(Q37623566)

English

Differential effects of the mitochondrial uncoupling agent, 2,4-dinitrophenol, or the nitroxide antioxidant, Tempol, on synaptic or nonsynaptic mitochondria after spinal cord injury

scientific article published on January 2009

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

Differential effects of the mitochondrial uncoupling agent, 2,4-dinitrophenol, or the nitroxide antioxidant, Tempol, on synaptic or nonsynaptic mitochondria after spinal cord injury (English)

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

1 reference

based on heuristic

inferred from title

author name string

Samir P Patel

1

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

Patrick G Sullivan

2

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

Jignesh D Pandya

3

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

Alexander G Rabchevsky

4

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

publication date

1 January 2009

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

Journal of Neuroscience Research

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

87

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

1

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

130-140

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

Mitochondrial permeability transition in CNS trauma: cause or effect of neuronal cell death?

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

23 August 2017

Mitochondrial uncoupling as a potential therapeutic target in acute central nervous system injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

23 August 2017

Mitochondrial uncoupling as a therapeutic target following neuronal injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

23 August 2017

Degenerative and regenerative mechanisms governing spinal cord injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

23 August 2017

Mitochondrial uncoupling protein-2 protects the immature brain from excitotoxic neuronal death.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

23 August 2017

Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

23 August 2017

Pathophysiology of spinal cord trauma

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

28 September 2017

Free radicals in CNS injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

28 September 2017

The double-edged role of nitric oxide in brain function and superoxide-mediated injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

28 September 2017

NMDA-induced superoxide production and neurotoxicity in cultured rat hippocampal neurons: role of mitochondria.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

25 June 2018

Uncoupling: new approaches to an old problem of bioenergetics

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

25 June 2018

Mitochondrial production of reactive oxygen species in cortical neurons following exposure to N-methyl-D-aspartate

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

25 June 2018

Inhibition of lipid peroxidation in CNS trauma.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

25 June 2018

High cyclophilin D content of synaptic mitochondria results in increased vulnerability to permeability transition.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Temporal characterization of mitochondrial bioenergetics after spinal cord injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Post-Injury Administration of Mitochondrial Uncouplers Increases Tissue Sparing and Improves Behavioral Outcome following Traumatic Brain Injury in Rodents.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Isoflurane preconditioning uncouples mitochondria and protects against hypoxia-reoxygenation.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Cyclosporin A increases mitochondrial calcium uptake capacity in cortical astrocytes but not cerebellar granule neurons

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Synaptic mitochondria are more susceptible to Ca2+overload than nonsynaptic mitochondria

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Tempol, a nitroxide antioxidant, improves locomotor and histological outcomes after spinal cord contusion in rats

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

The mitochondrial uncoupling agent 2,4-dinitrophenol improves mitochondrial function, attenuates oxidative damage, and increases white matter sparing in the contused spinal cord

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Polyethylene glycol improves function and reduces oxidative stress in synaptosomal preparations following spinal cord injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Intrinsic differences in brain and spinal cord mitochondria: Implication for therapeutic interventions.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Importance of renal mitochondria in the reduction of TEMPOL, a nitroxide radical.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Calcium-induced Cytochrome c release from rat brain mitochondria is altered by digitonin

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

The Mechanism by which 4-hydroxy-2,2,6,6-tetramethylpiperidene-1-oxyl (tempol) diverts peroxynitrite decomposition from nitrating to nitrosating species

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Mitochondria and neuronal glutamate excitotoxicity.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5291118

1 September 2018

Spectrophotometric Assay of Cytochrome c Oxidase

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Neuronal excitotoxicity: the role of mitochondria

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

4-Hydroxy-2,2,6,6-tetramethylpiperidine-1-oxyl (Tempol) inhibits peroxynitrite-mediated phenol nitration

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Glutamate-induced neuron death requires mitochondrial calcium uptake

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Pretreatment with the cyclosporin derivative, NIM811, improves the function of synaptic mitochondria following spinal cord contusion in rats

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Mitochondrial damage and dysfunction in traumatic brain injury

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Differences in the reduction kinetics of incorporated spin labels in undifferentiated and differentiated mouse neuroblastoma cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/18709657

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1002/JNR.21814

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

21 August 2017

ResearchGate publication ID

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