(Q38351071)

English

Hypoxia induces apoptosis in SV40-immortalized rat proximal tubular cells through the mitochondrial pathways, devoid of HIF1-mediated upregulation of Bax.

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Europe PubMed Central

PubMed publication ID

2 September 2017

Hypoxia induces apoptosis in SV40-immortalized rat proximal tubular cells through the mitochondrial pathways, devoid of HIF1-mediated upregulation of Bax. (English)

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Europe PubMed Central

PubMed publication ID

2 September 2017

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Tetsuhiro Tanaka

1

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Europe PubMed Central

PubMed publication ID

2 September 2017

Norio Hanafusa

2

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Europe PubMed Central

PubMed publication ID

2 September 2017

Julie R Ingelfinger

3

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Europe PubMed Central

PubMed publication ID

2 September 2017

Takamoto Ohse

4

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Europe PubMed Central

PubMed publication ID

2 September 2017

Toshiro Fujita

5

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Europe PubMed Central

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2 September 2017

Masaomi Nangaku

6

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2 September 2017

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publication date

1 September 2003

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2 September 2017

number of pages

10

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Biochemical and Biophysical Research Communications

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Europe PubMed Central

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2 September 2017

309

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Europe PubMed Central

PubMed publication ID

2 September 2017

1

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Europe PubMed Central

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2 September 2017

222-231

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Europe PubMed Central

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2 September 2017

Chronic renal failure: pathophysiology

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The pathogenesis of chronic renal failure

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Apoptosis in tubulointerstitial renal disease

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Apoptosis and necrosis during ischaemia in renal tubular cells (LLC-PK1 and MDCK).

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Disparate mechanisms for hypoxic cell injury in different nephron segments. Studies in the isolated perfused rat kidney

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Involvement of the bcl-2 gene in human follicular lymphoma

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Movement of Bax from the cytosol to mitochondria during apoptosis

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Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

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Bcl-2 family: life-or-death switch

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Bcl-2 family proteins

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The Bcl-2 protein family: arbiters of cell survival

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Hypoxia induces accumulation of p53 protein, but activation of a G1-phase checkpoint by low-oxygen conditions is independent of p53 status.

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Bax suppresses tumorigenesis and stimulates apoptosis in vivo

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bax-deficiency promotes drug resistance and oncogenic transformation by attenuating p53-dependent apoptosis

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A novel form of Epstein-Barr virus latency in normal B cells in vivo

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Is HIF-1alpha a pro- or an anti-apoptotic protein?

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p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes

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Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.

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Establishment of rat fetal liver lines and characterization of their metabolic and hormonal properties: use of temperature-sensitive SV40 virus

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Sphingosine 1-phosphate stimulates rat mesangial cell proliferation from outside the cells

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Efficient induction of apoptosis in cultured rat glomerular mesangial cells by dimeric monoclonal IgA anti-Thy-1 antibodies

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Hypoxia-inducible factor-1alpha mediates hypoxia-induced delayed neuronal death that involves p53.

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The binding properties and biological activities of Bcl-2 and Bax in cells exposed to apoptotic stimuli

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Transcriptional regulation of the rat vascular endothelial growth factor gene by hypoxia

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Activation of a 15-kDa endonuclease in hypoxia/reoxygenation injury without morphologic features of apoptosis

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Anoxia-induced apoptosis occurs through a mitochondria-dependent pathway in lung epithelial cells

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Prior heat stress inhibits apoptosis in adenosine triphosphate-depleted renal tubular cells

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Role of hypoxia-induced Bax translocation and cytochrome c release in reoxygenation injury

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Prevention of hypoxia-induced cell death by Bcl-2 and Bcl-xL.

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Programmed cell death and Bcl-2 protection in very low oxygen.

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Effects of microinjection of synthetic Bcl-2 domain peptides on apoptosis of renal tubular epithelial cells.

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Differential interaction of temperature-sensitive simian virus 40 T antigens with tumor suppressors pRb and p53

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Identifiers

10.1016/S0006-291X(03)01557-2

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

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