(Q39376541)

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Transcriptional regulation of T-type calcium channel CaV3.2: bi-directionality by early growth response 1 (Egr1) and repressor element 1 (RE-1) protein-silencing transcription factor (REST).

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Europe PubMed Central

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11 September 2017

Transcriptional regulation of T-type calcium channel CaV3.2: bi-directionality by early growth response 1 (Egr1) and repressor element 1 (RE-1) protein-silencing transcription factor (REST). (English)

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11 September 2017

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Susanne Schoch

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11 September 2017

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Karen M J van Loo

1

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11 September 2017

Christina Schaub

2

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11 September 2017

Katharina Pernhorst

3

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11 September 2017

Yoel Yaari

4

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11 September 2017

Heinz Beck

5

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11 September 2017

Albert J Becker

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11 September 2017

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19 March 2012

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11 September 2017

Journal of Biological Chemistry

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11 September 2017

287

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Europe PubMed Central

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11 September 2017

19

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Europe PubMed Central

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11 September 2017

15489-15501

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Europe PubMed Central

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11 September 2017

Neuron-restrictive silencer factor-mediated hyperpolarization-activated cyclic nucleotide gated channelopathy in experimental temporal lobe epilepsy

1 reference

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T-type calcium channel expression and function in the diseased heart

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Role of the T-type calcium channel CaV3.2 in the chronotropic action of corticosteroids in isolated rat ventricular myocytes

1 reference

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An acquired channelopathy involving thalamic T-type Ca2+ channels after status epilepticus

1 reference

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17Beta-estradiol regulation of the mRNA expression of T-type calcium channel subunits: role of estrogen receptor alpha and estrogen receptor beta

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Gene expression analysis in absence epilepsy using a monozygotic twin design

1 reference

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Ca(V)3 T-type calcium channel isoforms differentially distribute to somatic and dendritic compartments in rat central neurons

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Molecular pathways underlying the modulation of T-type calcium channels by neurotransmitters and hormones.

1 reference

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Specific T-type calcium channel isoforms are associated with distinct burst phenotypes in deep cerebellar nuclear neurons.

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1 reference

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The transcription factor Egr1 is a direct regulator of multiple tumor suppressors including TGFbeta1, PTEN, p53, and fibronectin.

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Effects of Cav3.2 channel mutations linked to idiopathic generalized epilepsy

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Gating effects of mutations in the Cav3.2 T-type calcium channel associated with childhood absence epilepsy

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NRSF regulates the fetal cardiac gene program and maintains normal cardiac structure and function

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Homotypic regulatory clusters in Drosophila.

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Ischemic insults derepress the gene silencer REST in neurons destined to die

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Specific contribution of human T-type calcium channel isotypes (alpha(1G), alpha(1H) and alpha(1I)) to neuronal excitability

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Role of EGR1 in hippocampal synaptic enhancement induced by tetanic stimulation and amputation

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Nickel block of three cloned T-type calcium channels: low concentrations selectively block alpha1H.

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Protein kinase A regulates cholinergic gene expression in PC12 cells: REST4 silences the silencing activity of neuron-restrictive silencer factor/REST.

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1 reference

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Neuronal expression of zinc finger transcription factor REST/NRSF/XBR gene

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Identification of potential target genes for the neuron-restrictive silencer factor

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NAB2, a corepressor of NGFI-A (Egr-1) and Krox20, is induced by proliferative and differentiative stimuli

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Identification of NAB1, a repressor of NGFI-A- and Krox20-mediated transcription

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Functional significance of an overlapping consensus binding motif for Sp1 and Zif268 in the murine adenosine deaminase gene promoter

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Transcriptional control of KCNQ channel genes and the regulation of neuronal excitability

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A Cav3.2 T-type calcium channel point mutation has splice-variant-specific effects on function and segregates with seizure expression in a polygenic rat model of absence epilepsy

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based on heuristic

inferred from PubMed ID database lookup

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1 reference

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based on heuristic

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10.1074/JBC.M111.310763

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Europe PubMed Central

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11 September 2017

release_mymebjbf55dcvf6gfelnolextq

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24 November 2022

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Europe PubMed Central

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11 September 2017

PubMed publication ID

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Europe PubMed Central

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11 September 2017

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