(Q39177064)

9 September 2017

RIP3, a kinase promoting necroptotic cell death, mediates adverse remodelling after myocardial infarction (English)

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9 September 2017

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myocardial infarction

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16

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Andreas Linkermann

12

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Renate Lüllmann-Rauch

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Renate Lüllmann-Rauch

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Mathias Heikenwalder

18

object named as

Mathias Heikenwalder

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Stefan Rose-John

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Stefan Rose-John

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author name string

Mark Luedde

1

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9 September 2017

Matthias Lutz

2

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9 September 2017

Natalie Carter

3

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9 September 2017

Justyna Sosna

4

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9 September 2017

Christoph Jacoby

5

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9 September 2017

Mihael Vucur

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9 September 2017

Jérémie Gautheron

7

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9 September 2017

Christoph Roderburg

8

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9 September 2017

Nadine Borg

9

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9 September 2017

Florian Reisinger

10

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9 September 2017

Hans-Joerg Hippe

11

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9 September 2017

Monika J Wolf

13

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9 September 2017

Renate Lüllmann-Rauch

15

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9 September 2017

Ulrich Flögel

17

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9 September 2017

Tom Luedde

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9 September 2017

Norbert Frey

20

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9 September 2017

11 June 2014

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9 September 2017

Cardiovascular Research

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9 September 2017

103

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9 September 2017

2

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9 September 2017

206-216

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The cardiac Fas (APO-1/CD95) Receptor/Fas ligand system : relation to diastolic wall stress in volume-overload hypertrophy in vivo and activation of the transcription factor AP-1 in cardiac myocytes

9 September 2017

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Myocyte apoptosis during acute myocardial infarction in the mouse localizes to hypoxic regions but occurs independently of p53

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Programmed necrosis, not apoptosis, in the heart

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

RIP kinases at the crossroads of cell death and survival

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Necroptosis as an alternative form of programmed cell death

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

FADD prevents RIP3-mediated epithelial cell necrosis and chronic intestinal inflammation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Inhibition of RIP1-dependent necrosis prevents adverse cardiac remodeling after myocardial ischemia-reperfusion in vivo.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Necrostatin: a potentially novel cardioprotective agent?

21 January 2018

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https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Regulation of cardiac cAMP synthesis and contractility by nucleoside diphosphate kinase B/G protein beta gamma dimer complexes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Kinase RIP3 is dispensable for normal NF-kappa Bs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and Toll-like receptors 2 and 4.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

In vivo monitoring of inflammation after cardiac and cerebral ischemia by fluorine magnetic resonance imaging

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Mitochondria: master regulators of danger signalling

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

RIP3 mediates the embryonic lethality of caspase-8-deficient mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

TNF-induced necroptosis in L929 cells is tightly regulated by multiple TNFR1 complex I and II members

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Programmed necrosis: backup to and competitor with apoptosis in the immune system

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Post-infarct remodelling: contribution of wound healing and inflammation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Cell death in the pathogenesis of heart disease: mechanisms and significance

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Role of oxidative stress in cardiac hypertrophy and remodeling

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Oxidative stress and left ventricular remodelling after myocardial infarction

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

RIP3 finds partners in crime

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Necroptosis, necrosis and secondary necrosis converge on similar cellular disintegration features.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Endosomal Nox2 facilitates redox-dependent induction of NF-kappaB by TNF-alpha

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Up-regulation of PPARgamma in myocardial infarction

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Widespread mitochondrial depletion via mitophagy does not compromise necroptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

Excessive Tumor Necrosis Factor Activation After Infarction Contributes to Susceptibility of Myocardial Rupture and Left Ventricular Dysfunction

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVU146

21 January 2018

Identifiers

DOI

10.1093/CVR/CVU146

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9 September 2017

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