(Q36301907)

13 August 2017

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title

JAK-STAT pathway in cardiac ischemic stress (English)

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13 August 2017

author name string

Radha Ananthakrishnan

1

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13 August 2017

Kellie Hallam

2

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13 August 2017

Qing Li

3

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13 August 2017

Ravichandran Ramasamy

4

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13 August 2017

publication date

2 November 2005

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13 August 2017

published in

Vascular Pharmacology

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13 August 2017

volume

43

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13 August 2017

issue

5

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13 August 2017

page(s)

353-356

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STAT3beta, a splice variant of transcription factor STAT3, is a dominant negative regulator of transcription

13 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Prevention of Organ Allograft Rejection by a Specific Janus Kinase 3 Inhibitor

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

STATs and gene regulation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

The functional role of the JAK-STAT pathway in post-infarction remodeling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Ischemic protection and myofibrillar cardiomyopathy: dose-dependent effects of in vivo deltaPKC inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Role of STAT3 in ischemic preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Signal transducer and activator of transcription 3 is required for myocardial capillary growth, control of interstitial matrix deposition, and heart protection from ischemic injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

STAT proteins and transcriptional responses to extracellular signals

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Aldose reductase activation is a key component of myocardial response to ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Sorbitol dehydrogenase: a novel target for adjunctive protection of ischemic myocardium

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Central role for aldose reductase pathway in myocardial ischemic injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Aldose reductase pathway mediates JAK-STAT signaling: a novel axis in myocardial ischemic injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Additive protection of the ischemic heart ex vivo by combined treatment with delta-protein kinase C inhibitor and epsilon-protein kinase C activator

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Cardiomyocyte-restricted knockout of STAT3 results in higher sensitivity to inflammation, cardiac fibrosis, and heart failure with advanced age

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Cardiotrophin-1 (CT-1) can protect the adult heart from injury when added both prior to ischaemia and at reperfusion.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Mutations of Jak-3 gene in patients with autosomal severe combined immune deficiency (SCID)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

JAK/STAT signaling is associated with cardiac dysfunction during ischemia and reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Activation of signal transducer and activator of transcription (STAT) pathways in failing human hearts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Mutation of Jak3 in a patient with SCID: essential role of Jak3 in lymphoid development.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Transcriptional responses to polypeptide ligands: the JAK-STAT pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

High glucose augments the angiotensin II-induced activation of JAK2 in vascular smooth muscle cells via the polyol pathway.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Genetic depletion of cardiac myocyte STAT-3 abolishes classical preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Cardiotrophin-1 induces heat shock protein accumulation in cultured cardiac cells and protects them from stressful stimuli.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Opposing actions of STAT-1 and STAT-3 on the Bcl-2 and Bcl-x promoters.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Ischemia-induced STAT-1 Expression and Activation Play a Critical Role in Cardiomyocyte Apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Induction of apoptosis and Fas receptor/Fas ligand expression by ischemia/reperfusion in cardiac myocytes requires serine 727 of the STAT-1 transcription factor but not tyrosine 701.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

Two distinct mechanisms mediate a differential regulation of protein kinase C isozymes in acute and prolonged myocardial ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

An essential role of the JAK-STAT pathway in ischemic preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VPH.2005.08.020

10.1016/J.VPH.2005.08.020

7 January 2021

Identifiers

DOI

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13 August 2017

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