(Q37837590)

24 August 2017

1 reference

title

Glutamine: pleiotropic roles in tumor growth and stress resistance (English)

1 reference

24 August 2017

1 reference

Naval P Shanware

1

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24 August 2017

Andrew R Mullen

series ordinal

2

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24 August 2017

Ralph J DeBerardinis

series ordinal

3

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24 August 2017

Robert T Abraham

series ordinal

4

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24 August 2017

publication date

8 February 2011

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Journal of Molecular Medicine

24 August 2017

published in

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24 August 2017

volume

89

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24 August 2017

issue

3

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24 August 2017

page(s)

229-236

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The control of the metabolic switch in cancers by oncogenes and tumor suppressor genes

24 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Q's next: the diverse functions of glutamine in metabolism, cell biology and cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glutamine and glucose metabolism during thymocyte proliferation. Pathways of glutamine and glutamate metabolism

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Beyond aerobic glycolysis: transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Quiescent fibroblasts exhibit high metabolic activity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glioblastoma cells require glutamate dehydrogenase to survive impairments of glucose metabolism or Akt signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glucose addiction of TSC null cells is caused by failed mTORC1-dependent balancing of metabolic demand with supply

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glutaminolysis yields a metabolic by-product that stimulates autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Cellular metabolic stress: considering how cells respond to nutrient excess.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

c-Myc suppression of miR-23a/b enhances mitochondrial glutaminase expression and glutamine metabolism

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Deficiency in glutamine but not glucose induces MYC-dependent apoptosis in human cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Antisense glutaminase inhibition decreases glutathione antioxidant capacity and increases apoptosis in Ehrlich ascitic tumour cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

c-Myc phosphorylation is required for cellular response to oxidative stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Myc stimulates nuclearly encoded mitochondrial genes and mitochondrial biogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

DNA damage signaling and p53-dependent senescence after prolonged beta-interferon stimulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

The DNA damage signaling pathway is a critical mediator of oncogene-induced senescence.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Targeting mitochondrial glutaminase activity inhibits oncogenic transformation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glutamine targeting inhibits systemic metastasis in the VM-M3 murine tumor model

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glutaminase 2, a novel p53 target gene regulating energy metabolism and antioxidant function

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Phosphate-activated glutaminase (GLS2), a p53-inducible regulator of glutamine metabolism and reactive oxygen species

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glutamine homeostasis and mitochondrial dynamics

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Regulation of glutaminase activity and glutamine metabolism

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Autophagy fights disease through cellular self-digestion

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Mammalian target of rapamycin: discovery of rapamycin reveals a signaling pathway important for normal and cancer cell growth

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

mTOR regulation of autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Bidirectional transport of amino acids regulates mTOR and autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

ASCT2 silencing regulates mammalian target-of-rapamycin growth and survival signaling in human hepatoma cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Beyond rapalog therapy: preclinical pharmacology and antitumor activity of WYE-125132, an ATP-competitive and specific inhibitor of mTORC1 and mTORC2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Glutamine increases autophagy under Basal and stressed conditions in intestinal epithelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Current concepts in the pathogenesis of urea cycle disorders

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Ammonia metabolism, the brain and fatigue; revisiting the link.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Pathophysiology of hepatic encephalopathy: a new look at ammonia

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

A role for glutamate in growth and invasion of primary brain tumors

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Why do cancers have high aerobic glycolysis?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Acid treatment of melanoma cells selects for invasive phenotypes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Ammonia mediates communication between yeast colonies

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00109-011-0731-9

Evidence that glutamine, not sugar, is the major energy source for cultured HeLa cells

21 January 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21301794

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Ammonia derived from glutaminolysis is a diffusible regulator of autophagy

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21301794

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Targeting lactate-fueled respiration selectively kills hypoxic tumor cells in mice

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21301794

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1007/S00109-011-0731-9

1 reference

24 August 2017

1 reference