(Q38885002)

7 September 2017

1 reference

title

Immune responses and immunopathology in acute and chronic viral hepatitis (English)

1 reference

7 September 2017

1

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27374637%20AND%20SRC:MED&resulttype=core&format=json

10 June 2020

author name string

Pil Soo Sung

series ordinal

2

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27374637%20AND%20SRC:MED&resulttype=core&format=json

10 June 2020

Su-Hyung Park

series ordinal

3

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27374637%20AND%20SRC:MED&resulttype=core&format=json

10 June 2020

publication date

4 July 2016

1 reference

Nature Reviews Immunology

7 September 2017

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27374637%20AND%20SRC:MED&resulttype=core&format=json

10 June 2020

volume

16

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27374637%20AND%20SRC:MED&resulttype=core&format=json

10 June 2020

issue

8

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27374637%20AND%20SRC:MED&resulttype=core&format=json

10 June 2020

page(s)

509-523

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27374637%20AND%20SRC:MED&resulttype=core&format=json

https://scigraph.springernature.com/pub.10.1038/nri.2016.69

10 June 2020

exact match

0 references

cites work

Immune responses to HCV and other hepatitis viruses

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Immunobiology and pathogenesis of viral hepatitis

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Immunological aspects of antiviral therapy of chronic hepatitis B virus and hepatitis C virus infections

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Reassessing immune control of hepatitis A virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Update on hepatitis E virology: Implications for clinical practice

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Interferon-stimulated genes: a complex web of host defenses

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Regulation of hepatic innate immunity by hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Nonlinear partial differential equations and applications: Genomic analysis of the host response to hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Acute hepatitis A virus infection is associated with a limited type I interferon response and persistence of intrahepatic viral RNA.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Genomic analysis of the host response to hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Stealth and cunning: hepatitis B and hepatitis C viruses

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Control of temporal activation of hepatitis C virus-induced interferon response by domain 2 of nonstructural protein 5A.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis C virus reveals a novel early control in acute immune response

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Activation of chemokine and inflammatory cytokine response in hepatitis C virus-infected hepatocytes depends on Toll-like receptor 3 sensing of hepatitis C virus double-stranded RNA intermediates

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A diverse range of gene products are effectors of the type I interferon antiviral response

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Mitochondrial-associated endoplasmic reticulum membranes (MAM) form innate immune synapses and are targeted by hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Activation of Type I and III Interferon Response by Mitochondrial and Peroxisomal MAVS and Inhibition by Hepatitis C Virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Cleavage of mitochondrial antiviral signaling protein in the liver of patients with chronic hepatitis C correlates with a reduced activation of the endogenous interferon system

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Immune evasion by hepatitis C virus NS3/4A protease-mediated cleavage of the Toll-like receptor 3 adaptor protein TRIF

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Simultaneous detection of hepatitis C virus and interferon stimulated gene expression in infected human liver

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

HepG2 cells mount an effective antiviral interferon-lambda based innate immune response to hepatitis C virus infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

IL-29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Interferon lambda alleles predict innate antiviral immune responses and hepatitis C virus permissiveness

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Short-range exosomal transfer of viral RNA from infected cells to plasmacytoid dendritic cells triggers innate immunity

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Roles of unphosphorylated ISGF3 in HCV infection and interferon responsiveness.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

IFNβ-dependent increases in STAT1, STAT2, and IRF9 mediate resistance to viruses and DNA damage

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Reduced IFNλ4 activity is associated with improved HCV clearance and reduced expression of interferon-stimulated genes

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatic gene expression discriminates responders and nonresponders in treatment of chronic hepatitis C viral infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatic gene expression during treatment with peginterferon and ribavirin: Identifying molecular pathways for treatment response

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Interferon-induced gene expression is a stronger predictor of treatment response than IL28B genotype in patients with hepatitis C.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Silencing of USP18 potentiates the antiviral activity of interferon against hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Flying under the radar: the immunobiology of hepatitis C.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Human intracellular ISG15 prevents interferon-α/β over-amplification and auto-inflammation

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Comparison of functional variants in IFNL4 and IFNL3 for association with HCV clearance

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Induction and suppression of innate antiviral responses by picornaviruses

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Disruption of innate immunity due to mitochondrial targeting of a picornaviral protease precursor

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Disruption of TLR3 signaling due to cleavage of TRIF by the hepatitis A virus protease-polymerase processing intermediate, 3CD.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis A virus 3C protease cleaves NEMO to impair induction of beta interferon

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Human pDCs preferentially sense enveloped hepatitis A virions

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A pathogenic picornavirus acquires an envelope by hijacking cellular membranes

