(Q36657811)

15 August 2017

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title

Axonal conduction and injury in multiple sclerosis: the role of sodium channels (English)

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15 August 2017

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Stephen G Waxman

1

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15 August 2017

language of work or name

English

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publication date

1 December 2006

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Nature Reviews Neuroscience

15 August 2017

published in

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15 August 2017

volume

7

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15 August 2017

issue

12

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15 August 2017

page(s)

932-941

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Conduction in myelinated, unmyelinated, and demyelinated fibers

15 August 2017

cites work

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Multiple sclerosis and chronic autoimmune encephalomyelitis: a comparative quantitative study of axonal injury in active, inactive, and remyelinated lesions

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31 March 2020

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Reorganization of the Axon Membrane in Demyelinated Peripheral Nerve Fibers: Morphological Evidence

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Persistent functional deficit in multiple sclerosis and autosomal dominant cerebellar ataxia is associated with axon loss

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Low density of sodium channels supports action potential conduction in axons of neonatal rat optic nerve

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Compact myelin dictates the differential targeting of two sodium channel isoforms in the same axon.

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Functional specialization of the axon initial segment by isoform-specific sodium channel targeting.

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Sodium channel Na(v)1.6 is localized at nodes of ranvier, dendrites, and synapses

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Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE.

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31 March 2020

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31 March 2020

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31 March 2020

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Sensory neuron-specific sodium channel SNS is abnormally expressed in the brains of mice with experimental allergic encephalomyelitis and humans with multiple sclerosis

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Annexin II light chain regulates sensory neuron-specific sodium channel expression

31 March 2020

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Annexin II/p11 is up-regulated in Purkinje cells in EAE and MS

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A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

31 March 2020

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31 March 2020

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1 reference

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31 March 2020

1 reference

Initiation and spread of sodium action potentials in cerebellar Purkinje cells

31 March 2020

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31 March 2020

1 reference

A missense mutation in the sodium channel Scn8a is responsible for cerebellar ataxia in the mouse mutant jolting.

31 March 2020

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Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar Purkinje cells

31 March 2020

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NGF has opposing effects on Na+ channel III and SNS gene expression in spinal sensory neurons

31 March 2020

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Rescue of alpha-SNS sodium channel expression in small dorsal root ganglion neurons after axotomy by nerve growth factor in vivo

31 March 2020

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31 March 2020

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31 March 2020

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Upregulation and colocalization of p75 and Nav1.8 in Purkinje neurons in experimental autoimmune encephalomyelitis.

31 March 2020

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Expression of trk receptors in the developing and adult human central and peripheral nervous system

31 March 2020

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An immunohistochemical study of the nerve growth factor receptor in developing rats

31 March 2020

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Sodium channels contribute to microglia/macrophage activation and function in EAE and MS.

31 March 2020

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31 March 2020

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Microglial cells qualify as the stimulators of unprimed CD4+ and CD8+ T lymphocytes in the central nervous system

31 March 2020

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Prevention of experimental allergic encephalomyelitis by targeting nitric oxide and peroxynitrite: implications for the treatment of multiple sclerosis

31 March 2020

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Microglia-derived macrophages in early multiple sclerosis plaques

31 March 2020

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Voltage-gated currents expressed by rat microglia in culture

31 March 2020

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Sodium channel in isolated human brain macrophages (microglia).

31 March 2020

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5-HT3 receptor-channels coupled with Na+ influx in human T cells: role in T cell activation

31 March 2020

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Manipulating neuroinflammatory reactions in the injured spinal cord: back to basics

31 March 2020

1 reference

https://api.fatcat.wiki/v0/release/w7hwqdrb25cs3ooievlajvzdhu

31 March 2020

Identifiers

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Dimensions Publication ID

1002258781

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Fatcat ID

release_w7hwqdrb25cs3ooievlajvzdhu

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https://api.fatcat.wiki/v0/release/w7hwqdrb25cs3ooievlajvzdhu

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30 July 2022

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https://api.fatcat.wiki/v0/release/w7hwqdrb25cs3ooievlajvzdhu

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