(Q39222665)

English

Prolonged classical NF-kappaB activation prevents autophagy upon E. coli stimulation in vitro: a potential resolving mechanism of inflammation

scientific article published on January 2008

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scholarly article

1 reference

Europe PubMed Central

10 September 2017

Prolonged classical NF-kappaB activation prevents autophagy upon E. coli stimulation in vitro: a potential resolving mechanism of inflammation (English)

1 reference

Europe PubMed Central

10 September 2017

0 references

Escherichia coli

0 references

0 references

0 references

author name string

Silke Schlottmann

1

1 reference

Europe PubMed Central

10 September 2017

Franziska Buback

2

1 reference

Europe PubMed Central

10 September 2017

Bettina Stahl

3

1 reference

Europe PubMed Central

10 September 2017

Rainer Meierhenrich

4

1 reference

Europe PubMed Central

10 September 2017

Paul Walter

5

1 reference

Europe PubMed Central

10 September 2017

Michael Georgieff

6

1 reference

Europe PubMed Central

10 September 2017

Uwe Senftleben

7

1 reference

Europe PubMed Central

10 September 2017

publication date

1 January 2008

1 reference

Europe PubMed Central

10 September 2017

Mediators of Inflammation

1 reference

Europe PubMed Central

10 September 2017

2008

1 reference

Europe PubMed Central

10 September 2017

725854

1 reference

Europe PubMed Central

10 September 2017

copyright license

Creative Commons Attribution 3.0 Unported

1 January 2008

1 reference

April 2022 Public Data File from Crossref

copyright status

1 reference

April 2022 Public Data File from Crossref

Escherichia coli prevents phagocytosis-induced death of macrophages via classical NF-kappaB signaling, a link to T-cell activation

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice

1 reference

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1 reference

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Src and Syk kinases: key regulators of phagocytic cell activation.

1 reference

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1 reference

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The serine/threonine kinase Pim-1

1 reference

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1 reference

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Autophagy is a defense mechanism inhibiting BCG and Mycobacterium tuberculosis survival in infected macrophages

1 reference

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1 reference

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Autophagy in health and disease: a double-edged sword

1 reference

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Function and regulation of tumor necrosis factor receptor type 2.

1 reference

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Modulation of phagocyte apoptosis by bacterial pathogens.

1 reference

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Regulation of TLR signaling and inflammation by SOCS family proteins

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A Salmonella protein causes macrophage cell death by inducing autophagy

1 reference

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Caspase-activation pathways in apoptosis and immunity

1 reference

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10 September 2017

Necrosis: a specific form of programmed cell death?

1 reference

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IAP proteins: blocking the road to death's door

1 reference

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10 September 2017

NF-kappaB at the crossroads of life and death

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2430012

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Activation by IKKalpha of a second, evolutionary conserved, NF-kappa B signaling pathway

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2430012

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MCP-1 and RANTES are mediators of acute and chronic inflammation

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Beclin 1 contains a leucine-rich nuclear export signal that is required for its autophagy and tumor suppressor function

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Tumor necrosis factor receptor-associated factor (TRAF) 2 and its role in TNF signaling.

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LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

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The role of NF-AT transcription factors in T cell activation and differentiation

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A20 and A20-binding proteins as cellular inhibitors of nuclear factor-kappa B-dependent gene expression and apoptosis

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Control of myeloid differentiation and survival by Stats

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Caspase-3: A vulnerability factor and final effector in apoptotic death of dopaminergic neurons in Parkinson's disease.

1 reference

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Granulocyte apoptosis and its role in the resolution and control of lung inflammation

1 reference

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ATF3 and stress responses.

1 reference

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Biological role of the CCAAT/enhancer-binding protein family of transcription factors

1 reference

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Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

1 reference

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Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B

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Developmental cell death: morphological diversity and multiple mechanisms

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Transcriptional cross talk between NF-kappaB and p53.

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Cytomegalovirus primary envelopment occurs at large infoldings of the inner nuclear membrane.

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NF-kappaB activation represses tumor necrosis factor-alpha-induced autophagy

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Possible new role for NF-kappaB in the resolution of inflammation.

1 reference

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Immune-mediated phagocytosis and killing of Streptococcus pneumoniae are associated with direct and bystander macrophage apoptosis.

1 reference

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1 reference

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Mitochondria are selectively eliminated from eukaryotic cells after blockade of caspases during apoptosis

1 reference

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Salmonella induces macrophage death by caspase-1-dependent necrosis.

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1 reference

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Bcl-2 down-regulation causes autophagy in a caspase-independent manner in human leukemic HL60 cells

1 reference

https://api.crossref.org/works/10.1155%2F2008%2F725854

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IKKα limits macrophage NF-κB activation and contributes to the resolution of inflammation

1 reference

https://api.crossref.org/works/10.1155%2F2008%2F725854

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Bcl-3 and NFkappaB p50-p50 homodimers act as transcriptional repressors in tolerant CD4+ T cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/18566685

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Autophagy in human tumors: cell survival or death?

1 reference

https://pubmed.ncbi.nlm.nih.gov/18566685

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

NF-kappaB functions as both a proapoptotic and antiapoptotic regulatory factor within a single cell type

1 reference

https://pubmed.ncbi.nlm.nih.gov/18566685

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1155/2008/725854

1 reference

Europe PubMed Central

10 September 2017

1 reference

Europe PubMed Central

10 September 2017

1 reference

Europe PubMed Central

10 September 2017

ResearchGate publication ID

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