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The ABCs of viral hepatitis that define biomarker signatures of acute viral hepatitis

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The RNA sensor RIG-I dually functions as an innate sensor and direct antiviral factor for hepatitis B virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Melanoma differentiation-associated gene 5 senses hepatitis B virus and activates innate immune signaling to suppress virus replication

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis B virus polymerase blocks pattern recognition receptor signaling via interaction with DDX3: implications for immune evasion

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis B virus polymerase disrupts K63-linked ubiquitination of STING to block innate cytosolic DNA-sensing pathways

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis B virus polymerase impairs interferon-α-induced STA T activation through inhibition of importin-α5 and protein kinase C-δ.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The hepatitis B virus X protein disrupts innate immunity by downregulating mitochondrial antiviral signaling protein

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis B virus limits response of human hepatocytes to interferon-α in chimeric mice.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

HLA and NK cell inhibitory receptor genes in resolving hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Increased natural killer cell cytotoxicity and NKp30 expression protects against hepatitis C virus infection in high-risk individuals and inhibits replication in vitro

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Protection against hepatitis C infection via NK cells in highly-exposed uninfected injecting drug users

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Innate immune responses in hepatitis C virus-exposed healthcare workers who do not develop acute infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

CD27 engagement by a soluble CD70 protein enhances non-cytolytic antiviral activity of CD56bright natural killer cells by IFN-γ secretion

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

An effective interferon-gamma-mediated inhibition of hepatitis C virus replication by natural killer cells is associated with spontaneous clearance of acute hepatitis C in human immunodeficiency virus-positive patients.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Natural killer p46High expression defines a natural killer cell subset that is potentially involved in control of hepatitis C virus replication and modulation of liver fibrosis.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Race- and gender-related variation in natural killer p46 expression associated with differential anti-hepatitis C virus immunity

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Activation of natural killer cells during acute infection with hepatitis C virus.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Increased degranulation of natural killer cells during acute HCV correlates with the magnitude of virus-specific T cell responses

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Altered interferon-alpha-signaling in natural killer cells from patients with chronic hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Early changes in interferon signaling define natural killer cell response and refractoriness to interferon-based therapy of hepatitis C patients.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Temporal analysis of early immune responses in patients with acute hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Natural killer cell functional dichotomy in chronic hepatitis B and chronic hepatitis C virus infections

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Blockade of immunosuppressive cytokines restores NK cell antiviral function in chronic hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

TGF-β1 down-regulation of NKG2D/DAP10 and 2B4/SAP expression on human NK cells contributes to HBV persistence

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Interferon-γ and Tumor Necrosis Factor-α Produced by T Cells Reduce the HBV Persistence Form, cccDNA, Without Cytolysis.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Analysis of CD8+ T-cell-mediated inhibition of hepatitis C virus replication using a novel immunological model

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Natural Killer Cells in Viral Hepatitis

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis A virus: from discovery to vaccines

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Cellular immune responses persist and humoral responses decrease two decades after recovery from a single-source outbreak of hepatitis C

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Selection pressure from neutralizing antibodies drives sequence evolution during acute infection with hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis C virus cell-cell transmission in hepatoma cells in the presence of neutralizing antibodies

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Neutralizing antibody response during acute and chronic hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Discordant role of CD4 T-cell response relative to neutralizing antibody and CD8 T-cell responses in acute hepatitis C.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The Humoral Immune Response to HCV: Understanding is Key to Vaccine Development.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Rapid and sustained autologous neutralizing response leading to early spontaneous recovery after HCV infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Clearance of hepatitis C infection is associated with the early appearance of broad neutralizing antibody responses

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Spontaneous clearance of chronic hepatitis C virus infection is associated with appearance of neutralizing antibodies and reversal of T-cell exhaustion

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis C Virus E2 Envelope Glycoprotein Core Structure

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Broadly neutralizing antibodies protect against hepatitis C virus quasispecies challenge

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Broadly neutralizing antibodies abrogate established hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Memory CD8+ T cells are required for protection from persistent hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

HCV Persistence and Immune Evasion in the Absence of Memory T Cell Help

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

CD8(+) T cells mediate viral clearance and disease pathogenesis during acute hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Viral and immunological determinants of hepatitis C virus clearance, persistence, and disease

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Delayed induction, not impaired recruitment, of specific CD8⁺ T cells causes the late onset of acute hepatitis C

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Virus-induced type I IFN stimulates generation of immunoproteasomes at the site of infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Analysis of successful immune responses in persons infected with hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Determinants of viral clearance and persistence during acute hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The frequency of CD127(+) hepatitis C virus (HCV)-specific T cells but not the expression of exhaustion markers predicts the outcome of acute HCV infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Mutational escape from CD8+ T cell immunity: HCV evolution, from chimpanzees to man

