(Q35909272)

English

A Conserved HIV-1-Derived Peptide Presented by HLA-E Renders Infected T-cells Highly Susceptible to Attack by NKG2A/CD94-Bearing Natural Killer Cells.

scientific article published in February 2016

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scholarly article

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Europe PubMed Central

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6 March 2020

A Conserved HIV-1-Derived Peptide Presented by HLA-E Renders Infected T-cells Highly Susceptible to Attack by NKG2A/CD94-Bearing Natural Killer Cells (English)

1 reference

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6 March 2020

Andrew Cogswell

2

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6 March 2020

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6 March 2020

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Edward Barker

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6 March 2020

Vicente Planelles

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Vicente Planelles

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6 March 2020

author name string

Zachary B Davis

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6 March 2020

Hamish Scott

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6 March 2020

Amanda Mertsching

4

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6 March 2020

Julie Boucau

5

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6 March 2020

Daniel Wambua

6

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6 March 2020

Kerry S Campbell

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6 March 2020

publication date

1 February 2016

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6 March 2020

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6 March 2020

12

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6 March 2020

2

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6 March 2020

e1005421

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6 March 2020

copyright license

Creative Commons Attribution 4.0 International

1 February 2016

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April 2022 Public Data File from Crossref

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0 references

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HIV-1 adaptation to NK-cell-mediated immune pressure

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Degranulation of natural killer cells following interaction with HIV-1-infected cells is hindered by downmodulation of NTB-A by Vpu.

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CD16- natural killer cells: enrichment in mucosal and secondary lymphoid tissues and altered function during chronic SIV infection

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HIV-1 Vpr triggers natural killer cell-mediated lysis of infected cells through activation of the ATR-mediated DNA damage response

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Differential HIV epitope processing in monocytes and CD4 T cells affects cytotoxic T lymphocyte recognition

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Beyond licensing and disarming: a quantitative view on NK-cell education

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Structural basis for NKG2A/CD94 recognition of HLA-E.

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Portable flanking sequences modulate CTL epitope processing

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A subset of natural killer cells achieves self-tolerance without expressing inhibitory receptors specific for self-MHC molecules

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Characterization of CD56-/CD16+ natural killer (NK) cells: a highly dysfunctional NK subset expanded in HIV-infected viremic individuals

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Heat shock protein 70 protects cells from cell cycle arrest and apoptosis induced by human immunodeficiency virus type 1 viral protein R

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Analysis of HLA-E peptide-binding specificity and contact residues in bound peptide required for recognition by CD94/NKG2.

1 reference

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A signal peptide derived from hsp60 binds HLA-E and interferes with CD94/NKG2A recognition

1 reference

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Cutting edge: the human cytomegalovirus UL40 gene product contains a ligand for HLA-E and prevents NK cell-mediated lysis

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The selective downregulation of class I major histocompatibility complex proteins by HIV-1 protects HIV-infected cells from NK cells

1 reference

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27 July 2018

HLA-E is a major ligand for the natural killer inhibitory receptor CD94/NKG2A

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4735451

27 July 2018

HLA-E binds to natural killer cell receptors CD94/NKG2A, B and C

1 reference

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Human diversity in killer cell inhibitory receptor genes

1 reference

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27 July 2018

The human major histocompatibility complex class Ib molecule HLA-E binds signal sequence-derived peptides with primary anchor residues at positions 2 and 9

1 reference

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27 July 2018

Human natural killer cell receptors involved in MHC class I recognition are disulfide-linked heterodimers of CD94 and NKG2 subunits

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In vivo gene delivery and stable transduction of nondividing cells by a lentiviral vector

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Apparent ineffectiveness of natural killer cells vis-à-vis retrovirus-infected targets

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Variable processing and cross-presentation of HIV by dendritic cells and macrophages shapes CTL immunodominance and immune escape.

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Eradicating HIV-1 infection: seeking to clear a persistent pathogen

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Mechanisms of HIV protein degradation into epitopes: implications for vaccine design

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Sequence-specific alterations of epitope production by HIV protease inhibitors

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HIV protective KIR3DL1/S1-HLA-B genotypes influence NK cell-mediated inhibition of HIV replication in autologous CD4 targets

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Chronic HIV-1 viremia reverses NKG2A/NKG2C ratio on natural killer cells in patients with human cytomegalovirus co-infection.

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1 reference

https://pubmed.ncbi.nlm.nih.gov/26828202

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Comparative studies of human FcRIII-positive and negative natural killer cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/26828202

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

HIV-1 infection leads to increased HLA-E expression resulting in impaired function of natural killer cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/26828202

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Switch from inhibitory to activating NKG2 receptor expression in HIV-1 infection: lack of reversion with highly active antiretroviral therapy

1 reference

https://pubmed.ncbi.nlm.nih.gov/26828202

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PPAT.1005421

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26828202%20AND%20SRC:MED&resulttype=core&format=json

6 March 2020

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26828202%20AND%20SRC:MED&resulttype=core&format=json

6 March 2020

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26828202%20AND%20SRC:MED&resulttype=core&format=json

6 March 2020

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