(Q35660187)

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Toll-Like Receptor 3 Signaling via TRIF Contributes to a Protective Innate Immune Response to Severe Acute Respiratory Syndrome Coronavirus Infection

scientific article published on 26 May 2015

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Europe PubMed Central

PubMed publication ID

9 August 2017

Toll-Like Receptor 3 Signaling via TRIF Contributes to a Protective Innate Immune Response to Severe Acute Respiratory Syndrome Coronavirus Infection (English)

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Europe PubMed Central

PubMed publication ID

9 August 2017

toll-like receptor

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inferred from title

7

object named as

Ralph S Baric

1 reference

Europe PubMed Central

PubMed publication ID

9 August 2017

5

object named as

Michael G Katze

1 reference

Europe PubMed Central

PubMed publication ID

9 August 2017

6

object named as

Mark T Heise

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Europe PubMed Central

PubMed publication ID

9 August 2017

Alexandra Schäfer

4

object named as

Alexandra Schäfer

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Europe PubMed Central

PubMed publication ID

9 August 2017

Allison L Totura

1

object named as

Allison L Totura

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Europe PubMed Central

PubMed publication ID

9 August 2017

Sudhakar Agnihothram

3

object named as

Sudhakar Agnihothram

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Europe PubMed Central

PubMed publication ID

9 August 2017

Alan C Whitmore

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Alan Whitmore

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Europe PubMed Central

PubMed publication ID

9 August 2017

publication date

26 May 2015

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Europe PubMed Central

PubMed publication ID

9 August 2017

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Europe PubMed Central

PubMed publication ID

9 August 2017

6

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Europe PubMed Central

PubMed publication ID

9 August 2017

3

1 reference

Europe PubMed Central

PubMed publication ID

9 August 2017

e00638-15

1 reference

Europe PubMed Central

PubMed publication ID

9 August 2017

Mucosal polyinosinic-polycytidylic acid improves protection elicited by replicating influenza vaccines via enhanced dendritic cell function and T cell immunity.

1 reference

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Highly pathogenic avian influenza A H5N1 and pandemic H1N1 virus infections have different phenotypes in Toll-like receptor 3 knockout mice.

1 reference

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Effects of Toll-like receptor stimulation on eosinophilic infiltration in lungs of BALB/c mice immunized with UV-inactivated severe acute respiratory syndrome-related coronavirus vaccine

1 reference

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Pathogenic influenza viruses and coronaviruses utilize similar and contrasting approaches to control interferon-stimulated gene responses.

1 reference

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Viral infection of the lung: host response and sequelae

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The TLR4 antagonist Eritoran protects mice from lethal influenza infection

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Both TRIF and IPS-1 adaptor proteins contribute to the cerebral innate immune response against herpes simplex virus 1 infection

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Inhibition of novel β coronavirus replication by a combination of interferon-α2b and ribavirin

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CXCL10-CXCR3 enhances the development of neutrophil-mediated fulminant lung injury of viral and nonviral origin

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Monocyte recruitment during infection and inflammation

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A double-inactivated severe acute respiratory syndrome coronavirus vaccine provides incomplete protection in mice and induces increased eosinophilic proinflammatory pulmonary response upon challenge

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DDX1, DDX21, and DHX36 helicases form a complex with the adaptor molecule TRIF to sense dsRNA in dendritic cells

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MDA5 and TLR3 initiate pro-inflammatory signaling pathways leading to rhinovirus-induced airways inflammation and hyperresponsiveness

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Type I interferon signaling regulates Ly6C(hi) monocytes and neutrophils during acute viral pneumonia in mice

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Does unrestrained single-chamber plethysmography provide a valid assessment of airway responsiveness in allergic BALB/c mice?

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Response of human pulmonary epithelial cells to lipopolysaccharide involves Toll-like receptor 4 (TLR4)-dependent signaling pathways: evidence for an intracellular compartmentalization of TLR4.

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Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

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Noninvasive measurement of airway responsiveness in allergic mice using barometric plethysmography

1 reference

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cutting edge: cooperation of IPS-1- and TRIF-dependent pathways in poly IC-enhanced antibody production and cytotoxic T cell responses

1 reference

https://pubmed.ncbi.nlm.nih.gov/26015500

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/MBIO.00638-15

1 reference

Europe PubMed Central

PubMed publication ID

9 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

9 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

9 August 2017

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