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Preferential loss of IL-2-secreting CD4+ T helper cells in chronic HCV infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Broadly directed virus-specific CD4+ T cell responses are primed during acute hepatitis C infection, but rapidly disappear from human blood with viral persistence

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Virus-Specific CD4+ T Cells Have Functional and Phenotypic Characteristics of Follicular T-Helper Cells in Patients With Acute and Chronic HCV Infections

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Elevated CD4+/CD25+ T cell frequency and function during acute hepatitis C presage chronic evolution.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Functional suppression by FoxP3+CD4+CD25(high) regulatory T cells during acute hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

FOXP3 expression in hepatitis C virus-specific CD4+ T cells during acute hepatitis C.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Galectin-9 and IL-21 mediate cross-regulation between Th17 and Treg cells during acute hepatitis C.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Pathogenesis of hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The cytotoxic T lymphocyte response to multiple hepatitis B virus polymerase epitopes during and after acute viral hepatitis

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Adaptation of the hepatitis B virus core protein to CD8(+) T-cell selection pressure.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Immunology of hepatitis B virus and hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The size of the viral inoculum contributes to the outcome of hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Characterization of CD8+ T-cell response in acute and resolved hepatitis A virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Dominance of the CD4(+) T helper cell response during acute resolving hepatitis A virus infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Liver injury in acute hepatitis A is associated with decreased frequency of regulatory T cells caused by Fas-mediated apoptosis

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Binding of hepatitis A virus to its cellular receptor 1 inhibits T-regulatory cell functions in humans

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

T-cell dysfunction and inhibitory receptors in hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Synergistic Reversal of Intrahepatic HCV-Specific CD8 T Cell Exhaustion by Combined PD-1/CTLA-4 Blockade

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Upregulation of PD-1 Expression on Circulating and Intrahepatic Hepatitis C Virus-Specific CD8+ T Cells Associated with Reversible Immune Dysfunction

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Negative immune regulator Tim-3 is overexpressed on T cells in hepatitis C virus infection and its blockade rescues dysfunctional CD4+ and CD8+ T cells

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Dual function of the NK cell receptor 2B4 (CD244) in the regulation of HCV-specific CD8+ T cells

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Coexpression of PD-1, 2B4, CD160 and KLRG1 on exhausted HCV-specific CD8+ T cells is linked to antigen recognition and T cell differentiation

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

CD39 Expression Identifies Terminally Exhausted CD8+ T Cells

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Tim-3 expression on PD-1+ HCV-specific human CTLs is associated with viral persistence, and its blockade restores hepatocyte-directed in vitro cytotoxicity

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Liver environment and HCV replication affect human T-cell phenotype and expression of inhibitory receptors

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Liver-Infiltrating Lymphocytes in Chronic Human Hepatitis C Virus Infection Display an Exhausted Phenotype with High Levels of PD-1 and Low Levels of CD127 Expression

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A heterogeneous hierarchy of co-regulatory receptors regulates exhaustion of HCV-specific CD8 T cells in patients with chronic hepatitis C.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Immunotherapy of chronic hepatitis C virus infection with antibodies against programmed cell death-1 (PD-1).

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A randomized, double-blind, placebo-controlled assessment of BMS-936558, a fully human monoclonal antibody to programmed death-1 (PD-1), in patients with chronic hepatitis C virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Restoration of HBV-specific CD8+ T cell function by PD-1 blockade in inactive carrier patients is linked to T cell differentiation

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Antiviral Intrahepatic T-Cell Responses Can Be Restored by Blocking Programmed Death-1 Pathway in Chronic Hepatitis B

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Role of the coinhibitory receptor cytotoxic T lymphocyte antigen-4 on apoptosis-Prone CD8 T cells in persistent hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Upregulation of the Tim-3/galectin-9 pathway of T cell exhaustion in chronic hepatitis B virus infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The immunoregulatory role of CD244 in chronic hepatitis B infection and its inhibitory potential on virus-specific CD8+ T-cell function

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Characterization of hepatitis B virus (HBV)-specific T-cell dysfunction in chronic HBV infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Combined blockade of programmed death-1 and activation of CD137 increase responses of human liver T cells against HBV, but not HCV.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The third signal cytokine IL-12 rescues the anti-viral function of exhausted HBV-specific CD8 T cells

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Dysfunctional CD8+ T cells in hepatitis B and C are characterized by a lack of antigen-specific T-bet induction

