(Q39349033)

11 September 2017

title

STAT3 mutations correlated with hyper-IgE syndrome lead to blockage of IL-6/STAT3 signalling pathway (English)

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11 September 2017

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Jianxin He

1

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11 September 2017

Jie Shi

2

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11 September 2017

Ximing Xu

3

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11 September 2017

Wenhua Zhang

4

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11 September 2017

Yuxin Wang

5

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11 September 2017

Xing Chen

6

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11 September 2017

Yuping Du

7

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11 September 2017

Ning Zhu

8

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11 September 2017

Jing Zhang

9

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11 September 2017

Qin Wang

10

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11 September 2017

Jinbo Yang

11

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11 September 2017

publication date

1 June 2012

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11 September 2017

published in

Journal of Biosciences

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11 September 2017

volume

37

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11 September 2017

issue

2

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11 September 2017

page(s)

243-257

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https://scigraph.springernature.com/pub.10.1007/s12038-012-9202-x

11 September 2017

exact match

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cites work

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Mutational switch of an IL-6 response to an interferon-gamma-like response

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STATs and gene regulation

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Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

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Serine phosphorylation of STATs

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Clinical manifestations of hyper IgE syndromes

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Causes of death in hyper-IgE syndrome

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Genetic linkage of hyper-IgE syndrome to chromosome 4.

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Predicting changes in the stability of proteins and protein complexes: a study of more than 1000 mutations

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Divergent roles of IL-23 and IL-12 in host defense against Klebsiella pneumoniae

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STAT3 mutations in the hyper-IgE syndrome

21 January 2018

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Novel and recurrent STAT3 mutations in hyper-IgE syndrome patients from different ethnic groups.

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Analysis of Stat3 (signal transducer and activator of transcription 3) dimerization by fluorescence resonance energy transfer in living cells

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STAT3 nuclear import is independent of tyrosine phosphorylation and mediated by importin-alpha3.

21 January 2018

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Deficiency of Th17 cells in hyper IgE syndrome due to mutations in STAT3

21 January 2018

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A Novel Sequence in the Coiled-coil Domain of Stat3 Essential for Its Nuclear Translocation

21 January 2018

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Impaired T(H)17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

21 January 2018

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21 January 2018

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Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome

21 January 2018

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IL-6-induced homodimerization of gp130 and associated activation of a tyrosine kinase

21 January 2018

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Accounting for human polymorphisms predicted to affect protein function

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A novel mutation in the signal transducer and activator of transcription 3 (STAT3) gene, in hyper-IgE syndrome

21 January 2018

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Ups and downs: the STAT1:STAT3 seesaw of Interferon and gp130 receptor signalling.

21 January 2018

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Novel signal transducer and activator of transcription 3 (STAT3) mutations, reduced T(H)17 cell numbers, and variably defective STAT3 phosphorylation in hyper-IgE syndrome

21 January 2018

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Transcriptional responses to polypeptide ligands: the JAK-STAT pathway

21 January 2018

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JAK-STAT signaling: from interferons to cytokines

21 January 2018

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Interleukin-6-induced STAT3 transactivation and Ser727 phosphorylation involves Vav, Rac-1 and the kinase SEK-1/MKK-4 as signal transduction components

21 January 2018

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c-Jun and c-Fos cooperate with STAT3 in IL-6-induced transactivation of the IL-6 respone element (IRE).

21 January 2018

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Prediction of water and metal binding sites and their affinities by using the Fold-X force field

21 January 2018

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Spacing of palindromic half sites as a determinant of selective STAT (signal transducers and activators of transcription) DNA binding and transcriptional activity

21 January 2018

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Interferon activation of the transcription factor Stat91 involves dimerization through SH2-phosphotyrosyl peptide interactions

21 January 2018

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A family of cytokine-inducible inhibitors of signalling

21 January 2018

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Opposing actions of STAT-1 and STAT-3.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12038-012-9202-X

Mapping of Stat3 serine phosphorylation to a single residue (727) and evidence that serine phosphorylation has no influence on DNA binding of Stat1 and Stat3.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12038-012-9202-X

Maximal activation of transcription by Stat1 and Stat3 requires both tyrosine and serine phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12038-012-9202-X

Reversible methylation of promoter-bound STAT3 by histone-modifying enzymes

21 January 2018

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STATs in cancer inflammation and immunity: a leading role for STAT3

21 January 2018

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Stat3: a STAT family member activated by tyrosine phosphorylation in response to epidermal growth factor and interleukin-6

21 January 2018

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STAT3Mutation in the Original Patient with Job's Syndrome

21 January 2018

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https://api.crossref.org/works/10.1007%2FS12038-012-9202-X

Differential Activation of Acute Phase Response Factor/STAT3 and STAT1 via the Cytoplasmic Domain of the Interleukin 6 Signal Transducer gp130

21 January 2018

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https://api.crossref.org/works/10.1007%2FS12038-012-9202-X

Job's Syndrome. Recurrent, "cold", staphylococcal abscesses

21 January 2018

1 reference

12 December 2020

Defects along the T(H)17 differentiation pathway underlie genetically distinct forms of the hyper IgE syndrome

1 reference

12 December 2020

STAT3 is required for IL-21-induced secretion of IgE from human naive B cells

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12 December 2020

Differential activation of acute phase response factor/STAT3 and STAT1 via the cytoplasmic domain of the interleukin 6 signal transducer gp130. I. Definition of a novel phosphotyrosine motif mediating STAT1 activation

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12 December 2020

Extreme hyperimmunoglobulinemia E and undue susceptibility to infection

1 reference

12 December 2020

10.1007/S12038-012-9202-X

Identifiers

DOI

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11 September 2017

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