(Q36315214)

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The 29-nucleotide deletion present in human but not in animal severe acute respiratory syndrome coronaviruses disrupts the functional expression of open reading frame 8.

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Europe PubMed Central

PMC publication ID

13 August 2017

The 29-nucleotide deletion present in human but not in animal severe acute respiratory syndrome coronaviruses disrupts the functional expression of open reading frame 8. (English)

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

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protein non-structural 8a [SARS-Cov]

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Peter J M Rottier

3

object named as

Peter J M Rottier

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Cornelis A. M. de Haan

2

object named as

Cornelis A M de Haan

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

author name string

Monique Oostra

1

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

language of work or name

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publication date

10 October 2007

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Journal of Virology

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

81

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

24

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

13876-13888

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Open reading frame 8a of the human severe acute respiratory syndrome coronavirus not only promotes viral replication but also induces apoptosis

1 reference

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Severe acute respiratory syndrome coronavirus open reading frame (ORF) 3b, ORF 6, and nucleocapsid proteins function as interferon antagonists

1 reference

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The ORF7b protein of severe acute respiratory syndrome coronavirus (SARS-CoV) is expressed in virus-infected cells and incorporated into SARS-CoV particles

1 reference

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The human severe acute respiratory syndrome coronavirus (SARS-CoV) 8b protein is distinct from its counterpart in animal SARS-CoV and down-regulates the expression of the envelope protein in infected cells

1 reference

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Severe acute respiratory syndrome coronavirus 7a accessory protein is a viral structural protein

1 reference

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Expression and functional characterization of the putative protein 8b of the severe acute respiratory syndrome-associated coronavirus

1 reference

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1 reference

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Severe acute respiratory syndrome coronavirus group-specific open reading frames encode nonessential functions for replication in cell cultures and mice

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A severe acute respiratory syndrome-associated coronavirus-specific protein enhances virulence of an attenuated murine coronavirus

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Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein

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Civets are equally susceptible to experimental infection by two different severe acute respiratory syndrome coronavirus isolates

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Overexpression of 7a, a protein specifically encoded by the severe acute respiratory syndrome coronavirus, induces apoptosis via a caspase-dependent pathway

1 reference

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Live, attenuated coronavirus vaccines through the directed deletion of group-specific genes provide protection against feline infectious peritonitis

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Expression, post-translational modification and biochemical characterization of proteins encoded by subgenomic mRNA8 of the severe acute respiratory syndrome coronavirus

1 reference

https://pubmed.ncbi.nlm.nih.gov/17928347

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Downstream ribosomal entry for translation of coronavirus TGEV gene 3b

1 reference

https://pubmed.ncbi.nlm.nih.gov/17928347

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The group-specific murine coronavirus genes are not essential, but their deletion, by reverse genetics, is attenuating in the natural host

1 reference

https://pubmed.ncbi.nlm.nih.gov/17928347

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.01631-07

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

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