(Q44918596)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15174070%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

title

Intrinsic differences in brain and spinal cord mitochondria: Implication for therapeutic interventions (English)

1 reference

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29 December 2019

1 reference

3

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29 December 2019

Ronald P. Hart

6

1 reference

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29 December 2019

author name string

Patrick G Sullivan

1

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29 December 2019

Alexander G Rabchevsky

2

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29 December 2019

Mark Lovell

4

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29 December 2019

Ajeet Sodhi

5

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29 December 2019

Stephen W Scheff

7

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29 December 2019

language of work or name

English

0 references

publication date

1 July 2004

1 reference

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The Journal of Comparative Neurology

29 December 2019

published in

1 reference

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29 December 2019

volume

474

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29 December 2019

issue

4

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29 December 2019

page(s)

524-534

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Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

29 December 2019

cites work

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Cytochrome c release from brain mitochondria is independent of the mitochondrial permeability transition.

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Impaired mitochondrial function, oxidative stress and altered antioxidant enzyme activities following traumatic spinal cord injury.

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Limitations of cyclosporin A inhibition of the permeability transition in CNS mitochondria

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Calcium-induced cytochrome c release from CNS mitochondria is associated with the permeability transition and rupture of the outer membrane

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Postinjury cyclosporin A administration limits axonal damage and disconnection in traumatic brain injury

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AMPA exposures induce mitochondrial Ca(2+) overload and ROS generation in spinal motor neurons in vitro.

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Oxidative stress, glutamate, and neurodegenerative disorders

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Mitochondrial production of reactive oxygen species in cortical neurons following exposure to N-methyl-D-aspartate

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The role of excitatory amino acids and NMDA receptors in traumatic brain injury

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The mitochondrion: is it central to apoptosis?

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Differences in the activation of the mitochondrial permeability transition among brain regions in the rat correlate with selective vulnerability.

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Oxidative stress, thiol reagents, and membrane potential modulate the mitochondrial permeability transition by affecting nucleotide binding to the adenine nucleotide translocase

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Importance of pharmacologic considerations in the evaluation of new treatments for acute spinal cord injury

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APOPTOSIS: Death of a Monopoly?

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Zn(2+) induces permeability transition pore opening and release of pro-apoptotic peptides from neuronal mitochondria.

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Mitochondrial manganese superoxide dismutase prevents neural apoptosis and reduces ischemic brain injury: suppression of peroxynitrite production, lipid peroxidation, and mitochondrial dysfunction.

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Oxidative stress-associated impairment of proteasome activity during ischemia-reperfusion injury

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Preparation of synaptic and nonsynaptic mitochondria from mammalian brain

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Mitochondrial superoxide production in kainate-induced hippocampal damage.

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7 January 2021

Mitochondria and neuronal glutamate excitotoxicity.

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7 January 2021

An intrathecal bolus of cyclosporin A before injury preserves mitochondrial integrity and attenuates axonal disruption in traumatic brain injury

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7 January 2021

Cyclosporin A limits calcium-induced axonal damage following traumatic brain injury

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7 January 2021

Mitochondrial transmembrane potential and free radical production in excitotoxic neurodegeneration

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7 January 2021

Cyclosporin A treatment following spinal cord injury to the rat: behavioral effects and stereological assessment of tissue sparing

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7 January 2021

Efficacy of methylprednisolone therapy for the injured rat spinal cord

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7 January 2021

Creatine diet supplement for spinal cord injury: influences on functional recovery and tissue sparing in rats

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7 January 2021

Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation

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7 January 2021

Product differentiation by analysis of DNA melting curves during the polymerase chain reaction

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7 January 2021

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

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7 January 2021

Effects of oxidants and glutamate receptor activation on mitochondrial membrane potential in rat forebrain neurons.

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7 January 2021

Cyclosporin A significantly ameliorates cortical damage following experimental traumatic brain injury in rodents.

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7 January 2021

NMDA-induced superoxide production and neurotoxicity in cultured rat hippocampal neurons: role of mitochondria.

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7 January 2021

Modulation of mitochondrial function by endogenous Zn2+ pools.

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7 January 2021

Thiol oxidation and loss of mitochondrial complex I precede excitatory amino acid-mediated neurodegeneration.

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7 January 2021

Glutamate-induced neuron death requires mitochondrial calcium uptake

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7 January 2021

Traumatic brain injury alters synaptic homeostasis: implications for impaired mitochondrial and transport function.

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7 January 2021

Exacerbation of damage and altered NF-kappaB activation in mice lacking tumor necrosis factor receptors after traumatic brain injury.

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7 January 2021

Cyclosporin A attenuates acute mitochondrial dysfunction following traumatic brain injury.

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7 January 2021

Dietary supplement creatine protects against traumatic brain injury

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7 January 2021

Dose-response curve and optimal dosing regimen of cyclosporin A after traumatic brain injury in rats

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7 January 2021

Cytochrome c release and caspase activation after traumatic brain injury.

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7 January 2021

Mitochondrial uncoupling protein-2 protects the immature brain from excitotoxic neuronal death.

1 reference

7 January 2021

Identifiers

DOI

10.1002/CNE.20130

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15174070%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

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