(Q39538428)

12 September 2017

title

Twist2 contributes to breast cancer progression by promoting an epithelial-mesenchymal transition and cancer stem-like cell self-renewal (English)

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12 September 2017

1 reference

X Fang

1

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12 September 2017

Y Cai

2

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12 September 2017

J Liu

3

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12 September 2017

Z Wang

4

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12 September 2017

Q Wu

5

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12 September 2017

Z Zhang

6

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12 September 2017

C J Yang

7

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12 September 2017

L Yuan

8

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12 September 2017

G Ouyang

9

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12 September 2017

language of work or name

English

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publication date

23 May 2011

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12 September 2017

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12 September 2017

volume

30

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12 September 2017

issue

47

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12 September 2017

page(s)

4707-4720

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Epithelial-mesenchymal transitions: the importance of changing cell state in development and disease.

12 September 2017

cites work

1 reference

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Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

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Epithelial-mesenchymal transition-derived cells exhibit multilineage differentiation potential similar to mesenchymal stem cells

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Opinion: migrating cancer stem cells - an integrated concept of malignant tumour progression

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Disruption of Stat3 reveals a critical role in both the initiation and the promotion stages of epithelial carcinogenesis

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twist is required in head mesenchyme for cranial neural tube morphogenesis

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Twist is transcriptionally induced by activation of STAT3 and mediates STAT3 oncogenic function

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Morphogenesis and oncogenesis of MCF-10A mammary epithelial acini grown in three-dimensional basement membrane cultures

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Translational activation of snail1 and other developmentally regulated transcription factors by YB-1 promotes an epithelial-mesenchymal transition

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The T-box transcription factor Brachyury promotes epithelial-mesenchymal transition in human tumor cells

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Redundant or separate entities?--roles of Twist1 and Twist2 as molecular switches during gene transcription

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Dermo-1, a multifunctional basic helix-loop-helix protein, represses MyoD transactivation via the HLH domain, MEF2 interaction, and chromatin deacetylation

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MicroRNAs differentially regulated by Akt isoforms control EMT and stem cell renewal in cancer cells.

21 January 2018

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TWIST family of basic helix-loop-helix transcription factors mediate human mesenchymal stem cell growth and commitment.

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Hedgehog signaling and Bmi-1 regulate self-renewal of normal and malignant human mammary stem cells

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Mammary stem cells, self-renewal pathways, and carcinogenesis

21 January 2018

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Epidermal growth factor receptor cooperates with signal transducer and activator of transcription 3 to induce epithelial-mesenchymal transition in cancer cells via up-regulation of TWIST gene expression

21 January 2018

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Cadherin switching: essential for behavioral but not morphological changes during an epithelium-to-mesenchyme transition.

21 January 2018

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The epithelial-mesenchymal transition generates cells with properties of stem cells

21 January 2018

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Six1 expands the mouse mammary epithelial stem/progenitor cell pool and induces mammary tumors that undergo epithelial-mesenchymal transition

21 January 2018

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Generation of breast cancer stem cells through epithelial-mesenchymal transition

21 January 2018

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Loss of E-cadherin promotes metastasis via multiple downstream transcriptional pathways

21 January 2018

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Molecular signaling of the epithelial to mesenchymal transition in generating and maintaining cancer stem cells

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Snail, Zeb and bHLH factors in tumour progression: an alliance against the epithelial phenotype?

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The multifaceted role of periostin in tumorigenesis

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EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer

21 January 2018

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Osteopontin promotes gastric cancer metastasis by augmenting cell survival and invasion through Akt-mediated HIF-1alpha up-regulation and MMP9 activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2011.181

The polycomb group protein Bmi-1 represses the tumor suppressor PTEN and induces epithelial-mesenchymal transition in human nasopharyngeal epithelial cells

21 January 2018

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https://api.crossref.org/works/10.1038%2FONC.2011.181

Interleukin-6 induces an epithelial-mesenchymal transition phenotype in human breast cancer cells.

21 January 2018

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Epithelial-mesenchymal transitions in development and disease

21 January 2018

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Complex networks orchestrate epithelial-mesenchymal transitions

21 January 2018

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The twist gene: isolation of a Drosophila zygotic gene necessary for the establishment of dorsoventral pattern

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Epithelial-mesenchymal transition induced by growth suppressor p12CDK2-AP1 promotes tumor cell local invasion but suppresses distant colony growth

21 January 2018

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Twist modulates breast cancer stem cells by transcriptional regulation of CD24 expression

21 January 2018

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The EMT-activator ZEB1 promotes tumorigenicity by repressing stemness-inhibiting microRNAs

21 January 2018

1 reference

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Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

21 January 2018

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Epithelial-mesenchymal transition: at the crossroads of development and tumor metastasis

21 January 2018

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Regulation of transforming growth factor-beta 1-induced apoptosis and epithelial-to-mesenchymal transition by protein kinase A and signal transducers and activators of transcription 3.

21 January 2018

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TWISTing stemness, inflammation and proliferation of epithelial ovarian cancer cells through MIR199A2/214.

21 January 2018

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https://api.crossref.org/works/10.1038%2FONC.2011.181

A developmentally regulated inducer of EMT, LBX1, contributes to breast cancer progression

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2011.181

Activation of the PTEN/mTOR/STAT3 pathway in breast cancer stem-like cells is required for viability and maintenance

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2011.181

Stat proteins and oncogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2011.181

Bmi1 is essential in Twist1-induced epithelial–mesenchymal transition

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2011.181

31 March 2020

Identifiers

DOI

10.1038/ONC.2011.181

1 reference

12 September 2017

1 reference