(Q37519545)

20 August 2017

1 reference

title

Is Alzheimer's disease a result of presynaptic failure? Synaptic dysfunctions induced by oligomeric beta-amyloid (English)

1 reference

20 August 2017

0 references

Volker Nimmrich

1

1 reference

20 August 2017

Ulrich Ebert

series ordinal

2

1 reference

20 August 2017

publication date

1 January 2009

1 reference

Reviews in the Neurosciences

20 August 2017

published in

1 reference

20 August 2017

volume

20

1 reference

20 August 2017

issue

1

1 reference

20 August 2017

page(s)

1-12

1 reference

Anatomical and physiological plasticity of dendritic spines

20 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Activity-dependent regulation of dendritic spine density on cortical pyramidal neurons in organotypic slice cultures

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Presynaptic NMDA receptors modulate glutamate release from primary sensory neurons in rat spinal cord dorsal horn.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Globular amyloid beta-peptide oligomer - a homogenous and stable neuropathological protein in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Human amyloid-beta synthesis and clearance rates as measured in cerebrospinal fluid in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Synapse elimination accompanies functional plasticity in hippocampal neurons

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Tonic facilitation of glutamate release by presynaptic N-methyl-D-aspartate autoreceptors in the entorhinal cortex.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Do thin spines learn to be mushroom spines that remember?

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Rapid, concurrent alterations in pre- and postsynaptic structure induced by naturally-secreted amyloid-beta protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Cellular and subcellular localization of the 2B-subunit of the NMDA receptor in the adult rat telencephalon.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Accelerating amyloid-beta fibrillization reduces oligomer levels and functional deficits in Alzheimer disease mouse models.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Contribution of different calcium channels to long-term potentiation in superior cervical ganglion of the rat.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Strategies for disease modification in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Plasticity in the human central nervous system

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Abeta oligomers induce neuronal oxidative stress through an N-methyl-D-aspartate receptor-dependent mechanism that is blocked by the Alzheimer drug memantine

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Novel presynaptic mechanisms for coincidence detection in synaptic plasticity

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Optical quantal analysis reveals a presynaptic component of LTP at hippocampal Schaffer-associational synapses

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Evidence that long-term potentiation occurs within individual hippocampal synapses during learning.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Postsynaptic contributions to hippocampal network hyperexcitability induced by chronic activity blockade in vivo.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Abeta-globulomers are formed independently of the fibril pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Abnormal phosphorylation of the microtubule-associated protein tau (tau) in Alzheimer cytoskeletal pathology

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Alzheimer's disease: the amyloid cascade hypothesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Presynaptic induction of heterosynaptic associative plasticity in the mammalian brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Absence of synaptophysin near cortical neurons containing oligomer Abeta in Alzheimer's disease brain.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Beta-amyloid-induced dynamin 1 depletion in hippocampal neurons. A potential mechanism for early cognitive decline in Alzheimer disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

beta-Amyloid-induced dynamin 1 degradation is mediated by N-methyl-D-aspartate receptors in hippocampal neurons.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Beta-amyloid disrupted synaptic vesicle endocytosis in cultured hippocampal neurons.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Amyloid beta protein immunotherapy neutralizes Abeta oligomers that disrupt synaptic plasticity in vivo.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Abeta oligomer-mediated long-term potentiation impairment involves protein phosphatase 1-dependent mechanisms.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Soluble Abeta oligomers ultrastructurally localize to cell processes and might be related to synaptic dysfunction in Alzheimer's disease brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Synaptic targeting by Alzheimer's-related amyloid beta oligomers.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

A Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Visual stimuli-induced LTD of GABAergic synapses mediated by presynaptic NMDA receptors.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

A specific amyloid-beta protein assembly in the brain impairs memory

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Plaque-bearing mice with reduced levels of oligomeric amyloid-beta assemblies have intact memory function.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Presynaptic mechanisms involved in the expression of STP and LTP at CA1 synapses in the hippocampus.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Neurosteroid-induced plasticity of immature synapses via retrograde modulation of presynaptic NMDA receptors.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Amyloid plaque core protein in Alzheimer disease and Down syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Factors critical for the plasticity of dendritic spines and memory storage.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Soluble pool of Abeta amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Amyloid-degrading enzymes as therapeutic targets in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Amyloid beta oligomers (A beta(1-42) globulomer) suppress spontaneous synaptic activity by inhibition of P/Q-type calcium currents.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Inhibition of Calpain Prevents N-Methyl-D-aspartate-Induced Degeneration of the Nucleus Basalis and Associated Behavioral Dysfunction

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Presenile dementia and Alzheimer's disease in mongolism.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

No global neocortical nerve cell loss in brains from patients with senile dementia of Alzheimer's type.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Long-term potentiation involves increases in the probability of neurotransmitter release

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Dendritic spines shaped by synaptic activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Dendritic spine formation and pruning: common cellular mechanisms?

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Formation of dendritic spines in cultured striatal neurons depends on excitatory afferent activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Regulation of NMDA receptor trafficking by amyloid-beta

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

A new amyloid beta variant favoring oligomerization in Alzheimer's-type dementia.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Effects of secreted oligomers of amyloid beta-protein on hippocampal synaptic plasticity: a potent role for trimers.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Fibrillar amyloid deposition leads to local synaptic abnormalities and breakage of neuronal branches.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

A beta oligomers - a decade of discovery

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

The levels of soluble versus insoluble brain Abeta distinguish Alzheimer's disease from normal and pathologic aging.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Soluble oligomers of beta amyloid (1-42) inhibit long-term potentiation but not long-term depression in rat dentate gyrus.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

NSAIDs: small molecules for prevention of Alzheimer's disease or precursors for future drug development?

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Roscovitine: a novel regulator of P/Q-type calcium channels and transmitter release in central neurons

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Defects in expression of genes related to synaptic vesicle trafficking in frontal cortex of Alzheimer's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Shrinkage of dendritic spines associated with long-term depression of hippocampal synapses

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

Dendritic Spine Pathology: Cause or Consequence of Neurological Disorders?

21 January 2018

1 reference

https://api.crossref.org/works/10.1515%2FREVNEURO.2009.20.1.1

10.1515/REVNEURO.2009.20.1.1

21 January 2018

Identifiers

DOI

1 reference

20 August 2017

1 reference