeast to BAX and BCL-X(L)

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Role of oxidative phosphorylation in Bax toxicity.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

The Mitochondrial Permeability Transition Augments Fas-induced Apoptosis in Mouse Hepatocytes

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bax directly induces release of cytochrome c from isolated mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

The proto-oncogene Bcl-2 and its role in regulating apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Mitochondrial control of cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

The Destabilization of Lipid Membranes Induced by the C-terminal Fragment of Caspase 8-cleaved Bid Is Inhibited by the N-terminal Fragment

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Solution structure of the proapoptotic molecule BID: a structural basis for apoptotic agonists and antagonists

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bcl-x(L) forms an ion channel in synthetic lipid membranes.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Formation of bimolecular membranes from lipid monolayers and a study of their electrical properties

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

The overexpression of Bax produces cell death upon induction of the mitochondrial permeability transition

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Comparison of the effects of bax -expression in yeast under fermentative and respiratory conditions: investigation of the role of adenine nucleotides carrier and cytochrome c

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Investigation of bax-induced release of cytochrome c from yeast mitochondria permeability of mitochondrial membranes, role of VDAC and ATP requirement

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Double identity for proteins of the Bcl-2 family

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Two CD95 (APO-1/Fas) signaling pathways

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Apoptosis signaling in lymphocytes.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Channel formation by antiapoptotic protein Bcl-2.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Ion channel activity of the BH3 only Bcl-2 family member, BID.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Purification of mitochondria for apoptosis assays

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Molecular characterization of mitochondrial apoptosis-inducing factor

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Outer mitochondrial membrane permeability can regulate coupled respiration and cell survival

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bcl-2 proteins: regulators of apoptosis or of mitochondrial homeostasis?

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Subcellular and submitochondrial mode of action of Bcl-2-like oncoproteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

The mitochondrial permeability transition.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204030

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1204030

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Dimensions Publication ID

24 September 2017

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1 reference