(Q39043749)

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Evaluation of tubulin polymerization and chronic inhibition of proteasome as citotoxicity mechanisms in bortezomib-induced peripheral neuropathy.

scientific article published on 12 December 2013

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Europe PubMed Central

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3 March 2020

Evaluation of tubulin polymerization and chronic inhibition of proteasome as citotoxicity mechanisms in bortezomib-induced peripheral neuropathy (English)

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3 March 2020

Federica La Russa

10

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3 March 2020

David L Bennett

14

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3 March 2020

Guido Cavaletti

15

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3 March 2020

Valentina Alda Carozzi

3

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Valentina A Carozzi

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3 March 2020

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Cristina Meregalli

1

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3 March 2020

Alessia Chiorazzi

2

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3 March 2020

Annalisa Canta

4

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3 March 2020

Barbara Sala

5

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3 March 2020

Matteo Colombo

6

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3 March 2020

Norberto Oggioni

7

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3 March 2020

Cecilia Ceresa

8

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3 March 2020

Dana Foudah

9

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3 March 2020

Mariarosaria Miloso

11

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3 March 2020

Gabriella Nicolini

12

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3 March 2020

Paola Marmiroli

13

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3 March 2020

publication date

12 December 2013

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3 March 2020

number of pages

10

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3 March 2020

13

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3 March 2020

4

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3 March 2020

612-621

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3 March 2020

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Bortezomib alters microtubule polymerization and axonal transport in rat dorsal root ganglion neurons

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Neurophysiological, histological and immunohistochemical characterization of bortezomib-induced neuropathy in mice

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21 January 2018

Microtubule-disrupting chemotherapeutics result in enhanced proteasome-mediated degradation and disappearance of tubulin in neural cells

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Chemotherapy-Induced Peripheral Neurotoxicity assessment: a critical revision of the currently available tools

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21 January 2018

Pathological adaptive responses of Schwann cells to endoplasmic reticulum stress in bortezomib‐induced peripheral neuropathy

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21 January 2018

Chemotherapy-induced neuropathy

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21 January 2018

Proteasome inhibitors increase tubulin polymerization and stabilization in tissue culture cells: a possible mechanism contributing to peripheral neuropathy and cellular toxicity following proteasome inhibition.

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21 January 2018

Bortezomib-induced peripheral neurotoxicity: a neurophysiological and pathological study in the rat.

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Bortezomib induces the formation of nuclear poly(A) RNA granules enriched in Sam68 and PABPN1 in sensory ganglia neurons.

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Proteasome inhibition induces DNA damage and reorganizes nuclear architecture and protein synthesis machinery in sensory ganglion neurons

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