(Q36862386)

16 August 2017

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title

Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection (English)

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16 August 2017

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Sabzali Javadov

1

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16 August 2017

Morris Karmazyn

series ordinal

2

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16 August 2017

publication date

1 January 2007

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Cellular Physiology and Biochemistry

16 August 2017

published in

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16 August 2017

volume

20

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16 August 2017

issue

1-4

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16 August 2017

page(s)

1-22

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Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

16 August 2017

cites work

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The mitochondrial death pathway and cardiac myocyte apoptosis

21 January 2018

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The mitochondrial permeability transition pore and its role in cell death

21 January 2018

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Calcium, mitochondria and reperfusion injury: a pore way to die.

21 January 2018

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Role of apoptosis in remodeling after myocardial infarction

21 January 2018

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Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

21 January 2018

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Further evidence that cyclosporin A protects mitochondria from calcium overload by inhibiting a matrix peptidyl-prolyl cis-trans isomerase. Implications for the immunosuppressive and toxic effects of cyclosporin

21 January 2018

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Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

21 January 2018

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Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

21 January 2018

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Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

21 January 2018

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Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia

21 January 2018

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The mitochondrial permeability transition.

21 January 2018

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Calcium-dependent opening of a non-specific pore in the mitochondrial inner membrane is inhibited at pH values below 7. Implications for the protective effect of low pH against chemical and hypoxic cell damage

21 January 2018

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Contribution of the mitochondrial permeability transition to lethal injury after exposure of hepatocytes to t-butylhydroperoxide

21 January 2018

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The myocardial Na(+)-H(+) exchange: structure, regulation, and its role in heart disease

21 January 2018

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Mitochondrial ion channels: gatekeepers of life and death

21 January 2018

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Calcium, ATP, and ROS: a mitochondrial love-hate triangle

21 January 2018

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Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury

21 January 2018

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Amphipathic metabolites and membrane dysfunction in ischemic myocardium

21 January 2018

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Influence of L-carnitine and its derivatives on myocardial metabolism and function in ischemic heart disease and during cardiopulmonary bypass

21 January 2018

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Antihypertrophic effect of Na+/H+ exchanger isoform 1 inhibition is mediated by reduced mitogen-activated protein kinase activation secondary to improved mitochondrial integrity and decreased generation of mitochondrial-derived reactive oxygen speci

21 January 2018

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Autoactivation of procaspase-9 by Apaf-1-mediated oligomerization

21 January 2018

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The mitochondrial apoptosome: a killer unleashed by the cytochrome seas

21 January 2018

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Role of cytochrome c and dATP/ATP hydrolysis in Apaf-1-mediated caspase-9 activation and apoptosis

21 January 2018

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

21 January 2018

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Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

21 January 2018

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Endonuclease G: a mitochondrial protein released in apoptosis and involved in caspase-independent DNA degradation.

21 January 2018

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Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain

21 January 2018

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Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

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Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

21 January 2018

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Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

21 January 2018

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Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane

21 January 2018

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Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

21 January 2018

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VDAC2 inhibits BAK activation and mitochondrial apoptosis

21 January 2018

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Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

21 January 2018

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Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux

21 January 2018

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Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury

21 January 2018

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Interference with calcium-dependent mitochondrial bioenergetics in cardiac myocytes isolated from doxorubicin-treated rats

21 January 2018

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Mitochondrial Ca(2+)-induced Ca(2+) release mediated by the Ca(2+) uniporter

21 January 2018

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The mitochondrial calcium uniporter is a highly selective ion channel.

21 January 2018

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Antioxidant MCI-186 inhibits mitochondrial permeability transition pore and upregulates Bcl-2 expression

21 January 2018

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Ischemic preconditioning: from basic mechanisms to clinical applications

21 January 2018

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Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

21 January 2018

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Inhibit GSK-3beta or there's heartbreak dead ahead

21 January 2018

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Tissue protection mediated by mitochondrial K+ channels.

21 January 2018

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21 January 2018

Identifiers

DOI

10.1159/000103747

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16 August 2017

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