(Q38311364)

English

Ability of the glucocorticoid modulatory element to modify glucocorticoid receptor transactivation indicates parallel pathways for the expression of glucocorticoid modulatory element and glucocorticoid response element activities

scientific article published on April 2000

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Ability of the glucocorticoid modulatory element to modify glucocorticoid receptor transactivation indicates parallel pathways for the expression of glucocorticoid modulatory element and glucocorticoid response element activities (English)

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

author name string

Zeng H

1

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Plisov SY

2

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Simons SS Jr

3

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

publication date

1 April 2000

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Molecular and Cellular Endocrinology

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

162

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

1-2

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

221-234

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Synergistic action of glucocorticoid and estradiol responsive elements

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AIB1, a steroid receptor coactivator amplified in breast and ovarian cancer

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Specific cytoplasmic glucocorticoid hormone receptors in hepatoma tissue culture cells

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A cyclic AMP response element mediates repression of tyrosine aminotransferase gene transcription by the tissue-specific extinguisher locus Tse-1.

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A steroid response element can function in the absence of a distal promoter

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Positive and negative effects of DNA bending on activation of transcription from a distant site

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A negative tyrosine aminotransferase gene element that blocks glucocorticoid modulatory element-regulated modulation of glucocorticoid-induced gene expression

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Negative effect of the transcriptional activator GAL4

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Cyclic AMP stimulates somatostatin gene transcription by phosphorylation of CREB at serine 133

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Interaction at a distance between λrepressers disrupts gene activation

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Modulation of TAT gene induction by glucocorticoids involves a neutralizing sequence

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Cooperativity of glucocorticoid response elements located far upstream of the tyrosine aminotransferase gene

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A CBP integrator complex mediates transcriptional activation and AP-1 inhibition by nuclear receptors

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The progesterone receptor stimulates cell-free transcription by enhancing the formation of a stable preinitiation complex

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Antiglucocorticoid steroids have increased agonist activity in those hepatoma cell lines that are more sensitive to glucocorticoids

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Composite co-activator ARC mediates chromatin-directed transcriptional activation

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Promoter context- and response element-dependent specificity of the transcriptional activation and modulating functions of retinoic acid receptors

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Activation of the tyrosine aminotransferase gene is dependent on synergy between liver-specific and hormone-responsive elements

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Identifiers

10.1016/S0303-7207(99)00208-7

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

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