(Q38328012)

English

Oxidative stress transiently decreases the IKK complex (IKKalpha, beta, and gamma), an upstream component of NF-kappaB signaling, after transient focal cerebral ischemia in mice

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Oxidative stress transiently decreases the IKK complex (IKKalpha, beta, and gamma), an upstream component of NF-kappaB signaling, after transient focal cerebral ischemia in mice (English)

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

cerebral infarction

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

author name string

Yun S Song

1

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Yong-Sun Lee

2

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Pak H Chan

3

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

publication date

1 October 2005

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Journal of Cerebral Blood Flow & Metabolism

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

25

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

10

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

1301-1311

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Selective proteasomal dysfunction in the hippocampal CA1 region after transient forebrain ischemia

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Induction of neuroprotective kappa B-dependent transcription by secreted forms of the Alzheimer's beta-amyloid precursor.

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A catalytic antioxidant (AEOL 10150) attenuates expression of inflammatory genes in stroke

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Nuclear factor-kappa B activation during cerebral reperfusion: effect of attenuation with N-acetylcysteine treatment

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Signal-induced Ubiquitination of IκB Kinase-β

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Role of oxidants in ischemic brain damage

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Signaling from toxic metals to NF-kappaB and beyond: not just a matter of reactive oxygen species

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Signal-induced site-specific phosphorylation targets I kappa B alpha to the ubiquitin-proteasome pathway

1 reference

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Site-specific phosphorylation of IkappaBalpha by a novel ubiquitination-dependent protein kinase activity

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Cerebral ischemia: gene activation, neuronal injury, and the protective role of antioxidants

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Global cerebral ischemia activates nuclear factor-kappa B prior to evidence of DNA fragmentation

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Positive and negative regulation of IkappaB kinase activity through IKKbeta subunit phosphorylation

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NF-kappaB transcription factors: critical regulators of hematopoiesis and neuronal survival

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Transgenic mice with increased Cu/Zn-superoxide dismutase activity: animal model of dosage effects in Down syndrome

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Early decrease of apurinic/apyrimidinic endonuclease expression after transient focal cerebral ischemia in mice

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Activation of nuclear factor-kappaB in the rat brain after transient focal ischemia

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The ubiquitin-proteasome proteolytic pathway: destruction for the sake of construction

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Caspase-mediated cleavage converts the tumor necrosis factor (TNF) receptor-associated factor (TRAF)-1 from a selective modulator of TNF receptor signaling to a general inhibitor of NF-kappaB activation

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SOD1 down-regulates NF-kappaB and c-Myc expression in mice after transient focal cerebral ischemia

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Ubiquitin stress response in postischemic hippocampal neurons under nontolerant and tolerant conditions

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Activation of NF-kappa B by reactive oxygen intermediates in the nervous system

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Inhibition of NF-kappa B activation by arsenite through reaction with a critical cysteine in the activation loop of Ikappa B kinase

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The beginning of the end: IkappaB kinase (IKK) and NF-kappaB activation

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NF-kappaB at the crossroads of life and death

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Attenuation of focal cerebral ischemic injury in transgenic mice overexpressing CuZn superoxide dismutase

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NF-kappaB regulation in the immune system

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Evidence of phosphorylation of Akt and neuronal survival after transient focal cerebral ischemia in mice

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The ubiquitin-proteasome pathway is required for processing the NF-kappa B1 precursor protein and the activation of NF-kappa B

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Anti-inflammatory cyclopentenone prostaglandins are direct inhibitors of IkappaB kinase

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Alteration of Transcription Factor Binding Activities in the Ischemic Rat-Brain

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Signal transduction. Docking IkappaB kinases.

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NF-kappaB is activated and promotes cell death in focal cerebral ischemia

1 reference

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Oxidative stress interference with the nuclear factor-kappa B activation pathways

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Blocking caspase-3-mediated proteolysis of IKKbeta suppresses TNF-alpha-induced apoptosis

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I kappa B proteins: structure, function and regulation

1 reference

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Intracellular localization of proteasomes

1 reference

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Human copper-zinc superoxide dismutase transgenic mice are highly resistant to reperfusion injury after focal cerebral ischemia

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Neuroprotective role for the p50 subunit of NF-kappaB in an experimental model of Huntington's disease

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Reactive oxygen species activate p90 ribosomal S6 kinase via Fyn and Ras

1 reference

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Loss of NF-kappaB activity during cerebral ischemia and TNF cytotoxicity.

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NEMO/IKK gamma: linking NF-kappa B to human disease.

1 reference

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Decreased nuclear factor-kappaB DNA binding activity following permanent focal cerebral ischaemia in the rat

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N-acetylcysteine suppresses TNF-induced NF-kappaB activation through inhibition of IkappaB kinases

1 reference

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Overexpression of copper/zinc superoxide dismutase in transgenic mice protects against neuronal cell death after transient focal ischemia by blocking activation of the Bad cell death signaling pathway.

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Transcription factor nuclear factor-kappa B is activated in neurons after focal cerebral ischemia

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based on heuristic

inferred from DOI database lookup

Identifiers

10.1038/SJ.JCBFM.9600123

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

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