(Q36315214)

English

The 29-nucleotide deletion present in human but not in animal severe acute respiratory syndrome coronaviruses disrupts the functional expression of open reading frame 8.

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scholarly article

1 reference

Europe PubMed Central

13 August 2017

The 29-nucleotide deletion present in human but not in animal severe acute respiratory syndrome coronaviruses disrupts the functional expression of open reading frame 8. (English)

1 reference

Europe PubMed Central

13 August 2017

0 references

protein non-structural 8a [SARS-Cov]

0 references

Peter J M Rottier

3

object named as

Peter J M Rottier

1 reference

Europe PubMed Central

13 August 2017

Cornelis A. M. de Haan

2

object named as

Cornelis A M de Haan

1 reference

Europe PubMed Central

13 August 2017

author name string

Monique Oostra

1

1 reference

Europe PubMed Central

13 August 2017

language of work or name

0 references

publication date

10 October 2007

1 reference

Europe PubMed Central

13 August 2017

Journal of Virology

1 reference

Europe PubMed Central

13 August 2017

81

1 reference

Europe PubMed Central

13 August 2017

24

1 reference

Europe PubMed Central

13 August 2017

13876-13888

1 reference

Europe PubMed Central

13 August 2017

Open reading frame 8a of the human severe acute respiratory syndrome coronavirus not only promotes viral replication but also induces apoptosis

1 reference

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Antigen presentation and the ubiquitin-proteasome system in host-pathogen interactions

1 reference

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Severe acute respiratory syndrome coronavirus open reading frame (ORF) 3b, ORF 6, and nucleocapsid proteins function as interferon antagonists

1 reference

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The ORF7b protein of severe acute respiratory syndrome coronavirus (SARS-CoV) is expressed in virus-infected cells and incorporated into SARS-CoV particles

1 reference

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The human severe acute respiratory syndrome coronavirus (SARS-CoV) 8b protein is distinct from its counterpart in animal SARS-CoV and down-regulates the expression of the envelope protein in infected cells

1 reference

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Severe acute respiratory syndrome coronavirus 7a accessory protein is a viral structural protein

1 reference

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Expression and functional characterization of the putative protein 8b of the severe acute respiratory syndrome-associated coronavirus

1 reference

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Severe acute respiratory syndrome coronavirus group-specific open reading frames encode nonessential functions for replication in cell cultures and mice

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A severe acute respiratory syndrome-associated coronavirus-specific protein enhances virulence of an attenuated murine coronavirus

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Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein

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Civets are equally susceptible to experimental infection by two different severe acute respiratory syndrome coronavirus isolates

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Overexpression of 7a, a protein specifically encoded by the severe acute respiratory syndrome coronavirus, induces apoptosis via a caspase-dependent pathway

1 reference

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A novel severe acute respiratory syndrome coronavirus protein, U274, is transported to the cell surface and undergoes endocytosis

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Suppression of the induction of alpha, beta, and lambda interferons by the NS1 and NS2 proteins of human respiratory syncytial virus in human epithelial cells and macrophages [corrected]

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Live, attenuated coronavirus vaccines through the directed deletion of group-specific genes provide protection against feline infectious peritonitis

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Transmissible gastroenteritis coronavirus gene 7 is not essential but influences in vivo virus replication and virulence.

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At least six nucleotides preceding the AUG initiator codon enhance translation in mammalian cells

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The scanning model for translation: an update

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Expression, post-translational modification and biochemical characterization of proteins encoded by subgenomic mRNA8 of the severe acute respiratory syndrome coronavirus

1 reference

https://pubmed.ncbi.nlm.nih.gov/17928347

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Downstream ribosomal entry for translation of coronavirus TGEV gene 3b

1 reference

https://pubmed.ncbi.nlm.nih.gov/17928347

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The group-specific murine coronavirus genes are not essential, but their deletion, by reverse genetics, is attenuating in the natural host

1 reference

https://pubmed.ncbi.nlm.nih.gov/17928347

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.01631-07

1 reference

Europe PubMed Central

13 August 2017

1 reference

Europe PubMed Central

13 August 2017

1 reference

Europe PubMed Central

13 August 2017

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