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Bim-mediated deletion of antigen-specific CD8 T cells in patients unable to control HBV infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Up-regulation of a death receptor renders antiviral T cells susceptible to NK cell-mediated deletion.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

An immunomodulatory role for CD4(+)CD25(+) regulatory T lymphocytes in hepatitis C virus infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Abundant numbers of regulatory T cells localize to the liver of chronic hepatitis C infected patients and limit the extent of fibrosis

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Regulatory T cells contribute to the impaired immune response in patients with chronic hepatitis B virus infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis B Virus--Specific and Global T-Cell Dysfunction in Chronic Hepatitis B.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

T cells with a CD4+CD25+ regulatory phenotype suppress in vitro proliferation of virus-specific CD8+ T cells during chronic hepatitis C virus infection.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

PD-L1 negatively regulates CD4+CD25+Foxp3+ Tregs by limiting STAT-5 phosphorylation in patients chronically infected with HCV.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Persistent LCMV infection is controlled by blockade of type I interferon signaling

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Blockade of chronic type I interferon signaling to control persistent LCMV infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Living in the liver: hepatic infections

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Pathological functions of interleukin-22 in chronic liver inflammation and fibrosis with hepatitis B virus infection by promoting T helper 17 cell recruitment

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A proinflammatory role for interleukin-22 in the immune response to hepatitis B virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Increased Th17 cells contribute to disease progression in patients with HBV-associated liver cirrhosis

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis C virus promotes T-helper (Th)17 responses through thymic stromal lymphopoietin production by infected hepatocytes

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Th17 cells are increased with severity of liver inflammation in patients with chronic hepatitis C.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

HBV pathogenesis in animal models: recent advances on the role of platelets

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The role of virus-specific CD8(+) cells in liver damage and viral control during persistent hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Metabolic regulation of hepatitis B immunopathology by myeloid-derived suppressor cells.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Myeloid suppressor cells induced by hepatitis C virus suppress T-cell responses through the production of reactive oxygen species

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis C Virus-Induced Myeloid-Derived Suppressor Cells Suppress NK Cell IFN-γ Production by Altering Cellular Metabolism via Arginase-1.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

T-cell immunity and hepatitis C virus reinfection after cure of chronic hepatitis C with an interferon-free antiviral regimen in a chimpanzee

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Restoration of HCV-specific CD8+ T cell function by interferon-free therapy

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Restored Function of HBV-Specific T Cells After Long-term Effective Therapy With Nucleos(t)ide Analogues

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Natural killer cell phenotype modulation and natural killer/T-cell interplay in nucleos(t)ide analogue-treated hepatitis e antigen-negative patients with chronic hepatitis B.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Hepatitis B virus infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

The impact of differential antiviral immunity in children and adults

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

IL-21 is pivotal in determining age-dependent effectiveness of immune responses in a mouse model of human hepatitis B.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Preserved T-cell function in children and young adults with immune-tolerant chronic hepatitis B.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

IL28B is associated with response to chronic hepatitis C interferon-alpha and ribavirin therapy

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Genome-wide association of IL28B with response to pegylated interferon-alpha and ribavirin therapy for chronic hepatitis C

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Genetic variation in IL28B and spontaneous clearance of hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Interferon lambda 4 signals via the IFNλ receptor to regulate antiviral activity against HCV and coronaviruses

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Transcriptome analysis reveals a classical interferon signature induced by IFNλ4 in human primary cells.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Current progress in development of hepatitis C virus vaccines.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Cross-genotype immunity to hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A T-cell HCV vaccine eliciting effective immunity against heterologous virus challenge in chimpanzees

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Vaccine-induced early control of hepatitis C virus infection in chimpanzees fails to impact on hepatic PD-1 and chronicity

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Successful vaccination induces multifunctional memory T-cell precursors associated with early control of hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

A human vaccine strategy based on chimpanzee adenoviral and MVA vectors that primes, boosts, and sustains functional HCV-specific T cell memory

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Spontaneous control of primary hepatitis C virus infection and immunity against persistent reinfection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Vaccine-induced cross-genotype reactive neutralizing antibodies against hepatitis C virus

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Viral Clearance Without Destruction of Infected Cells During Acute HBV Infection

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Kinetics of CD4+ and CD8+ Memory T-Cell Responses during Hepatitis C Virus Rechallenge of Previously Recovered Chimpanzees

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FNRI.2016.69

Distinct KIR/HLA compound genotypes affect the kinetics of human antiviral natural killer cell responses

31 March 2020

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/27374637

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

CD8(+) T-Cell Responses in Acute Hepatitis C Virus Infection.

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/27374637